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logo-with-flagsCHLORINE DIOXIDE GAS DOES

NOT DEPLETE THE

OZONE LAYER AS CHLORINE GAS DOES.

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  New Link  

↑  ↑  ↑


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~  THE SCIENTIFIC METHOD ~

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↑  Click on His Image  for ‘The Book’  ↑

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 Chlorine dioxide – ClO2 +

 

  MANAGEMENT OF BIOFILM 

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Chlorine dioxide a better disinfection solution

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Chlorine Dioxide  does not produce THMs as chlorine does, and doesn’t give rise to the formation of chloroform and bromochloromethane as chlorine does.

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Chlorine Dioxide 
does not react with ammonia, nitrogenous compounds or precursors to form chloroform as chlorine does…”

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Chlorine dioxide as a disinfectant has the advantage that it directly reacts with the cell wall of micro-organisms. This reaction is not dependent on reaction time or concentration. In contrast to non-oxidizing disinfectants, chlorine dioxide kills micro-organisms even when they are inactive.

Therefore the chlorine dioxide concentration needed to effectively kill micro-organisms is lower than non-oxidising disinfectant concentrations. Micro-organisms cannot built up any resistance against chlorine dioxide.

See more below 

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EXTRACT FROM ABOVE:  

Discussion

Mechanism of inactivation of CIO2, on viruses

   The mechanism of killing bacteria by disinfectant, in some respects, suits virus also. However, due to the virus possessing its special structure, the disinfectants, i.e. CIO2 and liquid chlorine, do not express completely identical inactivation effect on virus and bacteria.  
A virus is a kind of micro, super parasite. Generally, its resistance to disinfectants is stronger than the bacteria (Meng Zao, 1986). Now generally speaking, chlorine does not only react with the protein of capsomeres to destroy semi-permeability of membrane of protein and making it disappear, but also reacts with internal RNA and the RNA is first deteriorated and then removed. Thus, chlorine can kill the viruses and the killed viruses are decomposed.  Chlorine dioxide can absorb and penetrate into the protein of capsomeres and react with internal RNA. Thus, CIO2 damages the capacity of genetic group as the model of RNA to be synthesized. Therefore, the surface of the virus absorbs high concentrations of CIO2 molecules. This strengthens the disinfection effect of chlorine dioxide considerably and results in viruses to be inactivated eventually. Different inactivation effects of using these two disinfectants. As can be seen in our experimental results, for liquid chlorine, under conditions of 7.0mg/L dose and contacting for 30 and 60 min, there were no inactivation effects on Policvirus-1, Coxsackie virus-B3, ECHO-11, Adenoviros-7, Herpes simplex virus-1 and Mumps virus. As CIO2, under conditions of 1.0mg/L dose and contacting for 30min may attain the inactivation effect on all six kinds of virus mentioned above. This shows that CIO2 has more obvious inactivation effect on viruses than liquid chlorine.

Notice the date – 1994 !

Full text → HERE

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↓ Watch ↓

  New Link  

More recent

Australian Drinking Water Guidelines
[The TGA and Chris Steger please note]
Chlorine dioxide has been approved by NHMRC 
for use in disinfecting drinking water since 2005…

The Australian Drinking Water Guidelines (the ADWG) published by
the National Health and Medical Research Council (NHMRC) and
Natural Resource Management Ministerial Council (NRMMC)
in 2004 are not mandatory standards but provide a basis
for determining drinking water quality  ↓  ↓  ↓  ↓  ↓

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 Natural Water Solutions-Chlorine Dioxide Approvals 

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  Chlorine dioxide a better disinfection solution 

Microorganisms cannot build resistance to chlorine dioxide.

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His book #2 → HERE 

Slow to load-208 pages

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158 » FILES

 

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Professional Perspectives: Fluoride in Tap Water – Video

 

httpv://www.youtube.com/watch?v=_Ys9q1cvKGk

 

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This is a hot issue – cover-up and denial is the ADA’s only option,
given 
the serious implications of  fluoridation on millions
of kidneys both
 human and animal (pets) and
the corrosion of 
infrastructure.

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F.blood lead : Afican Am. ss

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At present, U.S. public water systems serving over 140 million people are fluoridated with
200,000 tons of commercial grade hydrofluosilicic acid (H2SiF6) and sodium
silicofluoride (Na2SiF6), together called “silicofluorides” (or “SiFs”).

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Data from numerous studies show that, taking economic, social and racial factors into account,
where silicofluorides are used, children absorb more lead from the environment, and there
are higher rates of diseases and behavioral problems associated with lead poisoning
(including hyperactivity, substance abuse, and violent crime)
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Although some early studies showed differences between sodium fluoride and sodium silicofluoride, to this day the substitution of silicofluorides in public water treatment facilities HAS NEVER BEEN SUBJECTED TO APPROPRIATE ANIMAL OR HUMAN TESTING. Recently, the Assistant Administrator of the EPA admitted to Congress that his agency had no data on SiF toxicity and the Chief of the Treatment Technology Evaluation Branch at the National Risk Management Research Laboratory confirmed that the EPA has “no” data on the “health and behavioral effects of fluosilicic acid.”

Despite claims of safety by oral health officials, laboratory research in Germany revealed that silicofluorides do not dissociate completely and have important biological effects. To follow up on this issue, we have compared children’s blood lead levels in communities using SiF treated water with communities using sodium fluoride or with non-fluoridated water. In three separate samples, totaling over 400,000 children, SiF treated municipal water is ALWAYS significantly associated with increased blood lead levels in children.

This effect was evident in a Massachusetts survey of lead levels in 280,000 children (see graph for children exposed to SiF from the Greater Boston water system, from towns that add SiF locally, or from communities using sodium fluoride, and towns without fluoridation). For the state of New York, data was available on venous blood lead levels for 151,225 children in communities of 15,000 to 75,000. Controlling for other factors associated with higher blood lead, silicofluorides were again significantly associated with higher uptake of lead from the environment. For black children, who are especially at risk for high blood lead, those in towns using SiF were less likely to have low blood lead and more likely to have lead over 10µg/dL. To confirm that these results are not due to other socio-economic or demographic factors, additional statistical tests were run.

The third study concerned children’s blood lead levels in the National Health and Nutrition Evaluation Survey (NHANES III), which had reports for 7224 children from 80 counties with populations over 500,000. Since only 4 of these counties had any communities that used sodium fluoride, analysis of the NHANES III data focused on the percentage of the entire county population exposed to silicofluoride treated water.

Among the 1543 children of all ages from large urban counties with over 80% of the population exposed to fluoridation (almost all of whom receive water treated with SiF), average blood lead was 5.12 µg/dL whereas the average for 1139 children in low fluoride exposure counties was 3.64 µg/dL Blood lead in the 473 children sampled from the medium fluoridation counties was 3.23 µg/dL, which was significantly different from the high fluoridation counties but not from either low fluoridation counties or those with unknown fluoridation status, where average blood lead levels were 3.16 µg/dL (S.D. 2.83). Controlling for the Poverty, the effect of SiF use was highly significant (p < .0001). When the sample is divided by age and race, these findings provide six separate samples in which SiF is associated with high blood lead (see Graphs).

In all three populations studied, those children in each racial category and each age group who were highly likely to be exposed to silicofluorides differ strongly in levels of blood lead from those not exposed.

This conclusion was further checked by analyzing available data for health and behavioral traits that have been associated with high blood lead (such as violent crimes, cocaine use and asthma). In each case, those exposed to silicofluoride treated water were more likely to have behavioral or health problems that are more likely among those with high lead in their bodies.

The injection of silicofluorides in public water supplies is a practice whose elimination could possibly contribute to reduced rates of learning disabilities, substance abuse, violent crime, and asthma (all connected with lead poisoning and other toxins). Whatever the benefits to teeth (and this is highly controversial), our research shows that the issues facing the public concern silicofluoride chemistry, toxicology, and the linkage of neurotoxins with behavior or health. Before SiF chemicals are used, citizens must know that they are safe for all.

Dury & Young F.

Indigenous A ss

“Close The Gap”

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 Fluoridation and Renal Disease in Aboriginal and Torres Strait Islanders

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 Fluoride Ion Toxicity in Human Kidney Collecting Duct Cells

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PEOPLE WITH KIDNEY IMPAIRMENT ARE AT GREATER RISK FROM FLUORIDES
Indigenous Australians have smaller kidneys than non-indigenous Australians
a reduced number of nephrons and decreased renal reserve –
(Singh G, White A, Spencer J, Wang Z, Hoy W [1999]).

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Qld. Indigenous Water Fluoridated -Bamaga

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Inorganic fluoride. Divergent effects
on human proximal tubular cell viability.
R. A. Zager and M. Iwata. Fred Hutchinson Cancer Research Center, Seattle.
Inorganic fluoride. Divergent effects on human proximal tubular cell …

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We visited the affected villages with the medical team comprising
Dr. Tilak Abeyesekera 
and Dr. Nimmi Athureliye
where positive CKDU patients were identified.

Fluoride, Cadmium, Arsenic- Renal Failure – Sri Lanka – Prof. Oliver A. Ileperuma

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image-of-kidneyNational Kidney Foundation

There is consistent evidence that impairment of kidney function
results in changes to the way in which fluoride is metabolized and
eliminated 
from the body, resulting in an increased burden of fluoride.

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Impairment of Kidney Function – INDIA

Kidneys are among the most sensitive body organs in their histopathologial
and functional responses to excessive amounts of fluoride.
They are the primary organs concerned with
excretion and retention of fluoride…

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THE UNIVERSITY OF BURDWAN BURDWAN-713104
WEST BENGAL, INDIA 

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“FLUORIDE POLLUTION AND ITS EFFECT ON WATER AND
VEGETATION IN BIRBHUM DISTRICT, WEST BENGAL”

See thesis below   detailed diagrams + more photos.

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by

KARTICK CHANDRA PAL, M.Sc. (Chem)

We thank you  for such a mammoth undertaking.

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More info on Fluoride in India → HERE

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Endemic Fluorosis Patiala – 1962

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Infants Warning on Fluoridation – American Dental Association

↑  New York – 13 November  2006 ↑ 
To prevent tooth damage, the American Dental Association (ADA)
warned its members that fluoridated water should
not be mixed into concentrated formula
or foods intended for babies
one year and younger…

[ Exposure to Fluorides while teeth are forming
will result in visual damage – dental fluorosis.
This would be a bad look for fluoridation.
]

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 See also 
Dartmouth Researcher Warns of Chemicals Added to Drinking Water

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Fluoridation Queensland Logo

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Rudolf  Ziegelbecker began a successful career in
electron microscopy, where his remarkable
talents as a researcher soon emerged.

FLUORIDATION & RUDOLF ZIEGELBECKER

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OBITUARY

Fluoride 42(1)2 January-March 2009

Rudolf Ziegelbecker Obituary 2 Burgstahler, Spittle

RUDOLF ZIEGELBECKER

         Born 26 August 1924 in Veitsch, Austria Died 12 January 2009 in Graz, Austria.
After early training and work in electrical engineering,

Rudolf Ziegelbecker began a successful career in electron microscopy, where his remarkable talents as a researcher soon emerged. Additionally, because it had not been possible due to World War II, he also began to pursue university studies of electrical engineering and physics. Drawing on his knowledge and experience in research, he then directed his efforts to improve living conditions of his fellow citizens, co-founding an Institute for Environmental Research in Graz in 1970. As part of this activity, he was instrumental in ending environmentally hazardous industrial operations in his neighborhood, which also prompted his research on fluoride beginning in the late 1960s.

Rudolf Ziegelbecker, 1924–2009

At the local brick works in Graz-St. Peter, Ziegelbecker
discovered that fluoride emissions were not only damaging the local vegetation but were also impairing the health of his own and other children who were regularly receiving prophylactic fluoride tablets to protect against dental caries. The children were suffering from unexplained excessive fatigue, nausea, stomach-ache, and joint and limb pain, from which they recovered when they stopped taking the fluoride tablets and the polluting operations were terminated. When he pressed the local health authorities to provide data for a maximum allowable fluoride intake and evidence for an anti-caries benefit of the tablets, they were unable to do so.

He then undertook a critical examination of various aspects of the dental use of fluoride. From his systematic analyses of data and conclusions published by leading dental researchers, he was able to show Austrian authorities that neither fluoride tablets nor water fluoridation were effective in preventing tooth decay as claimed. As a result, first the local and then the entire fluoride tablet program in Austria was terminated.

Over the next four decades, Ziegelbecker campaigned tireless against water fluoridation, fluoride tablets, and salt fluoridation. In this connection, he wrote numerous scientific and popular articles and co-authored a book with Dr Max Otto Bruker (Vorsicht Fluor, reviewed by Jörg Spitz in Fluoride 2007;40/3:205-6). He also testified at expert hearings, reported at conferences and to governmental committees, participated in panel discussions, and appeared on radio and television. His vigorous and persistent campaigning eventually played an important role in ending water fluoridation and fluoride tablet programs in Austria, Germany, and other countries in Europe.

On April 19, 2007 (as reported by Peter Meiers in Fluoride 2007;40/3:160-1), Ziegelbecker’s untiring efforts to promote a better quality of life were recognized by the Town Council with a “Citizen of Graz” award for all he had achieved for the city and its citizens. During the past year, he lost his eyesight and was stricken with cancer from which he died this year on January 12. The next issue of Fluoride will carry an In Memoriam tribute with more details of his work and life.

Albert W Burgstahler, Editor Lawrence, Kansas, USA Bruce Spittle, Managing Editor Dunedin, New Zealand

Copyright © 2009 The International Society for Fluoride Research Inc.  www.fluorideresearch.org www.fluorideresearch.com  www.fluorideresearch.net  Editorial Office:
727 Brighton Road, Ocean View, Dunedin 9035, New Zealand. 

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…Fluoride is a sabotage toxin with the property that
by inhibiting enzymes people may die from problems with
 other organs without fluoride being discovered as the true cause…
~ Rudolf Ziegelbecker

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