This is a hot issue – cover-up and denial is the ADA’s only option,
given the serious implications of fluoridation on millions of
kidneys both human and animal (pets).
This is a Response to the American Dental Association’s Booklet -
Full Text → HERE
Dr. Heyroth is a proponent for fluoridation yet, when testifying under oath at Congressional Hearings to the question: “Would you give fluoridated water to one with kidney trouble,” he answered, “No, the advice would be that he drink fluoride-free spring water.” (Dr. Francis Heyroth of Kettering Institute, Cincinnati, Ohio.)
” … Since kidney damage can be caused by fluoride, there can be a vicious circle by which kidney damage causes more fluoride retention, which in turn causes further kidney damage.” (Fredrick B. Exner, M.D., F.A.C.R.)
“Once renal function is severely impaired, the excretion of fluoride in the urine decreases and serum fluoride concentration increases.” (K. Kono, et al, Industrial Health, 22:33-40, 1984.)
“Many studies … have indicated suspicious bio-chemical events, especially in the kidneys, when consuming 1 ppm or more (up to 5 ppm in our study) fluoride ion in drinking water.” (Harold Warner, Professor Emeritus, Yerkes Regional Primate Center, Emory University, June 20, 1983.)
” … bear in mind that all elderly folk eventually have had one degree or another of ‘diminished renal (kidney) function’ and will accumulate higher tissue levels of fluoride at a time when their tolerance capacities are diminished.” (John R. Lee, M.D.)
“It would also seem prudent to monitor the fluoride intake of patients with chronic renal impairment … particularly those living in areas of high naturally occurring fluoride, children, those with excessive thirst, and those with prolonged disease.” (“Position Paper on Fluoride,” National Kidney Foundation, page 2, Aug. 16, 1980.)
“Sodium fluoride is a very toxic chemical. It reacts with growing tooth enamel and with bones to produce irreversible damage. It may injure the nervous system, kidneys and other tissues of susceptible individuals. Fluoride is not a necessary trace element for dental health. Uniform dosage of any drug dissolved in the water supply is obviously impossible. Some individuals may drink 10 times as much as others. Thirst has many variables.” (Dr. Granville F. Knight, (M.D., F.A.C., F.I.A.A.) Santa Barbara, California.)
“Until recently, I favored fluoridation of the City Water supply in the proportion of 1 ppm, but information provided me by Dr. C. C. Bass, Dean Emeritus of Tulane Medical School for the past 15 years (who has pursued research and study pertaining to teeth) along with some research done by British scientists indicating possible bad effects on the kidneys from fluoridation, has caused me to reverse my opinion on the matter.” (Dr. Alton Ochsner, President Ochsner Foundation Hospital and Head of Department of Surgery, Tulane University Medical School, New Orleans, Louisiana.)
“You are absolutely correct in stating that many people have diabetes but don’t know it. In the U. S., approximately half of the estimated 12 million people who have the disease don’t realize it … Diabetic care also depends on whether the individual suffers from complications associated with diabetes, such as kidney failure, coronary artery disease, etc. According to the National Institute of Dental Research, fluoride levels in water are set according to normal consumption of water. If an individual is consuming abnormally large quantities of water, he should drink bottled water.” (See 31-1: Department of Health and Human Services letter, Jan. 4, 1991). Nearly all diabetics and many athletes drink abnormally large quantities of water-many drink over a gallon a day. Therefore the Public Health Service is saying these people should buy bottled water.
“Because it can be documented that fluorides were given as medication for hyperthyroid patients, it should be considered the obvious cause for hypothyroidism and other thyroid-hormone function-related disorders, including ADHD, arthritis, osteoporosis, etc., especially at intake levels as high as they are.” (Thyroid Hormones, pp. 5, by Andreas Schuld, Parents of Fluoride Poisoned Children, PFPC., Vancouver, B.C., Canada.)
“Fluoride decreases the function of the thyroid gland by 30% to 40% and this is one of the most important glands in the body. In several parts of our country, children receive tablets with fluorine, but the fluoridation of drinking water has not been accomplished. We trust it will not happen in view of the special circumstances here regarding our thyroid problem. Research here demonstrates clearly an antagonism between iodine and fluorine. We also showed, in another experiment, that the calcium metabolism is greatly affected by fluorine. Since the bone picks up 30% less calcium in the presence of fluorine, the danger of osteoporosis in a growing organism is very great.” (Dr. T. Gordonoff, Professor of Pharmacology, Bern, Switzerland.)
“Long continued ingestion of minute quantities of fluorine causes disease of the thyroid gland.” (Dr. Douw G. Steyn, Department of Pharmacology, University of Pretoria, South Africa.)
“We have posted over 100 studies documenting the adverse effects of fluoride on the thyroid gland from the last 70 years or so in the Virtual Library on Fluoride Research at www.bruha.com.” (Andreas Schuld, Parents of Fluoride Poisoned Children (PFPC), Vancouver, B.C., Canada.)
“A fairly substantial body of research indicates that patients with chronic renal insufficiency are at an increased risk of chronic fluoride toxicity. These patients may develop skeletal fluorosis even at 1 ppm fluoride in the drinking water.”
(Dr. Helmut Schiffl, 2008) → HERE
→ Fluoride Risks Are Still A Challenge ←
Kidney patients and diabetics are at special risk from fluoridated water
because they tend to drink more liquid than healthy individuals…
Low birth weight predisposes to renal disease in Aboriginal adults. This could be due to reduced nephron numbers, which might be reflected in lower kidney volumes, at least early in life. In this study we evaluated the association of birth weight with renal volume in 174 children and in adolescents 5 to 18 years old in an Aboriginal community with high rates of renal disease. Their mean birth weight was 2.9 kg, and 19% had been low birth weight (<2.5 kg). Kidney dimensions were measured by ultrasound by a single observer, and kidney volume (KV) was calculated from the formula KV (mL) = length x (depth 1 + depth 2)/2 x 0.523. Combined kidney volume, corrected for body surface area (corrKV), was independent of age and averaged 240 +/- 45 mL/1.73 m(2). The average corrKV was significantly lower (by about 20 mL) in low birth weight children than in those with “normal” birth weight. There was a difference of 32 mL in combined corrKV between children in the lowest versus the highest quintile of birth weight. Disparities in calculated kidney volume were driven more by lower kidney depth than length in low birth weight children. We conclude that low birth weight children have lower renal volumes than children of higher birth weights, after correction for current body size. This conclusion is compatible with the theory that intrauterine growth retardation is associated with reduced nephron endowment. The susceptibility to renal disease associated with low birth weight in this population might be mediated in part through this mechanism.