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Results reveal that anemia was reduced and pre-term and low-birth-weight
babies were considerably fewer in the fluoride-avoidance group as
compared to the control. Two stillbirths occurred in the
control group, none in the experimental group.

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FLUORIDE LINKED TO PRE-TERM BIRTH AND
ANEMIA IN PREGNANCY.

 

 Fluoride avoidance reduced anemia in pregnant women, decreased pre-term births and enhanced babies’ birth-weight, concludes leading fluoride expert, A K Susheela and colleagues, in a study published in Current Science (May 2010).

       Susheela’s team explains that anemia in pregnancy, which can lead to maternal and infant mortality, continues to plague many countries despite nutritional counseling and maternal iron and folic acid supplementation. This is the first examination of fluoride as an additional risk factor for anemia and low-birth-weight babies.

Anemic pregnant women living in India, whose urine contained 1 mg/L fluoride or more, were separated into two groups. The experimental group avoided fluoride in water, food and other sources and ate a nutritious diet per instruction. The control group received no instructions. Both groups supplemented with iron and folic acid.

Results reveal that anemia was reduced and pre-term and low-birth-weight babies were considerably fewer in the fluoride-avoidance group as compared to the control. Two stillbirths occurred in the control group, none in the experimental group.

Susheela et al. writes, “Maternal and child under-nutrition and anemia is not necessarily due to insufficient food intake but because of the derangement of nutrient absorption due to damage caused to GI (gastrointestinal) mucosa by ingestion of undesired chemical substances, viz. fluoride through food, water and other sources.”

Fluoride avoidance regenerated the intestinal lining which enhanced the absorption of nutrients as evidenced by the reduction in urinary fluoride followed by rise in hemoglobin levels, they report.

Could the same thing be happening in the United States? State University of New York researchers found more premature births in fluoridated than non-fluoridated upstate New York communities, according to a presentation made at the 2009 American Public Health Association’s annual meeting.

Current Science reports that adverse reactions of fluoride consumption are known to occur including reducing red blood cells, reducing blood folic acid activity, inhibiting vitamin B12 production and the non-absorption of nutrients for hemoglobin biosynthesis.

“Citizens must demand that water fluoridation be stopped,” says attorney Paul Beeber, President, New York State Coalition Opposed to Fluoridation, Inc. “It’s disturbing that public-health officials and organized dentistry continue to ignore the overwhelming evidence revealing fluoride to be non-nutritive, unnecessary and unsafe,” says Beeber.

     Source: NYS Coalition Opposed to Fluoridation, Inc.

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The fact that sodium fluoride would control certain
types of insects has been known for many years,
but all attempts to use it and other fluorine
compounds on plants failed because
of plant injury...

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“Fluorine has played a significant role in insect control since about 1896 when
sodium fluoride and various iron fluorides were patented in England as insecticides.

   Sodium fluoride was used in the United States for cockroach control before 1900 and was introduced in 1915 for the control of poultry lice. However, the use of fluorine insecticides did not become general until the 1930´s when the disadvantages of arsenical residues on food crops became apparent and the inorganic fluorine compounds were introduced as “safer substitutes”.

Systematic investigation of organofluorine insecticides began about 1935 in the I. G. Farbenindustrie and the fluoroalcohols and fluorophosphates (phosphorofluoridates) were intensively investigated largely through the research of Schrader (1952). During World War II fluoro-DDT or “Gix” was used for the control of insects of medical importance. More recently, fluoroacetamide and analogues have been used as systemic insecticides and a large variety of other fluorinated organic compounds have shown insecticidal activity. Sulfuryl fluoride has recently been marketed as a fumigant for household and structural pests…”

Alvord and Dietz, of Grasselli Chemical Company, Cleveland, Ohio, point out certain problems with the use of soluble fluorides as insecticides (Ind. Eng. Chem. 25 (June 1933) 629-633):

“The fact that sodium fluoride would control certain types of insects had been known for many years, but all attempts to use it and other fluorine compounds on plants failed because of plant injury. Progress along the line of utilizing the fluorine compounds in this connection really began with the discovery by Roark that the relatively insoluble fluorides would not injure the foliage and would control certain insects. About the time of this discovery, the Grasselli Chemical Company began to experiment with barium fluosilicate. The development of this material was held back for several years because of plant injury following its use, and it was not until the discovery, quite by accident, that the injury was due to an unsuspected impurity and that the pure compound was in reality safe to most foliage, that rapid progress was made.”

S. Marcovitch gives some details as to how those fluoride insecticides work (Ind. Eng. Chem. 16 (1924) 1249):

“The value of sodium fluosilicate as an insecticide is due to the fact that it is both a contact and stomach poison. Shafer has determined that when a roach walks over powdered sodium fluoride a little of the powder adheres to the lower part of the body, antennae and tarsi of the feet, and dissolves in the exudations of the integument. This seems to cause some irritation and uneasiness; the insect soon begins to clean the moistened powder from the body by licking it. In doing this enough of the poison may be brought into the mouth and swallowed, to kill after a period varying in from five to ten days. Other insects, such as Mexican bean beetles, also have the habit of cleaning themselves and by putting their feet in their mouths become very easy to kill. For this reason the sodium fluosilicate is more effective against the adult beetles than the larvae, which do not have these habits.”

Because of such habits, toxicity to higher animals became of concern (Marcovitch S.: “The fluosilicates as insecticides”, Ind. Eng. Chem. 18 (June 1926) 572-573

.
The Patents:

1896

Charles Henry HIGBEE, of New York City, N.Y., Manager of Manufacturing Company: “An improved composition or material for destroying insects”, British Patent GB 8236; filed April 18, 1896; pat. May 23, 1896. (“The compounds of fluorine which I employ for the purpose of destroying insects, are certain soluble ones, viz.: sodium fluoride, ferric fluoride, the silico-fluorides of the same bases, hydro-fluo-silicic acid, and the boro-fluo-silicates”, which the inventor claims to be less toxic for humans then many of the compounds then in use for the same purpose, i.e. “arsenic, copper, phosphorus, and the like”)

1906

Karl Heinrich WOLMAN and Bernard DIAMAND, Idaweiche, Oberschlesien, Germany, assignors to Max Marschall, Nice-Cimiez, France: “Preserving composition for fibrous material”, US Patent 934,871; filed Nov. 6, 1906; granted Sept. 21, 1909  (uses “sodium fluorid” and “sodium silico-fluorid”. “We have also found that the salts of hydrofluoric acid and of silicofluoric acid both of which are weak, bactericidal acids when used in connection wioth a strong mineral acid, as above set forth, will produce good results …”)

1908

Carleton ELLIS, assignor, by mesne assignments, to Chadeloid Chemical Company, of New York, N.Y.: “Insecticide”, US Patent 1,082,507; filed March 11, 1908; pat. Dec. 30, 1913 (“The composition comprises a solution of wax in carbon bisulfid, or similar penetrating organic liquid, emulsified with an aqueous solution, considerably thickened for the purpose of emulsification, and carrying in solution a powerful insecticide such as inorganic compounds like bichlorid of mercury and ammonium fluorid, or organic compounds like ammonium formate, etc.”)

1911

Jacques WITTLIN, of Vienna, Austria-Hungary, assignor of one-half to Siegfried Schlewinger, of New York: “Antiseptic”, US Patent 1,044,840; filed Jan. 12, 1911; granted Nov. 19, 1912 (“… my present invention further contemplates the incorporation of ammonium fluorid or equivalent fluorin-containing salts or fluorin compounds in the preparation of the antiseptic, whereby the germicidal or disinfectant properties thereof are very materially increased.”)

1921

Henry Edward Percy HUTCHINGS, of Barking Essex, UK: “Improvements in or relating to rat and other vermin poisons”, British Patent GB 187,424; filed Sept. 15, 1921; pat. Oct. 26, 1922  (a bait for the purpose of rat and mouse extermination, with additions of either sodium fluoride, barium carbonate, squill or oxalic acid, to serve as a basic poison)

1923

Rurik C. ROARK, Baltimore, Md.: “Insecticide”, US Patent 1,524,884; filed Aug. 6, 1923; granted Feb. 3, 1925 (“The poisonous action of soluble fluorides is well known and has been utilized for the control of injurious insects. For example, sodium fluoride, a salt readily soluble in water, is a very effective roach poison and is a common ingredient of roach powders. Potassium and barium fluorides have been similarly employed …”)

“There is nothing new in the use of sodium fluosilicate as an insecticide. Its use for that purpose was described nearly thirty years ago by HIGBEE (English Patent No. 8236, May 23, 1896). More recently, WILLE has reported tests with sodium fluosilicate against roaches and COBENZL mentions it as a common ingredient of rat and insect poisons” (Roark C., Department of Agriculture: “Fluorides vs. fluosilicates as insecticides”, Science 63 (April 23, 1926) 431-2)

1926

Bernard GEHAUF and Harold W. WALKER, of Edgewood, Md.: “Method of making silicofluorides and products thereof”, US Patent 1,617,708; filed May 14, 1926; pat. Feb. 15,1927 (“This invention … also comprises a new composition of matter for insecticidal and other purposes … made by neutralizing hydrofluosilicic acid with the appropriate base … Hydrofluosilicic acid ordinarily is prepared by contacting various waste gases containing silicon fluorid with water.. Waste gases containing silicon fluorid arise in various industries, as in the manufacture of superphosphates.”)

Martin J. FORSELL, Seattle, Washington: “Insecticide”, US Patent 1,618,702; filed Aug. 30, 1926; granted Feb. 22, 1927 (“The insecticide consists of using apple after it is dried and powdered and mixing therewith any well-known poison in powdered form … any one of the compounds of fluorine preferably sodium or potassium fluoride or sodium or potassium silico fluoride …)

Howard S. McQUAID, Cleveland, Ohio, assignor to The Grasselli Chemical Company, of Cleveland, Ohio: “Production of Barium Silicofluoride”; US Patent 1,648,143; filed Nov. 22, 1926; patented Nov. 8, 1927 (Process for production of barium silicofluoride from sodium silicofluoride for use as an insecticide)

1927

Hermann STÖTTER, Leverkusen, assignor to I.G. Farbenindustrie Akt.-Ges., Frankfurt a. M.: “Verfahren zum Schützen von Wolle, Pelzwerk u. dgl. gegen Mottenfraß”, German Patent (DE) 485,101; filed May 26, 1927; granted Oct. 10, 1929  (ammonium bifluoride, potassium ammonium fluoride)

1929

Roscoe H. CARTER, Washington D.C. (Government employee): “Process for the manufacture of insecticides and method of making same”, US Patent 1,842,443; filed Nov. 15, 1929; granted Jan. 26, 1932  (“As pointed out in other patent applications of mine, the double fluorides of the alkali metals are useful insecticidal materials and can be formed from water soluble salts of aluminum by treatment with alkali metal compounds and fluorine acids in the proper molecular proportions.”)

1931

Arthur H. HENNINGER, assignor to General Chemical Company, New York: “Process of making potassium aluminum fluoride”, US Patent 1,937,956; filed June 18, 1931; pat. Dec. 5, 1933 (“… for use as an insecticide. It has heretofore been proposed to use potassium aluminum fluoride as an insecticide for the control of various insect pests. This material is considered to possess advantages over lead arsenate as an insecticide for the reason that, although poisonous, the fluoride compound is less toxic to human beings and animals than is lead arsenate.”)

1932

Earl B. ALVORD, assignor to Grasselli Chemical Company, Cleveland, Ohio: “Noncorrosive insecticdal compositions”, US Patent 1,931,367; filed Aug. 24, 1932; patented Oct. 17, 1933 (addition to their barium fluosilicate of a slightly water-soluble substantially neutral fluoride (such as cryolite, or barium fluoride) to overcome corrosive effects of the barium fluosilicate upon spray pumps)

1938

John E. MORROW, assignor to Aluminum Company of America: “Insecticide and method of producing same”, US Patent 2,210,594; filed Jan. 6, 1938; pat. Aug. 6, 1940 (“Double fluorides of sodium and aluminum, such as natural and synthetic cryolite, have been used as insecticides, and the usefulness of such compounds as stomach poisons for various insects has been established. It has been demonstrated, for example, that these fluorides are particularly useful in combatting the codling moth and the Mexican bean beetle.”)

1948

Alan BELL, Kingsport, Tennessee, assignor to Eastman Kodak Company, Rochester, N. Y.: “Insecticidal compositions comprising either hexyl alkyl tetraphosphate or tetra-alkyl pyrophosphate and either an alkali metal fluoride or fluorosilicate”, US Patent 2,514,621; filed Dec. 26, 1948; granted July 11, 1950  (“Diethyl phosphate is the hydrolysis product produced by most of these phosphorus insecticides such as organic insecticides derived from triethyl phosphate – thionyl chloride reaction product, hexaethyltetraphosphate, tetraethylpyrophosphate. This hydrolysis product is not as toxic as parent compound in itself but mixed with NaF or Na2SiF6 has considerable toxicity.”)

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Lab Rat

Lab Rat f

fluorine-toxicosis

Rats Research – Fluoride 1934

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    Notice the advertisement below.  ↓   roch-r1

 

   news-clip-cockroach-poision-F.

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    One Tube of Toothpaste – Cheap 3 Months Rat & Mouse Free

            Safer to eat insects than to swallow toothpaste!

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Extract: The Complete Dinosaur’ + info from China

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    Fluoridation Queensland Logo  

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Coal-burning power plants produce 82% of the total
fluoride air pollution
 in the United States.
China is the biggest coal-producer
and coal-consumer in the world. 

-.-.-.-.-.-.-.-.-.-.-.-.-.-.-.-.-.-.-.-

Data On Coal-Burning And Endemic
Fluorosis Throughout China

FLUOROSIS-coal-burning-China-s

 106 Fluoride Vol. 36 No. 2 106-112 2003 Research Report

For Correspondence: Prof Wuyi Wang, Institute of

Geographical Sciences and Natural Resources

Research, Chinese Academy of Sciences,

Beijing 100101, China. E-mail:

wangwy@igsnrr.ac.cn

ENVIRONMENTAL EPIDEMIC CHARACTERISTICS

OF COALBURNING ENDEMIC FLUOROSIS AND

THE SAFETY THRESHOLD OF COAL FLUORIDE IN CHINA

Yonghua Li, Wuyi Wang, a Linsheng Yang, Hairong Li

Beijing, China

SUMMARY:
Data on coal-burning endemic fluorosis throughout China and on the exposure-response relationship between concentrations of fluoride determined in coal samples and the prevalence of dental fluorosis reported from 17 representative surveillance stations in Southwest China were used to estimate the safety threshold for coal fluoride. Coal-burning endemic fluorosis occurs mainly in the mountainous areas of this part of China, where the prevalence of the disease is closely linked to geochemical parameters of the local environment. In these regions the incidence of dental fluorosis has a significant positive correlation with the concentration of fluoride in coal. The safety threshold of coal fluoride is estimated to be 190 mg/kg by the criterion of 0% incidence of dental fluorosis.

Keywords: China; Coal fluoride; Endemic fluorosis; Safety threshold.


INTRODUCTION

Fluorine (F), the most electronegative and reactive of the halogens, is a common chemical element in the earth’s crust in combined form. F concentrations in rocks and soils are well documented, but data on the F concentration in coal are relatively limited.

1-4 Swaine reported the total F concentration in coal ranges from 20 to 500 mg/kg.

5 Statistical data indicate that the mean concentration of F in coal worldwide is 80 mg/kg, but in China it is 200 mg/kg.

6 In the mountainous areas of Southwest China, it is even higher— up to 3106 mg/kg in local coal.

7 Fluoride in coal can be released into the ambient environment as atmospheric F, waterborne F, and residue F during mining, handling, and combustion.

6-8 In Southwest China, F

            See also:

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Understand the difference ↓

COAL- Steaming & Coking

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  FLUORINE IN CHINESE COALS  

End line

Daishe Wu,a,b,c Baoshan Zheng,
a Xiuyi Tang, d Shehong Li, a Binbin Wang,
Mingshi Wang,a,b
Guiyang, China

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See also ↓

FLUORINE IN CHINESE COALS – Air Pollution

See also ↓

Bone/joint problems-Fluorosis

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Of the 400 children born every year in Mount Isa, about 11 %
according to the last blood lead study, have a blood lead
level in excess of the current acceptable guidelines.

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“…There are about 400 children born every year in Mount Isa
and about 11 per cent of those children, according to the last blood lead
study, have a blood lead level in excess of the current acceptable guideline value…”

The Environmental Protection Agency states on their website that exposure to lead in water that is being consumed above the action level, or 15 parts per Billion, can result in delays in physical and mental development in children, anemia, and muscle problems. In adults, it can cause increases in blood pressure and, eventually with heavy exposure, the development of kidney problems or nerve disorders.

Lead-IQ-

  http://www.naturalnews.com/033122_garden_hoses_lead.html#ixzz1TusKP1ey  

http://www.sonic.net/kryptox/environ/lead/romans.htm

http://fluoride-class-action.com/hhs/comments-re-lead

 LEAD IN ANCIENT ROME

LEAD IN AVIATION FUEL

FLUORIDATION AND LEAD CONTAMINATION

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INTELLECTUAL SUPPRESSION

Brian-Martin

❝ … Most environmental scientists are afraid to take a public stand if it means appearing to challenge powerful corporations, governments or professions. They are afraid of what top officials in their organisation may think and do. They are aware of legislation which prohibits them from speaking to the media about their work without permission. They are afraid that they might be blocked from promotion, shunted to less interesting work, or even dismissed…

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“As is normal, the solution to pollution is dilution.
You poison everyone a little bit rather than poison a few people a lot.
This way, people don’t know what’s going on.”

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Westendorf’s research was based on knowledge
that fluoride ion is an enzyme inhibitor.

Westendorf’s Research – Silicofluorides…

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Full research paper → HERE  

The oral defense took place on 2/18/1975

A Foreword intended to place the Westendorf research in current context indicating why it is relevant to a wide range of contemporary health and behavioral problems has been prepared by Myron J. Coplan and Roger D. Masters whose credentials are also attached.

Foreword by

M J Coplan and R D Masters, April 2001

Second, Westendorf’s research was based on knowledge that fluoride ion is an enzyme inhibitor. Indeed, that feature of ingested fluoride seemed to offer multiple benefits. Knappwost believed that ingested fluoride, by inhibiting cholinesterase, could achieve both greater expression of total saliva and an increase in its fluoride content. The research of his student quite logically examined different forms of ingestible fluoride for their effect on several variants of cholinesterase, Westendorf’s results showed that fluoride in the form of the silicofluoride complex (SiF), as well as several other complexes, was a substantially more powerful inhibitor of cholinesterases than the simple fluoride ion released by sodium fluoride (NaF). This was simply an objective finding.

Third, to account for the more powerful inhibition effect of SiF, Westendorf studied the course of its fluoride release in fine detail. He found that under physiological conditions, dissociation was no more than 66% in the concentration range considered “optimum” for fluoridated water by United States health authorities. If the released fluoride came uniformly from all of the initially injected SiF, the molar concentration of the residual non-dissociated species would be the same as that of the injected SiF. It would follow that dilution of fluosilicic acid to a nominal 1 part per million of free fluoride in water at pH 7.4 induces each [SiF6]2- to release 4 fluorides to be replaced by hydroxyls. The partially dissociated residue would be the ion [SiF2(OH)4]2- which would then be present in the water at the same concentration as the originally introduced SiF. The biological consequences of ingesting such a species are probably not innocuous, with enzyme inhibition being only one of several possibilities.

Westendorf’s visualized course of SiF dissociation, based on actual experimental evidence, is materially at odds with the dissociation route assumed by US EPA and CDC, based on theory. In judging the reliability of the theoretical approach and claims of health safety presented by these government agencies, one should be aware that both the nature of the complicated mixture called “fluosilicic acid” and the course of its dissociation upon dilution remain unresolved despite nearly a century of research. Two recent documents demonstrate this. In the first, an expert in the recovery of fluoride in phosphate rock processing, addressing a group of his peers at a 1999 International Fertilizer Association (a) meeting held in the former USSR, said:

“The chemical formula of fluosilicic acid is H2SiF6. However, things are not as simple as that due to the fact that rarely is fluosilicic acid present as pure H2SiF6. . . There are well reported references to the existence of H2SiF6 SiF4. . . Hereon in this presentation, FSA [fluosilicic acid] means a mixture of HF, H2SiF6 and H2SiF6 SiF4.”

This is a highly significant statement coming from someone who ought to know the subject under discussion. It means that a key intermediate dissociation product postulated by CDC and EPA theories to be transient species only fleetingly after SiF is introduced into the water at the water plant, may be present in concentrated fluosilicic acid before dissociation begins. Such a starting condition would cast serious doubt on the postulated theoretical equations predicting “virtually 100%” dissociation that supposedly “guarantee” no adverse health effects from undissociated SiF residues in drinking water treated with these compounds.

Equally important is a letter (b) dated March 15, 2001, written by the Director of the EPA Water Supply and Water Resources Division, which concludes with the statement:

“In January, representatives from the [EPA] Office of Research and Development (ORD) and the Office of Science and Technology and Ground Water and Drinking Water met to discuss a number of water related issues including Fluoridation. Several fluoride chemistry related research needs were identified including; (1) accurate and precise values for the stability constants of mixed fluorohydroxo complexes with aluminum (III), iron (III) and other metal cations likely to be found under drinking water conditions and (2) a kinetic model for the dissociation and hydrolysis of fluosilicates and stepwise equilibrium constants for the partial hydrolysis products.”

In plain English, senior EPA research staff now believe their staff needs to go back to the lab for at least another year or two to find out if the EPA’s longstanding confidence in the “virtually total” dissociation of SiFs may have been misplaced. Whatever the outcome may be of their new study of SiF dissociation, it is clear the EPA does not intend to perform animal tests to ascertain health effects of chronic ingestion of SiF treated water under controlled conditions.

Animal experiments according to accepted toxicology testing protocols would be the logical way to examine health effects of enzyme inhibition by SiF that Westendorf observed at the cellular level. Three published reports bearing directly on this matter should be noted. In the early 1930s, the Ohio agriculture department wanted to develop a replacement for bone meal as a source of calcium and phosphorus in the feed ration of farm animals. Natural “rock phosphate,” comprising largely calcium phosphate, was a candidate, but it was known to carry about 2 to 5% of fluoride bound in some chemical form. Thus it was necessary to study possible adverse health effects due to ingestion of fluoride from several sources.

A report (c) issued in 1935 compared health effects primarily from calcium fluoride, sodium fluoride, and rock phosphate. Highly significant for present purposes was one small experiment that included sodium fluosilicate. With equal dosage and equal amounts of fluoride retained, rats fed sodium fluosilicate excreted three times as much non-retained fluoride in urine as rats fed sodium fluoride, who eliminated more fluoride in feces. Apparently about three times as much fluoride had crossed the gut/blood membrane into the bloodstream from SiF than from NaF. A second report, this one by the US PHS, (d) was published about ten years after water fluoridation had begun. The study compared the time, starting from the date of fluoridation either with sodium fluosilicate or sodium fluoride, for urinary fluoride level to reach equilibrium with ingested fluoride from fluoridated water. The study populations were boys and men. There were two noteworthy results. First, for either fluoridating agent, urine fluoride levels in older males reached equilibrium with ingested fluoride levels sooner than in younger males. The longer time for young males can be accounted for by the fact that the weight of the older males was essentially constant, while the younger males were adding bone mass over the several years of the experiment. The bodies of younger males were therefore providing a time-related increase in storage compartment capacity for ingested fluoride.

A more important finding was that for the younger males it took longer for their urine level of fluoride to reach equilibrium with ingested water fluoride from SiF than from NaF. Apparently in growing boys SiF fluoride must have been metabolizing differently from NaF fluoride.

A third relevant study (e), conducted around the same time as Westendorf’s research, involved feeding water treated with the same fluosilicic acid used to fluoridate the local water supply to squirrel monkeys for up to 14 months. Morphological and cytochemical effects were reported for the liver, kidney, and nervous system due to ingestion of 1-5 ppm of fluoride in water. Although the study did not compare results from exposure to NaF, the report emphasizes the fact that the kidneys of monkeys ingesting SiF treated drinking water “Éshowed significant cytochemical changes, especially in the animals on 5 PPM fluoride intake in their drinking water.”

The report later observes that work by others in the 1940s and 1950s “Éshowed that fluoride has an inhibitive effect on the activity of succinate dehydrogenase. These studies indicate that under the effect of fluoride intake, a serious metabolic distress may develop in the kidneys.” In concluding, the report notes that “Earlier, some workers had also indicated that inorganic fluorides have a strongly adverse effect on the activity of some enzymes and of these, mitochondrial enzymes, acid and alkaline phosphatases and ATP-utilizing enzymes and aldolase may be the most affected (Batenburg & Van den Bergh, 1972; Katz & Tenenhouse, 1973).”

This study of squirrel monkeys is a rare (possibly singular) American experiment with SiF. If the research team had known that Westendorf was finding greater effects of silicofluoride than sodium fluoride on enzyme activity at virtually the same moment, the U.S. study might have taken a different turn. In any case, two of these three American experiments compared effects from NaF and SiF, and both found that SiF and NaF do not produce the same effect. Moreover, all three studies found the strongest adverse clinical effect of silicofluoride in the kidney. But damage to the kidney is hardly the only possible health effect of ingested SiF.

“Life” involves an incalculable number of chemically active molecules initiating, continuing and terminating a bewildering variety of chemical events. Throughout this panoply of events and in every organ where they occur, various enzymes play crucial roles. A particularly important example is the quenching by enzymes of muscle stimulation induced by the neurotransmitter acetylcholine (ACh), an ester comprising the acetyl moiety bound by an oxygen bridge to the choline molecule. The principal “quenching” enzyme, acetylycholinesterase (AChE), comes in several variations and the ACh/Ache dyads operate in numerous ways in many organs. Related enzymes called pseudocholinesterases are found in serum and include the butyrylcholinesterases.

At latest count over 7,000 enzymes have been detected and catalogued, (f) and there is no reason to suppose that the effect of SiF is limited only to a sub-class. In any event, one would be hard put to identify a more important enzyme subclass than “esterases,” which cleave molecules called “esters” at the right time and place in the healthy organism. While a great deal is known about many of the ways these enzymes function, there are still large knowledge gaps to be filled. To do just that, an extensive survey of contemporary knowledge about cholinesterases has recently been published (g) by an employee of the Office of Prevention, Pesticides and Toxic Substances in EPA’s Health Effects Division. The published article carries this disclaimer:

“Although this article was written as part of the author’s official duties as an EPA scientist, the opinions and conclusions expressed in it are his alone, and do not reflect the position of the Environmental Protection Agency.”

Dementi’s review deserves a great deal of attention, so one wonders why it was not published as official work of the EPA. The EPA has acknowledged (h) that it has no data on health effects of the SiFs, shown by Westendorf to be a significant cholinesterase inhibitor and being added to the diets of 140 million people at the rate of 200,000 tons a year. The many different biochemical responses this dosage can be expected to elicit may well support a recently published (l) hypothesis proposing an explanation for Fibromyalgia, Multiple Chemical Sensitivity, and Chronic Fatigue Syndrome. It is not at all unlikely that chronic ingestion of SiF treated water also bears on ADD/ADHD, teen violence, and even some of the ambiguities associated with Gulf War Syndrome.

Common sense suggests that wide-spread, albeit clinically vague, adverse health effects should be expected when a strong enzyme inhibitor is added to the daily diets of over half of US residents, as would be the case given the results of the research work described herein. With millions of people suffering from one or another poorly understood condition with likely roots in environmental toxins, it is time to re-examine entrenched governmental doctrines in the light of Westendorf’s research which, while 30 years old, has received little or no attention heretofore.

(Read Westendorf’s thesis)

Notes and Credits

NOTE 1. The following English language text, translated from the German in which it was written by Dr. Johannes Westendorf, (Toxicology Department, Eppendorf-Hamburg University Hospital) was submitted to him in March 2001 for his comments with a series of questions. This was his response.

“With respect to my thesis I finished this kind of work in 1976, when I changed to the Medical faculty, where I still am. After my thesis I continued the work on fluoride for another year and we especially worked on the stability of hexafluoro complexes of silicon and iron. We used radioactive isotopes, such as F-18 and Si-31 . . . when we analyzed the electrophoretic mobility. In the presence of silicon and iron, fluoride ions showed a different mobility compared to fluoride [ion] itself. Unfortunately I have no access to these old experiments and we did not publish it.

. . . During hydrolysis we got a continuous shifting of the mobility, indicating that the different forms of hydrolysis with 2-6 fluorine at the Si are present at the same time, ending up at the more stable form of Si(OH)4F2. If we increased the pH to 9 and higher, a total hydrolysis occurs.

…In answering your final paragraph I can say:

1) The English translation of my thesis is excellent.

2) I have no evidence from others that contradict to my old findings.

3) Your idea of the enzyme inhibition by the complex could be right, however slight changes in the pH, caused by the hydrolysis of hexafluorosilicate, would also result in an increased inhibition of acetylcholinesterase. Nevertheless, I agree with you that the toxicology of hexafluorosilicate should be investigated because it may be different from simple fluoride.

Please let me know if I can be of further assistance to you. Johannes Westendorf” Westendorf@uke.uni-hamburg.de

NOTE II. Although the main body of the Westendorf thesis was not published in a circulating journal as such, three short articles based on this work were. Copies of the two most relevant ones appear at the end of the English text of the full thesis.

CREDITS: The thesis was called to our attention and photocopied from the document on file in the archives at the University of Hamburg by Peter Meiers (Weissenburgerstr. 28, D-66113 Saarbrucken; the translation was prepared by Jakob von Moltke (Dartmouth College); final proof editing was done by Myron Coplan with the aid of Norman Mancuso.

References:

a) Smith, PA. “History of Fluorine Recovery Processes”: Paper delivered at the IFA Technical Sub-Committee and Committee Meeting in Novgorord, Russia; Sept 15-17, 1999 (http://www.fertilizer.org/ifa/publicat/techpprs/tech0999.asp)

b) Gutierrez, SB. (signed by Thurnau RC); Letter from the Director of the US EPA National Risk Management Laboratory to Roger D. Masters, dated March 15, 2001.

c) Kick CH, et al. “Fluorine in Animal Nutrition”; Bulletin 558, Ohio Agricultural Experiment Station; Wooster, Ohio; November 1935; pp 1-77.

d) Zipkin, I et al. “Urinary Fluoride Levels Associated with Use of Fluoridated Water”; Pub Hlth Rpts 71 PP 767-772; 1956.

e) Manocha SL, et al. “Cytochemical response of kidney, liver and nervous system to fluoride ions in drinking water”; Histochemical Journal, 7 (1975); 343-355.

f) On February 7, 2001, the Brookhaven Registry of Enzymes listed 7,164 enzymes on their web-site, http://www.biochem.ucl.ac.uk/bsm/enzymes/

g) Dementi, B. “Cholinesterase Literature Review and Comment”; Pesticides, People and Nature; 1 (2); 59-126; 1999.

h) Letter to the Honorable Ken Calvert, Chairman of the Subcommittee on Energy and the Environment, US House Committee on Science, from EPA Assistant Administrator J. Charles Fox, June 23, 1999.

i) Laylander, J. “A Nutrient/Toxin Interaction Theory of the Etiology and Pathogenesis of Chronic Pain-Fatigue Syndromes: Parts I & II,” Journal of Chronic Fatigue Syndrome; 5(1), 67-126, 1999.

Synopsis of Foreword Authors’ Relevant Professional History

Roger D. Masters, Ph.D., is President of the Foundation for Neuroscience and Society and Nelson A. Rockefeller Professor of Government Emeritus at Dartmouth College. For the last 30 years, he has studied the implications of modern biological science in understanding human behavior. He serves as editor of the “Biology and Social Life” section of Social Science Information (an international journal published at the Maison des Sciences de l’Homme in Paris) and member of the Council of the Association for Politics and the Life Sciences. He is a published expert in the history of Renaissance politics, especially the contribution of Niccolo Machiavelli.

After undergraduate studies at Harvard (where his instructors included Henry Kissinger), he served in the US Army before graduate studies at the University of Chicago. Despite his work in other areas, he retained a strong professional interest in military and international affairs. In addition to writing The Nation is Burdened: American Foreign Policy in a Changing World (Knopf, 1967), he served as US Cultural Attache to France. Among his many other books are The Political Philosophy of Rousseau (Princeton, 1968), The Nature of Politics (Yale, 1989), Machiavelli, Leonardo, and the Science of Power (Notre Dame Press, 1996) and Fortune is a River: Leonardo da Vinci and Niccolo Machiavelli’s Magnificent Dream to Change the Course of Florentine History (Free Press, 1998). Before turning to issues of environmental pollution, health and behavior, he also published widely on the effectiveness of leaders’ nonverbal behavior on television (working with colleagues on experiments in France and Germany as well as in the US).

Among many other publications on biological factors in human behavior, he was co-editor (with Michael T McGuire) of The Neurotransmitter Revolution, Serotonin, Social Behavior and the Law (Southern Illinois University Press, 1994); senior author (with Brian Hone and Anil Doshi) of “Environmental Pollution, Neurotoxicity, and Criminal Violence,” in J. Rose, ed., Aspects of Environmental Toxicity (London: Gordon & Breach, 1998), pp. 13-45; and co-author (with MJ Coplan) of “Water Treatment with Silicofluorides and Lead Toxicity,” International Journal of Environmental Studies, 56: 435-449 (July-August 1999) as well as of other publications.

In addition to an earlier teaching position in political science at Yale, he served as US Cultural Attache to France, Fellow of the Hastings Center, Chair of the Executive Committee of the Gruter Institute for Law and Behavioral Research (a foundation specialized in linking biology to the study and practice of law), a visiting professor at Yale Law School and Vermont Law School, and a consultant to Upjohn Corp, to the Commissioner of Corrections of Vermont, and to several agencies of the Federal Government. As a result of these varied professional activities, Dr. Masters has had extensive experience applying new scientific research in biology of human behavior to the establishment of successful government policies.

Myron J. Coplan, PE is a consultant in chemical engineering and chemical sciences, doing business at “Intellequity” after retirement in 1987 as Vice President and General Manager of the Albany International Co. Membrane Development Venture. The fruits of this latter activity include a product line of membranes now used by a major multi-national company to supply a market for industrial gases measured in the $ billions.

Coplan’s working career started during WWII first as a civilian employee of the US War Department and then as a production chemist for a firm supplying the military with two crucial commodities: DDT, without which the S. Pacific campaign might not have been successful, and a wire insulating chemical, without which the US Navy’s capacity to deal with disastrous convoy damage by Nazi mines might not have been achieved. He was one of the few civilians deferred throughout WWII for his critical occupation status.

Post WWII, while pursuing his own advanced degree studies, Coplan headed an academic chemical engineering department, supervising doctoral research of others. This was followed by a 37-year relationship with an independent consulting and r/d firm specializing in material sciences (chemistry, polymer systems, statistical analysis, physics, fluid dynamics, statistical mechanics, etc.) which eventually became the central research laboratory of a large multinational corporation.

Coplan is recognized in American Men of Science, holds 32 patents, is a member of several professional organizations and has published many technical papers. He authored a series of bench-mark articles on mathematical probability statistics and wrote a manual on statistical quality control for internal corporate use. He also personally carried out a wide range of laboratory research and engineering tasks and supervised the work of as many as 35 other professionals of many disciplines. He has been consulted by research staffs and corporate executives from some of the world’s largest corporations. To mention only one example, over about ten years he had 28 assignments from GE.

His services were also engaged by NASA, USDA, EPA, Interior Dept, Post Office Dept and several other government agencies, including virtually every branch of the DOD. In these assignments, Coplan was cleared on a “need-to-know” high level security basis several times for consulting and research work in such diverse fields as “decoy” chaff used to frustrate radar-tracked anti-aircraft fire to protective measures for ground-troops at risk of exposure to chemical, biological and nuclear attack.

In due course, Coplan’s activities became more focused on the interests of the large company which in 1972 had acquired the firm he had joined in 1951. After 1972, he took on the corporate mission of identifying and exploiting science-based new business opportunities, including direct management of scientific entrepreneurial r/d for new products and technologies. He became Senior Corporate Scientist and then Vice President and General Manager of a membrane development venture that eventually licensed his patented inventions to other large corporations. Membrane treatment of phosphate waste pond waters was among the applications studied. Coplan, therefore, has first-hand knowledge of the processes from which the principal water fluoridating agents (the silicofluorides) are derived.

Full research paper → HERE  

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Two chemicals H2SiF6 and Na2SiF6, jointly called “silicofluorides”
are used to treat public water supplies of 140 million Americans even
though, the EPA has admitted, they have never been tested for safety.

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www.dartmouth.edu/~rmasters

Roger D. Masters – Research Professor

-Nelson A. Rockefeller Professor of Government, Emeritus Dartmouth College

Abstract:

Toxic metals like lead, manganese, copper and cadmium damage neurons
and deregulate neurotransmitters like serotonin and dopamine
which are essential to normal impulse control and learning.

Earlier studies show that — controlling for socio-economic and demographic factors — environmental pollution with lead is a highly significant risk factor in predicting higher rates of crime, attention deficit disorder or hyperactivity, and learning disabilities.

Exposure and uptake of lead has been associated with industrial pollution, leaded paint and plumbing systems in old housing, lead residues in soil, dietary habits (such as shortages of calcium and iron), and demographic factors (such as poverty, stress, and minority ethnicity). We report here on an additional “risk co-factor” making lead and other toxic metals in the environment more dangerous to local residents: the use of silicofluorides as agents in water treatment. The two chemicals in question — fluosilicic acid and sodium silicofluoride — are toxins that, despite claims to the contrary, do not dissociate completely and change water chemistry when used under normal water treatment practices. As a result, water treatment with siliconfluorides apparently functions to increase the cellular uptake of lead. Data from lead screening of over 280,000 children in Massachusetts indicates that silicofluoride usage is associated with significant increases in average lead in children’s blood as well as percentage of children with blood lead in excess of 10μg/dL. Consistent with the hypothesized role of silicofluorides as enhancing uptake of lead whatever the source of exposure, children are especially at risk for higher blood lead in those communities with more old housing or lead in excess of 15 ppb in first draw water samples where silicofluorides are also in use. Preliminary findings from county-level data in Georgia confirm that silicofluoride usage is associated with higher levels of lead in children’s blood. In both Massachusetts and Georgia, moreover, behaviors associated with lead nurotoxicity are more frequent in communities using silicofluorides than in comparable localities that do not use these chemicals. Because there has been insufficient animal or human testing of silicofluoride treated water, further study of the effect of silicofluorides is needed to clarify the extent to which these chemicals are risk co-factors for lead uptake and the hazardous effects it produces.

Keywords: Lead; toxicity; pollution; children’s health; public water supplies

-.-.-.-.-.-.-.-.-.-.-.-

A Primer for Policymakers and Public Policy Advocates
(Westport: Praeger, 2003), pp. 23-56.

ABSTRACT:

It is impossible to deny that a revolution in neuroscience and other areas of biology has taken place over the last half-century.  The  estimates of 83 million Americans taking drugs like Prozac for depression and 11 million children on Ritalin for hyperactivity indicate it is time to reconsider the role of brain chemistry in social behavior and violent behavior.  Since it is obvious that loss of impulse control can contribute to violent outbursts – and evidence shows that some toxic chemicals (such as lead) can have this effect, it is time to consider neuroscientific evidence linking environmental toxins and rates of violent behavior.  To illustrate the implications of the new issues involved, I focus on a hitherto unexplored example.  Two chemicals (H2SiF6 and Na2SiF6, jointly called “silicofluorides” or SiFs) are used to treat public water supplies of 140 million Americans even though, as the EPA has admitted, they never been tested for safety.  To illustrate the interdisciplinary complexities entailed when linking brain chemistry to policy decisions concerning violent crime, our argument has four main stages: first, why might SiFs be dangerous? Second, what biochemical effects of SiF could have toxic consequences for humans?  Third, on this basis a research hypothesis is formulated to measure the types of harm.  In this case, we predict children in communities using SiF should have increased uptake of lead from environmental sources and higher rates of behavioral dysfunctions such as hyperactivity (ADHD) known to be caused by lead neurotoxicity.  Finally, the hypothesis is tested using multiple sources of data including rates of violent crime studied using a variety of multivariate statistical techniques (including analysis of variance, multiple regression, and stepwise regression).  As this outline should make clear, a combination of interdisciplinary perspectives and great prudence is needed to link research in neuroscience to policies concerning violent crime,  If confirmed, however, the potential benefits of hypotheses like the one tested below may be great, revealing the generally unsuspected value of including neuroscientific research in the analysis of human social behavior.

Requests for reprints and correspondence should be directed to: Prof. Roger D. Masters, Department of Government, HB 6222, Dartmouth College, Hanover, NH 03755. Email: Roger.D.Masters@Dartmouth.edu

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The full version of the above is on the net,
and as the above suggests adding silicofluorides
to drinking water amounts to domestic terrorism.

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see also: http://www.waterloowatch.com/hydrofluorosilicic%20acid.html

see also:  www.navigatingtheaether.com/…/the-dangers-of-fluoride-conspiracy..

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~  A MOTION CALLING FOR A BAN ON FLUORIDATION  ~

THE GREEN PARTY OF CANADA + Australia

Green-party-of-Canada-Sy.-s

Subcategory: Health and Environment

Code: G10-p19

     Toronto Danforth EDA, Davenport EDA, Vaughan EDA, York University Greens, Frank De Jong, Tom Eric, Halvorsen Jobi, Mike Kenny, Claudia Rodriguez-Larrain, Ingrid Sheriff, Constantine Kritsonis, Mike Dewar, Marie La Plume, Fay Neuber, Stefan Premdas, Bruce Hearns, Matt Turner

BE IT RESOLVED that the Green Party seeks to introduce and support legislation that will ban artificial fluoridation products in public drinking water.

Motion Operative:

WHEREAS the Green Party affirms that the precautionary principle should be used where there is evidence of potential harm in the absence of complete scientific consensus.

WHEREAS no statistical difference exists in rates of dental caries between areas that use artificial water fluoridation chemicals and those that do not (Statistics Canada);

WHEREAS fluoride is not removed in sewage treatment and remains a toxic constituent of the effluent discharged by treatment plants to rivers and lakes, and background levels of fluoride in the Great Lakes exceed the Canadian Water Quality Guideline (CWQG) for aquatic species, and fluoride concentrations in sewage effluent are 5-10 times in excess of the CWQG. At these concentrations fluoride is known to be toxic to a variety of water species such as salmon, caddisfly, daphnia & others;

WHEREAS the use of drinking water to deliver medications is not medically or environmentally sustainable, and the use of unregulated, unapproved medications in drinking water, without a prescription or informed consent is medically and environmentally not sustainable, and the daily dose of fluoride cannot be controlled and health effects from fluoride are not monitored;

Green Party of Canada website 22 August 2010 an area that has traditionally been within the jurisdiction of the provinces.

WHEREAS 97 percent of Europe does not use artificial water fluoridation products in their public drinking water supply, and 94 percent of the world’s population does not use artificial water fluoridation products in their public water supply;

Motion Preamble:

WHEREAS fluoridation products such as hexafluorosilicic acid and sodium silicofluoride are toxic by-products scrubbed from the smokestacks of the phosphate mining industry, and less than one percent of treated water is actually ingested by people and the remaining 99 percent is discharged into the environment…

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~  THE AUSTRALIAN GREENS ~

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The Queensland Greens:

Extract from their policy document 

36. Ensure fluoridation of potable water, to be funded by the state government.

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The Australian Greens are divided about science and when
it comes to fluoridation, or, far worse, vaccination; and
a large majority are firmly against all forms of 
Genetic Engineering, which is a big problem 
for The Greens Party of Australia.

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It is a conflict of policy to be
pro-fluoridation and anti-nuclear!
–  We at ‘Fluoridation Queensland’ are
anti-fluoridation & anti-nuclear.-

Loyalty to Medical…Greens ff

This split could be the end of Australian Greens! 

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Some one needs to explain to me why wanting clean drinking water
makes you an activist, and why proposing to destroy water with
chemical warfare doesn’t make a corporation a terrorist.”
Winona LaDuke
[She ran for vice president as the nominee of the
Green Party of the United States, on a ticket headed by Ralph Nader.]

There is no fluoridation in Wales, Scotland or Northern Ireland

See Film Below 3

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 Ralph  Nader  interview 
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The laws of nature cannot be reversed to suit pleasure,
profit or politics. 
It matters little how many millions
of dollars are spent promoting 
this absurdity,
it will not change the facts. – O
ne part per
million, is
 a medical dose; and it is
a guide to industrial polluters.

'No toxic F. no to anna f

Our Letter #1 of 4 sent to Anna Bligh
Copies sent to her electoral office and home address.

A BLIGH'S legacy

   14/12/2007

Dear Ms. Bligh,

Re your proposal to fluoridate Queensland’s Water Supply:

We are ‘The Smart State’ for not having artificially fluoridated water.

Queensland is at the moment in step with most of the world. See attachment ‘A’ ‘State of the Nations’ + ‘Tooth Decay Trends’

The majority of countries, either, avoided it, have banned it, or have ceased to fluoridate. In parts of India and China where it occurs naturally, it is removed from the water used for drinking.

Are you aware of the political history of fluoridation in our state?

The Gold Coast City Council stopped fluoridating its water supply in 1979 due to public pressure. See attachment ‘B’

Lord Mayor Atkinson reversed her original decision to fluoridate Brisbane. See attachment ‘C’

Dr. Lew Edwards lost his safe seat due to public pressure because of his commitment to fluoridate the State of Queensland.

In 1952 The Queensland Government funded research into the health of Qld. sheep drinking naturally fluoridated bore water. See attachment ‘D’ ‘Chronic Endemic Fluorosis of Merino Sheep in Queensland by J.M. Harvey’

Does your government know what the levels of hydrogen fluoride from pollution are in the air we breathe?

Page 2 of 2

Does the Queensland Government monitor fluoride air pollution?

Do you know which industries pollute with fluoride?

Do you know how much natural fluoride is in our drinking water at the moment?

Are you aware that natural fluoride CaF2 is different from the chemicals used to artificially fluoridate water? See attachments ‘E’ and ‘F’

What are your proposals for recycled water? Are you going to

re-fluoridate it?

If you put recycled water through Reverse Osmosis equipment, which removes fluoride, what are you going to do with the redeemed toxic fluoride sludge?

If you store fluoridated water in the dams you risk fish kills. Fresh water fish, bees, turkeys, some plants and animals are very sensitive to fluoride pollution.

One part per million, is not a medical dose; it is a guide to industrial polluters.

Dentists have been lobbied with endorsements, not scientific research. Long-term health problems have been ignored.

See attachments ‘G, H, I,’ and ‘J’.

Fluoridation is deliberate pollution. See attachment ‘K’

What safe guards are you going to put into place to protect people who are super sensitive to fluorides?

See attachment ‘L’ Death Certificate Brisbane Mater Hospital for Jason Burton – death by Fluoride poisoning + statement by mother and newspaper cutting.

You have a duty of care, a responsibility to every person in the state.

See attachment ‘M’ Informed Consent – Nuremberg Code

On the matter of water fluoridation we are directing you to do nothing. The people of Queensland want clean water not contaminated water.

Keep Queensland ‘The Smart State’ by not having artificially fluoridated water.

We look forward to hearing from you very soon.

Yours truly,

Chairman Brisbane Anti-Fluoridation Association

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[Gold Coast Commenced Fluoridation 5 Nov. 1968]

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Our Letter #2 of 4 sent to Anna Bligh.

Copies sent to her electoral office and home address.

Bligh #3 m

cc- our web, opposition, minister for health

   04/07/2008

Dear Ms. Bligh,

While you were in China and India recently, did you enquire as to the state of the natural contamination of Fluoride (CaF2) in the water in those two countries?  See our previous state of the nations report.

Ottawa, Canada removed Fluoride from its water last month.

Previous communications from your Department of Health have not addresses our questions.  By not answering them are they deceiving us?

Promotion by endorsement is not science and this is all we have received. History has shown this; doctors used to promote cigarettes! The FDA has just withdrawn its support for mercury in dental fillings.  See below.

The law you have passed on fluoridation does not permit you to add the heavy metals that are also in this industrial waste Hydrofluorosilicic acid, which contains up to 30 components including heavy metals. (See analysis below).  It is the acid spray waste from the chimney pollution scrubbers, mainly from the fertiliser industry. Fluoride is usually 20% – 38% of the total compound.

This industrial waste is not safe for human consumption.

TYPICAL ANALYSIS:

Note: acceptable safe levels for many heavy metals is zero

Nickel                              1742  ppm

Aluminium                        2.1  ppm

Cadmium                           4   ppb

Arsenic                       4826   ppb

Mercury                             5   ppb

Chromium                3763   ppb

Lead                                 15   ppb

Fluoride is toxic and corrosive. It rates ‘four’ on a one to five scale – more toxic than lead, slightly less toxic than arsenic.

While the uranium and radium in fluorosilicic acid are known carcinogens, two other products of uranium, even more carcinogenic are radon-222 and polonium-210, these are also present.

It is illegal to release fluoride into waterways or the ocean.

It seems to us, that someone needs to do some homework.

You have been ill advised on this mater, your advisors seem to know very little about poisons, human health, and eco-systems.

The laws of nature cannot be reversed to suit pleasure, profit or politics.

It matters little how many millions of dollars are spent promoting this absurdity, it will not change the facts.

Citizens have an expectation that their government will supply them with clean drinking water. We certainly do not expect it to be deliberately contaminated or medicated.

People living in units cannot install tanks. Reverses osmosis filters are expensive, and waste a lot of water. The city water supply system must not be used as a medicine bottle and certainly not used for pollution dispersal.

If you and your family drink fluoridated water, given the information we have provided to you, then you will be unwise. However, if you do not drink the fluoridated city water then you will be guilty of double-standards.

No one goes through life without making mistakes, and often the mistake cannot be remedied.  Fluoridation is a mistake and you can correct it.

The American Kidney Foundation has recently withdrawn its support from sodium fluoride in the water and is urging other medical bodies to do likewise. The health of 4,000,000 Queenslanders is at stake. This was bad science and a bad policy from the beginning, we urge you change positions, and act quickly to stop fluoridation.

We look forward to hearing from you very soon.

Yours truly,

Chairperson – Brisbane Anti-Fluoridation Association

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Our Letter #3 of 4 sent to Anna Bligh

Copies sent to her electoral office and home address.

Bligh 1 m

   14/09/2008

Dear Ms. Bligh,

Many of your electors will forgive you and “your”

government for problems you have inherited from

previous governments. But as you are solely

responsible for the project for state-wide water

fluoridation, on this matter it will not be so.

We have attempted in the past to alert you to

this error, which you seem to so far have chosen to ignore.

A little sole searching and goggling should have rung alarm bells for you. Fluoridation is at

best, a nineteenth century dream, at worse, an oppressive dangerous hoax. Considering

your University background we are surprised that you would condone such injustice –

As we stated previously, ‘Citizens have an expectation that their government will supply

them with clean drinking water. We certainly do not expect it to be deliberately

contaminated or medicated.’

It now seems that we will not have a “Smart State” or a “Fair State”

or one lead by an elected female premier.

Our organization will now actively work against your political future as we did for

Dr. Lew Edwards who attempted to fluoridate the State of Queensland (1975), and several

other politicians who originally put fluoride into the Gold Coast water supply many years

ago (ceased July 1979).

Even if you do install fluoride plants in various water systems throughout the state, they

will be removed in the future.

Yours truly,

Chairperson

Brisbane Anti-Fluoridation Association

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Our Letter #4 of 4 sent to Anna Bligh.

Copies sent to her electoral office and home address.

Bligh 4 m

   01/06/2009

We,

The Brisbane Anti-fluoridation Association

wish to draw your attention to two urgent problems:

1.

We are concerned that you may not have tested the eighteen bores used to augment Toowoomba’s water supply for calcium fluoride – CaF2.F. levels.

We are asking for the F. levels from each of these bores please.

The attached maps, charts and photos (9 Pages total), from research carried out by the Queensland Government: (J.M. Harvey Chronic Endemic Fluorosis of Merino Sheep in Queensland. Q J Ag Sci 1952;9:1-88.) – These charts illustrate, calcium fluoride is present in Queensland’s underground water, and some of the illustrations associated its problems.

2.

Some of Queensland’s aboriginal settlements also use underground water and it needs testing for levels of calcium fluoride before you undertake artificially fluoridation programs of their water supplies.

We request these figures too please.

We wish to further remind you that indigenous people have smaller kidneys – a reduced number of nephrons and decreased renal reserve – (Singh G, White A, Spencer J, Wang Z, Hoy W [1999]).

Kidney health problems are more common for indigenous people than for non-indigenous peopleand they are therefore more at risk from fluorosis and other diseases associated with fluorides.

(The National Kidney Foundation (USA) no longer endorses Water Fluoridation.)

See two enclosures

We look forward to hearing from you within 30 days with proof of your action on these two issues.

Yours truly,

Chairperson

Brisbane Anti-Fluoridation Association

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(Several thousand petition signatures were faxed to the premier’s office.)
Due to the lack of an honest response from Premier Anna Bligh
to our letters 1 – 4 etc., We are left with an unhappy
choice between her being dishonest
or incompetent, – or both.

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Anna B's Guarantee copy 2

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Bligh Puts House on The Line for Fluoride

By Andrew Wright 12 February 2009

Anna Bligh has signed a personal guarantee that could cost the Premier her personal assets in a court battle if people become sick from water fluoridation, a Brisbane lawyer said.

This week, Ms Bligh confirmed she had signed a legal document in which she accepts full personal liability for fluoridation and promises to provide financial compensation in the event that it causes adverse health affects.

Fluoride was introduced into the state’s water supply in December.  At the time, Health Minister Stephen Robertson was presented with two petitions totaling 6000 signatures against the move, which will see a final fluoride concentration of 0.6 parts per million.

The document, which Ms Bligh signed on October 15 at a community meeting, was first published in the Hinterland Voice independent newspaper.

“The Premier signed the document as a demonstration of her commitment to fluoridation,” the Premier’s spokeswoman said.

” She did not make this decision lightly and stands by it.”

But the promise could provide a loophole for anti-fluoridation activists to take legal action over the rollout of fluoride.

Mark O’Connor, a compensation specialist and partner at Brisbane law firm Bennett & Philp, said state laws prevent residents from taking any action against water authorities.

However, he said the Premier’s pledge means she could be liable as a private citizen, although it would be difficult to establish a case against her.

“You’d have to provide medical evidence the condition has been caused by consumption of fluoridated water and not another factor,” Mr. O’Connor explained.

He said anyone bringing a case against Ms Bligh would have to show they had changed their behavior on the basis of the promise.

“It’s an unusual case. I’ve never seen this done before,” he said.

“It would appear she is giving a personal guarantee, rather than a guarantee on behalf of the Government.

“Any claim would therefore be against Ms Bligh’s own assets.

“I doubt she would put her assets at risk, unless she was sure that fluoride wouldn’t cause disease.”

At an anti-fluoridation rally on Tuesday, Queenslanders Against Water Fluoridation spokeswoman Jeanie Ryan said the group was still calling on Ms Bligh to end fluoridation because of its health effects.

An article published in the respected medical journal Lancet said fluoride can cause nurotoxicity in laboratory animals, but hasn’t yet been proven to be toxic to humans.

Studies in rural communities in China have found high fluoride concentrations in well water may cause skeletal abnormalities and affect intelligence.

Australian Dental Association Queensland branch president, Rockhampton dentist Greg Moore, said there could be a 20-40 per cent reduction in tooth decay as a result of fluoridated water.

Rockhampton has now stopped fluoridation.   And  →  HERE

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1994 In November 1994 in Australia, the Victorian Parliament passes an

amendment to the “Fluoridation Act” to change the constitution in order to

stop the Supreme Court of Victoria from hearing any public cases or

evidence against artificial fluoridation.

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1995 In Australia, the Tasmanian Government passes a bill to prohibit the

holding of meetings on the subject of fluoridation. Called the

“Consequential Amendments Bill”, it was later withdrawn for rewording and

reissue at a later date…

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We can do better

One year later:

“No explanation from Queensland Health for falsified fluoridation data:

… It is exactly one year now since the Crime and Misconduct Commission

advised that Queensland Health would respond in “due course” in regard

to evidence presented to the CMC that Queensland Health had falsified

tooth decay data used by Premier Bligh to justify forced fluoridation...

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Ross-Fitzgerald

He is the author of 33 books.

“Premier in line for more water torture”

Ross Fitzgerald  2 December 2008

Article from:image of the australian

     QUEENSLAND Premier Anna Bligh’s spectacular back-downs on recycled water and on the controversial Traveston dam project will not save her at the next state election. Not unless she shelves her plan to add fluoride to the drinking water.

Those who think the introduction of fluoride is a minor issue should think again. It was, after all, a highway through koala habitat in southeast Queensland that ended the government of Wayne Goss.

Fluoride will be added to Queensland drinking water just before the new year break. The problem for Bligh is that the citizens who vociferously rally against fluoridated water are part of the same group that was instrumental in denouncing recycled water. It’s issues such as these, where governments run roughshod over the electorate, that really bite.

Bligh claims that most Queenslanders are in favour of fluoridation, but there is widespread scepticism, particularly among farmers, who have known for many years that some bore waters high in natural fluoride have a detrimental effect on the health of livestock.

Chronic fluorine toxicity results from continuous consumption of fluorine while the sheep are young and teeth and bones are growing. The teeth become chalky white, mottled and pitted. The bone of the lower jaw thickens and bony outgrowths may develop.

In some instances this can lead to lameness and fractures.

Selling the message that fluoride is good for human consumption is a hard task in country Queensland, especially as the Department of Primary Industries warns against a consumption of 2mg a litre for sheep. This amount would be drunk by a farmer in a normal day’s work if the drinking water had fluoride levels proposed by the Bligh Government of 0.8 to 0.9 parts per million.

During a visit to Queensland, Andrew Harms, past president of the South Australian branch of the Australian Dental Association, said the addition of fluoride to water in these mining towns would increase the uptake of lead by children and adults who already had gravely high lead levels in their blood samples.

City folk may be a different matter, especially as a substantial number of Queenslanders have migrated from NSW or Victoria. This group is seemingly content with the message that they have better teeth than their next-door neighbours. The problem is that not all Queenslanders agree with the message and that many citizens are against any form of mass medication in the water supply.

The Bligh mantra of “safe and effective” does not convince everyone and there is much evidence that contradicts the government line. In November 2006 the American Dental Association announced that baby formulas made up with fluoridated water should be avoided for infants younger than six months.

The Australian Dental Association and the National Health and Medical Research Council came on line with similar suggestions: babies six months to a year should have only about 600ml of fluoridated water, increasing slightly as the child grows.

Mistrust of government festers within a community that has started looking elsewhere for information. The Lancet medical journal and Scientific American put the cat among the pigeons with negative comments about water fluoridation and its effects on body systems.

Add to that data from the national survey of adult oral health (2004-06), published in 2007, which showed no difference in the dental health of Queenslanders and people in other states.

Some medical professionals have tried to stem the flow of indoctrination but have been ridiculed for their objections. A prime example is the highly respected Brisbane-based general practitioner John Ryan, who has postgraduate qualifications in nutrition and children’s diseases and in environmental medicine.

What irritates him is the failure of the Bligh Government to tell Queenslanders of the NHMCR fluoride warnings to mothers with babies. “Where is the Government’s duty of care?” he asks.

Ryan is angry that the Government would deceive the public about data from a much-publicised Townsville study. Oral health data was collected about children living in Brisbane (non-fluoridated) and Townsville (fluoridated). There was much publicity by the Government indicating a supposedly vast difference between the two cities. In fact, the study showed there was less that half a tooth difference.

This, Ryan says, is an ancient and poor quality study, on which the media indoctrination largely is based. “We were so shocked by five very significant untruths told to the public by the Government,” he says. As a consequence, opponents of introducing fluoride into the water supply took the matter to the Criminal Misconduct Commission. The CMC indicated it was not within its brief and referred the matter back to Queensland Health. Eight months later it has still not responded.

The primary aim of the new Queensland Safe Water Association is to inform metropolitan and country Queenslanders about the negative aspects of recycled and fluoridated water. The message is simple: the state Government is putting public health at risk. Adult Queenslanders do not have the worst teeth in Australia and babies should not be given fluoridated water.

The Bligh Government has estimated that about 30 per cent of the population is not in favour of water fluoridation, so you can bet the number is much higher. In the state election due next year, the Liberal National Party led by the urbane Lawrence Springborg, who is opposed to compulsory fluoride, may well ride to power on the back of this debacle.

Media Release 23 February 2008

NEWS ALERT

Qld Health staff doctored tooth decay data used in decision to force fluoridation.

This alert is to inform Queensland Members of Parliament and others, that Queensland Health staff have doctored tooth decay rates data used in the decision making process and the massive fluoridation advertising promotions.

A delegation to the Queensland Premier, Health Minister and Chief Health Officer on the 12th Feb made them aware of this situation, however it appears that this is being ignored by the Premier and that Legislation that has its basis in fraudulent data will be rammed through Parliament on Tuesday 26th Feb.

Premier Blighs announcement on 5th Dec that she had made the decision to force fluoridation was hilighted by comparison of tooth decay rates ( but using data only from baby teeth ) between Townsville and Brisbane and led into newspaper advertisements which stated “In Townsville, water supplies have been fluoridated since 1964, resulting in 65% less tooth decay in children than those in Brisbane” additionally “… fluoride is proven safe and effective”

Examination of the source of the data used reveals that Qld Health staff have changed results from “teeth surfaces” to “teeth” a four to five fold difference ( teeth each have 4 or 5 surfaces depending on the type of tooth) leading to a false and exaggerated impression of tooth decay favouring fluoridated Townsville.  Brisbane children were falsely shown as having  2 more decayed baby teeth than Townsville children, when in reality, averaged out, it would have been only a fraction of a tooth. The data from permanent teeth in children aged 6 to 12 years old which was available but not used by Qld Health, showed on average only a tiny fraction of a tooth difference in decay rates…

Queensland Health also portrayed that the data is from 1996 when in fact it is from mid 1991 to 1992. Queensland Health have refused to acknowledge tooth decay data in permanent teeth from recent Queensland Children’s Dental surveys and refused to acknowledge tooth decay data from children’s permanent teeth measured at 12 years of age which is the international standard of comparison. Recent Tooth decay data from permanent teeth shows that Queensland compares favourably to other Australian states.

There is no scientific evidence to show that water fluoridation results in 65 % less tooth decay as Queensland Health claimed. A major British Government commissioned review of water fluoridation done in 2000 by York University found that size of estimated benefit to be only of the order of 15% and additionally that the review did not show water fluoridation to be safe.

Queensland Health staff have illegimately dismissed creditable scientific publications showing links with harm from Osteoscarcoma and other adverse health effects and also recommendations from the Centre for Disease Control that infants under 12 months of age not consume fluoridated water.

Freedom of Information shows that a senior Oral Health advisor to the Health Minister had written in 2006 that there are no scientific studies to prove water fluoridation is safe.

Queensland Health would have spent millions promoting “safety“ of water fluoridation in newspaper, radio television advertising, even in posters in public toilets, yet the 2008 Water Fluoridation Bill assures the Queensland Government blanket protection from liability.

The Water Fluoridation Act 2008 will give ” protection from civil rights and remedies” to any person or corporation involved in water fluoridation. Unless Criminal negligence can be proved, no harmed person will ever be able to bring a case of harm against, or prosecute the State of Queensland, the Minister, any analyst, any official or any person authorized under the Fluoridation Act, or any Water Supplier.

It is contradictory for the Queensland Premier to claim water fluoridation is safe, and then introduce Legislation that forces fluoridation on the population but also prevents any people harmed by it (even by accident) from making a claim against the decision makers or any person authorized under the Act.

We urge the Members of the Parliament of Queensland to vote against this legislation which was based on and promoted by fraudulent data.

For further information contact www.qawf.org

info@qawf.org

Message authorised by Merilyn Haines

President

Queenslanders Against Water Fluoridation Inc.

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As can seen below fluoride, lead, and mercury have always been a
problem to human health and are a bad combination:

FLUORIDATION INCREASES LEAD ABSORPTION IN CHILDREN

The chemical most commonly used to fluoridate America’s drinking water is associated with an increase in children’s blood lead levels. Most studies that purport fluoridation’s safety and effectiveness in preventing cavities use the chemical sodium fluoride. However, most communities inject cheaper silicofluorides (fluosilicic acid and sodium silicofluoride) into their drinking water based on the theory that each chemical comes apart totally, so that freed fluoride can incorporate into tooth enamel.

However, the silicofluorides (SiF) do not separate completely, as sodium fluoride does, As a result, water treatment with silicofluorides apparently functions to increase the cellular uptake of lead.

In research published in the International Journal of Environmental Studies (September 1999), Masters and Coplan studied lead screening data from 280,000 Massachusetts children. They found that average blood lead levels are significantly higher in children living in communities whose water is treated with silicofluorides. Data from the Third National Health and Nutrition Evaluation Survey (NHANES III) and a survey of over 120,000 children in New York towns (population 15,000 to 75,000) corroborate this effect.

Masters and Coplan reported that some minorities are especially at risk in high SiF exposure areas, where Black and Mexican American children have significantly higher blood lead levels than they do in unfluoridated communities.

Children with higher blood lead levels also have more tooth decay

Lead poisoning can cause learning disabilities, behavioral problems, and at high levels, seizures, coma and even death, according to the U.S. Centers for Disease Control (CDC). Lead is a highly significant risk factor in predicting higher rates of crime, attention deficit disorder or hyperactivity and learning disabilities.

Fluoridation

…Future lawsuits on behalf of kidney patients…

“AWWA looks to the medical and dental communities, the U.S. EPA, the Centers for Disease Control, and other … organizations for their information and research on medical and dental health,” the statement said.

…Stockin said the association has ignored obvious conflicts between the research and glowing reports on the safety of fluorides and will likely face lawsuits on behalf of kidney patients and other groups. He suggests other organizations immediately rescind support of fluoride.

“This was bad science and bad policy from the beginning, and I would suggest cities and organizations that lend their name to supporting fluoridation very quickly and emphatically change positions,” he said. ‘Attorney Reeves’ letter should be a wake up call – to act quickly to stop fluoridation.”

Dental Mercury: FDA 6 June 2008

After years of asserting that mercury in fillings was safe, the Food and Drug Administration now says it may be harmful to pregnant women, children, fetuses, and people who are especially sensitive to mercury exposure.

“Dental amalgams contain mercury, which may have neurotoxic effects on the nervous systems of developing children and fetuses,” the FDA now says on its Web site.

The agency posted the revised assessment online Tuesday as part of a settlement with consumer advocates. The FDA also committed to issuing special controls on mercury fillings in July 2009.

Those controls could range from giving patients information to adding warnings prohibiting use of the fillings in some people.

While bans are unlikely, the FDA may conclude that the cavity-repair treatment, called dental amalgam, should not be used in certain patients such as pregnant women, FDA spokeswoman…

See also our posting: Fluoridation & Lead Contamination

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This web site may contain legal information but is presented with our policy of ‘A Citizen’s Right-To-Know’.  You are notified that dissemination of this information is encouraged. If you receive this transmission in error feel free to notify the authors or retain or make any copies you may need.

We reserve the right to publish any outgoing correspondence and incoming
responses or non responses on our
web sites as part of our policy of a
 ‘Citizen’s Right-To-Know’  .

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Pre-Anna Bligh Policy: 
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Deals done with ADA Qld.
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The Australian Fluoridation Skeptics

Are Working For A Fluoridation Free Australia.

We Wish To Thank All Our Team, Other Anti-Fluoride Groups

And The Public For The Support We Have Received So Far,

And Adam Lin, Who Administers This Web Site.

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  Cassowary  ↓
Casuarius casuarius johnsonii

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Fluoride air pollution can have a devastating effect on the total environment.
Over the past 50 years, a variety of industries have released into
the atmosphere more than 25,000,000 tons
of fluoride gases and particulates.

 The major industries with fluoride pollution problems include:

coal-burning power stations, petro-chemical refineries, aluminium,

zinc, copper, beryllium and magnesium producing factories, steel mills,

fertilizer works, plastics manufacturers, glass factories, cement works, 

pottery and tile makers, brick works, chemical factories and nuclear

processing plants. The most common and dangerous air pollutant

produced by these industries and many others, is —

National Emissions Inventry logo Hydrogen Fluoride

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Workers in the following occupations may be exposed to

Hydrogen Fluoride in the workplace air:

Aircraft workers,
 – Alkylation plant workers, – 
Alloy steel cleaners, – 
Alloy steel makers, – 
Aluminium fluoride makers, – 
Aluminium makers, – 
Beryllium workers, 
- Bleachers, – 
Brass cleaners, 
- Brewers, -
 Brick cleaners, – 
Brick makers, -
 Casting cleaners, – 
Cement workers, – 
Ceramic workers, -
 Chemists, – Copper cleaners, 
-  Cryolite makers, – 
Crystal glass polishers, – 
Dye makers, – 
Electric arc welders, – 
Electroplaters, 
Enamel etchers, – 
Fermentation workers, – 
Fertilizer makers, – Filter paper makers, 
- Fluoborate makers, 
Fluoride compound makers, – 
Fluorine makers, –  
Fluorocarbon makers, – 
Fluorochemical makers, 
-Fluosilicate makers, – Freon makers, – 
Genetron makers, 
- Glass etchers, 
- Graphite purifiers, – 
Hydrogen fluoride makers, – 
Isotron makers, 
- Laundry workers, – 
Metal cleaners, 
- Metal polishers, 
- Neon sign makers, 
- Oil well acidifiers, –  
Ore dissolvers, – 
Petrol refinery workers, 
- Phosphate rock workers, 
- Plastic makers, 
- Polish makers, 
- Pottery workers, 
- Power station workers, 
-  Quartz crystal makers, 
- Rocket fuel handlers, 
- Rocket fuel makers, 
- Silicon chip makers, – 
Stainless steel cleaners, – 
Stainless steel makers, 
-Steel casting picklers, 
- Steel millworkers, – Stone cleaners, – 
Tile makers, 
- Uranium refiners, 
- Yeast makers, and
 Zinc workers.

Fluoride air pollution can have a devastating effect on the total environment.

Angus Lazores is a Mohawk Indian. For centuries before the white-man reached Canada and the United States, the Mohawks hunted, fished, trapped, and farmed the islands of the Gulf of St. Lawrence, now known as the St. Regis Akwesasne Indian Reserve.

Angus Lazores, along with 1,500 other Mohawks, lives on Cornwall Island, a part of the reserve straddling the borders of Quebec, Ontario, and Upper New York State. The St. Regis Mohawk Band settled Cornwall Island just over a hundred years ago; they soon became known as an efficient and self-sufficient agricultural community. In 1959, there were 45 farmers, forty cattle barns and 364 dairy cattle on the Island.

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Twenty years later, only eight farmers and eight cattle barns were left.

During the twenty years the cattle population was decimated;

all the bees on the Island had disappeared; crop yields had fallen;

partridges, after which the Akwesasne Reserve is named, had declined drastically;

and the white pine trees on the Island were dying.

In 1959, Reynolds Metals Company had built an aluminium smelter on the south bank of the St. Lawrence River near Massena, New York State. Cornwall Island is downwind of the smelter at least 60 per cent of the time.

Angus Lazores dates his problems on the Island to 1962, just three years after the smelter became operational.

In that year, cattle became lame and developed swellings on their legs, eventually the lameness became so severe that the animals could no longer graze normally. They laid down to eat on pasture and then crawled to the next place to eat. With increasing age the cows had difficulty drinking cold water, and chewing was obviously painful. The animals would grab hay but let it go after unsuccessful attempts at mastication.

The first pregnancy and calving were usually uneventful, but the cows had small udders and too little milk for the calf. At the third pregnancy and delivery, the native cows had usually deteriorated, being unable to drink or chew properly. Cows died during delivery and neonatal calf mortality was high. If cows survived the third pregnancy they were sold for slaughter.

By 1971, the majority of farmers had switched from dairy to beef cattle and by November 1977, there were only 177 cattle on the Island compared with 364 in 1959.

The cause of the cattle disease was admitted only after many years. In 1969, officials of the Canadian Ministry of the Environment had expressed concern to Reynolds Metals about fluoride emissions impacting on the Island. Four years later, the St. Regis Local Council authorised an investigation into pollutants emitted by the smelter. In July 1973, the Council were advised that damage to the pine trees on the Island was due to fluoride gases.

Two years later, urine samples from Cornwall Island cattle showed abnormal levels of fluoride.

In November 1975, Angus Lazore’s cattle were examined by a veterinarian called Abbey, sent by Reynolds Metals. He claimed that internal and external parasites were responsible for the condition of the cattle – fluoride wasn’t even mentioned.

The Mohawk elders were disturbed by Abbey’s diagnosis and approached Professor Lennart Krook, an eminent veterinary scientist at Cornell University.

Krook ran extensive diagnostic and pathological tests on the St. Regis cattle, then announced his findings:

“Owing to extensive and serious chronic fluoride poisoning no cattle born on Cornwall Island were going to live for more than five years.”

During 1977 and 1978, the situation which had developed on the Island was investigated by a team of scientists from the New York State College of Veterinary Medicine, Cornell University. Leaders of the team were Professor Krook and Dr George Maylin. In the introduction to their published report, they point out:

“Of all pollutants that affect farm animals, fluorine has caused the most severe and widespread damage. The object of the present study is to record yet another man-made fluorine pollution disaster and to interpret the pathogenesis of the osseous changes in view of recent advances in the understanding of bone metabolism.”

While Krook and Maylin focused on the cattle, Dr Clancy Gordon of the University of Montana, examined 2,600 plant samples from Cornwall Island and found very high levels of fluoride in all the vegetation tested.

University of Illinois scientists were then recruited to see if the Islanders themselves were suffering health problems resulting from excessive exposure to fluoride. Doctors Bertram Carnow and Shirley Conibear reported:

“Significant numbers of people with abnormalities of the muscular, skeletal, nervous and blood systems.”

In addition, Cornwall Island physicians had noted high rates of anaemia, rashes, irritability, diabetes, high blood pressure and thyroid disease.

Carnow and Conibear concluded that there had been;

“Unquestionably heavy exposure to fluorine compounds that has affected all the life studied.”

They recommended an immediate reduction in smelter fluoride emissions. Chief Francis of the Mohawk Indian Band put it more dramatically, he advised anyone living in areas where smelters might be built, to:

“Block the project. Block them with everything you have. If you fail then move. Move as quickly as you can because there’s no money that can buy your health back.”

Reynolds Metals spent its first ten years of operation spewing over 130 kilos of fluoride emissions an hour, directly downstream to Cornwall Island. Even after New York State regulations forced the company to reduce its emissions to 30 kilos an hour by 1975, Reynolds’ “gift” to the Mohawks had been an appalling –

TWELVE MILLION KILOS OF AIRBORNE FLUORIDE CONTAMINANTS

OVER TWENTY YEARS.

The Mohawk way of life became a victim of a preventable man-made plague. And you don’t have to go to Canada to find fluoride pollution problems. For more than a century, the Hunter Valley Region of New South Wales has produced some of Australia’s finest wines.

On Tuesday July 8 1980, the Tyrrell’s and the Tulloch’s, Reg Drayton and Dr Max Lake together with Chris Barnes, who, as President of the Hunter Valley Vineyard Association represented virtually all the other wine-makers, held a press conference at the Hilton Hotel, Sydney.

Their message was simple – they could foresee the day when the Hunter Valley was finished as a wine-growing area. And the reason? For the past ten years the ALCAN aluminium smelter at Kurri-Kurri had rained 600 to 700 tonnes of fluoride pollutants onto the surrounding landscape annually. The wine-makers said they had known nothing about these fluoride emissions until 10 months previously, yet fluoride pollutants have, in the past, reduced grape yield and decimated vineyards in Spain, Greece, Bulgaria and the Rhone Valley.

Ever since the beginning of the industrial revolution but particularly in the second half of this century, wholesale pollution of air and of the countryside with fluoride fumes and fall-out has taken place; and the most common and most dangerous fluoride air pollutant is HYDROGEN FLUORIDE.

As mentioned previously, Dr Jag Cook, from Britain’s National Chemical Emergency Group – which is responsible for mopping up any major toxic spills in the UK – has said: “Hydrogen fluoride is about the only chemical that really scares me.”

Hardly surprising since amongst other things, hydrogen fluoride (HF) eats up glass and dissolves most metals.

Alright, you say, its dangerous, but I don’t live near a factory that releases HF into the atmosphere, nor do I work in an environment where HF is present. But consider this. Demand for lead-free petrol is growing quickly and the processes for making it involve the use of HF to achieve high octane ratings without using lead. In fact, between 1.26 and 3.14 kilos of HF are used in the production of every six barrels of alkylate.

As a result HF is present in the exhaust gases from vehicles using lead-free petrol. The levels of HF, three inches from the exhaust outlet measure 30 parts per billion, and remember at that concentration, HF can impair reflex activity in rats by acting as a CNS depressant – in other words, a mind-dulling drug.

HYDROGEN FLUORIDE, aka:, is used by an increasing number of industries, and it is also produced as a pollutant by an increasing number of industries.

A series of accidents in the United States have recently demonstrated that industrial HF sites are a major threat to public safety.

For instance, an HF leak on 30 October 1987 at the Marathon refinery in Texas City left 700 people in need of urgent medical treatment. Dr Fred Millar, of the Environmental Policy Institute, said that only luck had prevented the accident from becoming the major industrial catastrophe of the year. He pointed out:

“The release was from the vapour space of a storage tank. If the same release had been of HF liquid thousands would likely have died in the ensuing gas cloud. It would have been our Bhopal.”

A few months later, another HF leak occurred at Mobil’s refinery in Torrance, California. This caused a raging 41-hour fire and millions of dollars worth of damage. An official report of the accident suggested:

“The consequences may have been so great as to warrant regulations to direct industry to phase out its use or substitute processes with less environmental hazards.”

In March 1988, there was another HF leak, this time in Tulsa, Oklahoma. There, an accident at the Sun refinery produced a three-mile-long cloud which engulfed the town Only a prompt evacuation limited the casualties to 36 persons (none fatal).

A recent test by the US Government showed that relatively small amounts of HF liquid will release a dense, ground-hugging gas cloud which remains lethal for five kilometres.

In Britain, the location of HF manufacturing plants are, according to the Health and Safety Executive, officially secret – to prevent them becoming targets for terrorists.

Many people, particularly those working in the pot-rooms of aluminium smelters, are exposed to relatively high concentrations of hydrogen fluoride. What can it do to them? Well, lets see.

In the spring of 1986, one of the most modern aluminium smelters in the world went into production in Portland, Victoria. The smelter had been built by the Aluminum Company of America (ALCOA), who also owned a much older smelter at Point Henry, Geelong.

Two years later, on December 2 1988, the Melbourne Age reported:

“SMELTER WORKERS CLAIM FOR ASTHMA.”
 “Twelve workers from the 35 per cent State-owned Portland aluminium smelter have issued common-law claims against the joint-venture seeking damages for occupational asthma. 

The chairman of the Aluminium Development Council, Mr. Bruce Heister, said the incidence of occupational asthma varied from smelter to smelter but the reasons for this were not clear. 

Damages for a case of occupational asthma were claimed against another big aluminium producer, Comalco, at its Queensland smelter a few months ago. 

The cause of pot room asthma is suspected to be an agent, or agents, in emissions from smelter pot lines. 

Since production started in Portland in October 1986, 65 workers have been diagnosed as having occupational asthma.”

In other words, after just 25 months in operation, 65 workers at one of the most modern aluminium smelters in the world had been affected by mysterious agents in the pot room.

Worse was to follow. On 27 April 1989, the Melbourne Herald reported:

“ALUMINIUM IS LATEST WORKER HEALTH SCARE.”

“A senior Victorian union official claims workers at Geelong’s ALCOA smelter are suffering respiratory ailments potentially as deadly as those found in the asbestos industry. 

Mr. Royre Bird, slate secretary of the Federated Iron-workers Association, has called for a national inquiry into respiratory disease in aluminium smelter workers after a report by New South Wales researchers found evidence of long-term irreversible lung damage.
 
The report, by a team from Newcastle University medical school, found workers at Alcan Aluminium’s Kurri-Kurri smelter suffered reduced lung function equivalent to smoking a packet of cigarettes a day. 

Mr. Bird, who has worked in the industry for 18 years, claimed the findings had serious implications for the aluminium industry world-wide and for workers at Geelong. He said he believed that apart from respiratory diseases, aluminium workers were at risk of contracting cancer. 

He claimed to have observed a “slow but gradually developing history of cancers” at the Point Henry Plant in Geelong.

He also claimed workers at the Portland smelter, partly owned by the State Government, were suffering higher rates of pot room asthma than at Point Henry. 

Union solicitors had confirmed 176 cases of pot room asthma at Point Henry since 1964, compared with 76 at Portland. At least 20 more cases were being processed by other solicitors, he said.”

A few days later, a cancer specialist supported Mr Bird’s claim when the Melbourne Sun published the following article on May 1 1989:

“CANCER RISK AT SMELTERS: DOCTOR”.
“Workers at aluminium smelters are at risk of developing cancer as well as chronic asthma, according to a leading cancer specialist. At least 39 smelter workers across Australia are believed to have already died from work-related cancer.
 
Dr Cyril Minty, a specialist at the Peter McCallum cancer hospital, said fumes emitted from the smelters’ pot rooms could contain cancer-causing chemicals as well as irritants that produced the respiratory condition known as ‘pot room asthma’
 
Dr Minty said more than six sufferers of industrial asthma from Portland and ALCOA’s Geelong smelter had been referred to him during the past year.”

Now, there is no mystery at all. The major pollutants in the pot room are gaseous and particulate fluorides; and HYDROGEN FLUORIDE is the most common fluoride gas.

HYDROGEN FLUORIDE IS THE MAJOR CAUSE OF

POT ROOM ASTHMA AND A CONTRIBUTING FACTOR

IN THE DEVELOPMENT OF LUNG CANCER IN

SOME ALUMINIUM WORKERS.

But, industrialists live in fear of conclusive evidence linking a pollutant to ‘new’ occupational or Neighbourhood diseases. The reason is obvious. Employers and their insurers will face claims for compensation.

(Note: a “Neighbourhood disease” is one affecting people living in the vicinity of a pollutant producing factory.)

Industries with major fluoride pollution problems are amongst the most powerful interest groups in society. Fluoride emissions are amongst the most difficult of all pollutants to control effectively, and in a highly competitive economic system, many companies will fight for their very lives to avoid spending large amounts of money to control pollution since this will, almost inevitably, increase the price of the end-product.

Certain sections of industry will go to great lengths to suppress stories about fluoride pollution. Such reports might encourage people to sue for damages or, result in pressures for tougher anti-pollution laws.

The first symptoms of exposure to trace amounts of hydrogen fluoride are NOT physiological but psychological, and include such symptoms as confusion, fatigue, partial loss of memory and mental dullness. To put it another way, behaviour is exquisitely sensitive to minute traces of hydrogen fluoride (and other pollutants) in the environment.

Unfortunately, the tests to which chemical substances are usually subjected in efforts to determine their so-called “maximum permissible doses or concentration” do not take into account possible changes in mental function, and also would often fail to pick up long-term or chronic effects on the organism.

Minute concentrations of hydrogen fluoride inhaled over lengthy periods of time CAN DAMAGE VITAL COMPONENTS OF THE IMMUNE SYSTEM – this leaves the individual vulnerable to opportunistic diseases.

Last century, canaries were taken down coal mines because of the presence of trace amounts of deadly gases in the mines. The gases were undetectable by smell but if the canary died, the miners got out – quickly!

Some scientists suspect that FROGS have become analogous to the coal-mine canaries. All over the world frogs are disappearing and no-one knows why. The best guess so far is that pollution of the environment is responsible. I’d like to tell you about an experiment I recently completed.

In the adult human the immune system weighs about two pounds and consists of around a trillion lymphocytes and about 100 million trillion molecules called antibodies that are produced and secreted by the lymphocytes.

In a mouse, the immune system consists of about 300 million lymphocytes and around a trillion antibodies.

The smallest known immune system, that of a tadpole, is estimated to have a million lymphocytes and an antibody repertoire of about 10 million. Smaller immune systems do not exist presumably because such systems would recognize antigen so infrequently that they would provide little, if any, protective advantage.

I exposed tadpoles to a number of increasingly common environmental pollutants, including mercury, cadmium and hydrofluoric acid – which is hydrogen fluoride in water, and both gas and acid have the same formula, HF.

Incredibly low concentrations of these chemicals proved lethal to the tadpoles.

But technically speaking, the tadpoles didn’t die of “mercury poisoning” or “cadmium poisoning,” or “hydrofluoric acid” poisoning. They died because the chemicals ‘wrecked’ their immune systems leaving the tadpoles vulnerable to all the germs and parasites in their environment.

The significance of this is that scientists still evaluate the toxicity of a chemical by determining what amount of the chemical causes obvious damage or death.

For instance, lets look at a common chemical – sodium fluoride.

It would take at least 3 grams of sodium fluoride to kill a healthy adult. That’s the amount in 3,000 litres of fluoridated water.

If you ingested about 8 milligrams of sodium fluoride daily for ten years or more, you would develop a well-defined disease called skeletal fluorosis, which affects bones, tendons and secondarily, the nervous system. If an infant ingested 2 milligrams of fluoride daily, they would develop dental fluorosis or ‘mottled’ teeth.

Apparently therefore, the only problems that low doses of sodium fluoride can cause are either dental fluorosis or skeletal fluorosis. The CLINICAL symptoms of these conditions are easily detected – ‘mottled’ teeth and ‘bony outgrowths’ and the calcification of tendons in skeletal fluorosis.

BUT WHAT ABOUT SUB-CLINICAL SYMPTOMS

THOSE THAT WE CAN’T SEE?

Experiments have shown water containing 1 to 4 parts per million can have an effect on the Central Nervous System – a mind-dulling effect! Experiments have also demonstrated that fluoride at a concentration of just 0.6 parts per million can disturb antibody production, and thus interfere with the functioning of the immune system.

And many experiments have shown that concentrations of fluoride of about 4 parts per million can damage DNA – the vital core of every living cell.

In other words, at very low concentrations, fluoride can cause subtle changes in enzyme activities, nerve action potentials, altered behavioural reaction, and the immune system…

………..

About Dr. Smith

Dr Geoffrey Ernest Smith, L.D.S., R.C.S. (Eng.)
Dental Surgeon, (retired)

Curriculum Vitae

  • Born: 1 November 1932, Married, 5 children, 4 grandchildren.

  • Educated: Lawrence House School, St. Annes on Sea, Lancashire. Rossall School, Fleetwood, Lancashire. University of Manchester, Turner Dental School.

  • 1956: Qualified L.D.S., R.C.S. Royal College of Surgeons. (England).

  • 1957-59 Post-Graduate Studies. Queens University, Belfast.

  • 1959-60 Travelling Fellowship, UK Medical Research Council; WHO Regional Office, Brazzaville. 

    Based University of Ibaden W. Nigeria. Field work: Nigeria, Ghana, Sierra Leone, Gambia and Liberia.

  • 1961-68 General Dental Practice, London and Dublin.

  • 1965-66 Consultant, Aspro-Nicholas, Ireland, Ltd., Dublin.

  • 1969-71 Consultant, Glaxo Group Ltd., London.

  • 1972-74 Consultant, PIA Ltd., Lopex Group, London, New York.

  • 1974-76 Consultant, Nicholas International, Slough & Melbourne.

  • 1976-79 General Dental Practice and School Dentistry, Melbourne.

  • 1979-80 Hospital Dentist, Proserpine, North Queensland.

  • 1987-88 Anthropology and Archaeology, University of Melbourne.

  • 1980 – Consultant, Environmental and Public Health.

Some relevant papers in the scientific literature:

  • NZ Med.J. 1983;96, 1067-1068.

  • Persp. Biol.Med. 1986 29, 560-

  • New Scientist 1983;5 May 286-287

  • Trends.Pharm.Sci.1986, 7, 10

  • Fluoride, 1983, Editorial, Autumn.

  • Sci.Tot.Environ. 1987, 63, 1-11.

  • Aust.Dent.J. 1984, 29, 199-200.

  • Endeavour, 1987, 11, 16-

  • J.R.Coll Gen.Pract.1984, 34, 350-351.

  • Sci.Prog.(Oxf.), 1987, 71, 23

  • Xenobiotica, 1985, 15, 177-186.

  • The Scientist, 1987, 1, 24.

  • Sci.Prog.(Oxf.), 1985, 69, 429-442.

  • Sci.Tot.Env., 1988, 68, 79-86.

  • Sci.Tot.Env. 1985, 43, 41-61.

  • NZ Med.J. 1985, 90, 556-557.

  • NZ Med.J. 1985, 98, 454-455.

  • Pers.Biol.Med. 1988, 31, 440-45

  • NZ Med.J. 1988, 100, 669-670.

  • Sci.Tot.Env., 1988, 76, 167-

  • New Scientist, 1985, 1467-, 50-51.

  • NZ Med.J. 1988, 101, 802.

  • NZ Med.l. 1985, 30, 232-233.

  • Med.Hypoth., 1986, 19, 139-

  • Med.J.Aust., 1985, 143, 283-286.

  • Mutat.Res., 1990, 241, 339-

  • Med.J.Aust., 1986, 144, 152.

  • Fusso Kenkyo, 1990, 11, 38-48

  • Lawyer, 1986, 4(3), 6. (Japanese)

  • Fluoride, 1986, 19, 105-107.

  • Probe, 1989, 31, 1-2.

  • Nature 1986; 323, 198.

  • Aust. Dent.J., 1985, 30, 232-233

Note:

The Secret War was originally prepared as a personal submission to the

Brisbane City Council’s Task Force on Fluoridation, in March 1997.

Paper: Original Paper 
Citation : Caries Res 2001;35:125-128
 Title : Fluoride Deposition in the Aged Human Pineal Gland Author(s): J. Luke
Info : Figures: 2; Tables: 0; References: 32

 Keywords : Calcium; Distribution; Fluoride; Human pineal gland; Hydroxyapatite; Pineal concretions
 
Abstract : The purpose was to discover whether fluoride (F) accumulates in the aged human pineal gland. The aims were to determine (a) F-concentrations of the pineal gland (wet), corresponding muscle (wet) and bone (ash); (b) calcium-concentration of the pineal.

Pineal, muscle and bone were dissected from 11 aged cadavers and assayed for F using the HMDS-facilitated diffusion, F-ion-specific electrode method. Pineal calcium was determined using atomic absorption spectroscopy.

Pineal and muscle contained 297+/-257 and 0.5+/-0.4 mg F/kg wet weight, respectively; bone contained 2,037+/-1,095 mg F/kg ash weight. The pineal contained 16,000+/-11,070 mg Ca/kg wet weight.

There was a positive correlation between pineal F and pineal Ca (r = 0.73, p<0.02)

but no correlation between pineal F and bone F.

By old age (50-70), the  pineal gland  has readily accumulated F

and its F/Ca ratio is higher than bone.

red-line-copy

buffalo-lameeness-f

f-is-mor-dangerous-than-us-f

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