These findings suggest that vitamin C significantly reduced 
the severity and incidence of fluoride-induced embryo toxicity in rats.

Guinea pigs, apes, some fruit
bats and humans share another trait:

The tendency to develop coronary heart disease.
The development of heart disease only occurs in animals and
humans that lack adequate intake of ascorbates through dietary sources.

Lab Rat f

Original ⇒ HERE

PubMed logo

Vit C Rats F. Verma

Jai Research Foundation, Vapi, Valvada 396 108, India 

     Oral administration of sodium fluoride (40 mg/kg body weight) from day 6 to 19 of gestation caused, as comparedto control, significant reductions in body weight, feed consumption, absolute uterine weight and number of implantations. Significantly higher incidenceof skeletal (wavy ribs, 14th rib, <6 sternal centre, dumbell-shaped second and fifth sternebrae, incompleteossification of skull and thickening of tibia) and visceral (subcutaneous haemorrhage) abnormalities were also observed in NaF-treated dams than that of control. Oral administration of vitamin C (50 mg/kg bodyweight) and vitamin E (2 mg/0.2 ml olive oil/animal/day) from day 6 to 19 of gestation along with NaF significantly ameliorates NaF-induced reductions in body weight, feed consumption, absolute uterine weight (only with vitamin E treatment) and number of implantations. Ascompared with NaF-treated alone, the total percentage of skeletal and visceral abnormalities were significantly lowered in fluoride plus vitamin C-treated animals. Vitamin E was less effective. These findings suggest that vitamin C significantly reduced the severity and incidence of fluoride-induced embryo toxicity in rats.

Key Words: fluoride • vitamins • embryo toxicity • amelioration

   Human & Experimental Toxicology, Vol. 20, No. 12, 619-623 (2001)


          See also ↓ 


          See also ↓ 





… going by the above evidence, fluorinated drugs seem to pose
a number of risks associated with the fluorine or fluoride
contained in them. It raises even more concern when
fluoride itself is present in many industries and
products, including food and drinks … 

Prof. Abdul Razak

Professor Dzulkifli Abdul Razak

National Poison Centre
University Sains Malaysia – 
2 Sept. 2001


He is the First Vice-President of the International Association of Universities (IAU),
a UNESCO affiliated organisation based in Paris.
He was President of the Association of Southeast Asia Institutions of Higher Learning from 2007-2008.


   Following the recent withdrawal of the cholesterol-lowering drug Lipobay, there is now a new perspective to the issue, the drug being a fluoride-containing compound. The drug, also known by its generic name, cerivastatin, is one of the many such compounds pulled off the shelves in the last few years.

Cerivastatin was taken off because of at least 40 deaths worldwide, 31 in the US alone. According to a recently released commentary by a Canadian group, Parents of  Fluoride Poisoned Children, a series of fluoride containing drugs or so-called fluorinated drugs have been withdrawn from the market in the last 10 years due to their toxic effects on human beings. One notable example is the combination “Fen-Phen” (a generic combination of fenfluramine and phentermine, the former being a fluorinated drug type) which was said to have weight-reducing effects. Others are dexfenfluramine (Redux) and fenfluramine (Pondimin).

There are at least eight other examples of fluorinated drugs withdrawn so far, because serious side effects on the heart, and for suspected adverse influence on thyroid hormone activity.

They include, last year, cisapride (Propulsid) because of its severe side-effects on the heart. In 1999, two drugs were withdrawn.

These were an anti-allergy drug, astemizole (Hismanal); and grepafloxacin (an antibiotic, Raxar) because they too were associated with similar adverse events.

In 1998, patients with congestive heart failure using the drug mibedrafil (Posicor) showed a trend to higher mortality, causing it to be withdrawn.

Alredase (Tolrestat, an anti-diabetic) was withdrawn in 1997 after the appearance of severe liver toxicity and deaths among several patients. In the same year too fenfluramine (part of Fen-Phen) and dexfenfluramine were withdrawn.

In 1993, flosequinan (Manoplax, a heart drug) was withdrawn when it was shown that the beneficial effects on the symptoms of heart failure did not last beyond the first three months of therapy. After that, patients had a higher rate of hospitalization than patients taking a placebo.

Of the many fluorinated drugs that remain in the market some carry warnings of serious cardiac toxicity, for instance halofantrine, a schizonticidal drug. More specifically, other fluorinated drugs, although they have not yet been withdrawn, are known to cause muscle wasting or rhabdomyolysis; like cerivastatin.

For instance, the PFPC commentary noted that the fluorinated antibiotic fluoroquinolone, used to treat infections, is reported to cause tendonitis and rhabdomyolysis. In fact product information for such antibiotics (enoxacin, fleroxacin, norfloxacin, sparfloxacin, and tosufloxacin) was amended in Japan in October 1994, to state that rhabdomyolysis may occur. Reportedly, the tragic story involving fluorinated drugs (the fluorophenyls in particular, initially limited to industrial use involving dyes and pesticides) can be traced way back to the 1930s when they were used to treat hyperthyroidism.

The use followed a discovery by IG Farben (Bayer) and Knoll’s scientists that all fluoride compounds can interfere with thyroid hormone activity.

In the liver especially, organic fluoride compounds undergo extensive transformation, mainly via oxidative demethylation, involving the thyroid hormone (T3) mediated P-450 enzyme system. And the resulting metabolites may have higher activity and/or greater toxicity than the original compound.

The activity of organic fluoride compounds on the P-450 enzyme system is critical as it relates to the elimination of many other drugs. Inhibition of these enzymes can cause other drugs to accumulate to dangerous levels in the body, leading to hazardous drug interactions. In many cases fluorinated drugs are being implicated as documented in hundreds of well-established studies.

Moreover, adds PFPC, the metabolites produced by organic fluoride compounds in the liver can be transferred to the fetus through various pathways, including circulatory via placental passage, gastrointestinal via fetal swallowing, and respiratory secondary to fetal lung absorption. This may lead to congenital abnormalities as in the case of fluconsazole (Diflucan).

In short, going by the above evidence, fluorinated drugs seem to pose a number of risks associated with the fluorine or fluoride contained in them. It raises even more concern when fluoride itself is present in many industries and products, including food and drinks, without any rigorous evaluation or monitoring.

Of  late, we have managed to label all toothpastes containing fluoride in this country. But this is clearly a minuscule effort in the attempt to regulate the use of fluoride as an inherent poison. We need to do more now.

For more information, contact the National Poison Centre at Universiti Sains Malaysia,
tel: 04-657 0099, fax: 04-656 8417,

USM Uni Logo

Vice-Chancellor, Albukhary International University, Malaysia



Professor Dzulkifli Abdul Razes
recent post is Vice Chancellor
Albukhary University

Source: New Sunday Times (Focus) 2 September 2001


More info on fluorinated medications  HERE




… Fluoride air pollution can have a devastating
 effect on the total environment …

Water Samples – Probably Carcinogenic


Aust. Defence Bases Contaminated With Fluoride Chemicals

The Secret War .

Image of Dr. Smith

The major industries with fluoride pollution problems include:

coal-burning power stations, petro-chemical refineries, aluminium,

zinc, copper, beryllium and magnesium producing factories, steel mills,

fertilizer works, plastics manufacturers, glass factories, cement works, 

pottery and tile makers, brick works, chemical factories and nuclear

processing plants. The most common and dangerous air pollutant

produced by these industries and many others, is —

Hydrogen Fluoride

3 smoke stacks f

   Workers in the following occupations may be exposed to

Hydrogen Fluoride In The Workplace Air:

Aircraft workers,
 – Alkylation plant workers, – 
Alloy steel cleaners, – 
Alloy steel makers, – 
Aluminum fluoride makers, – 
Aluminum makers, – 
Beryllium workers, 
- Bleachers, – 
Brass cleaners, 
- Brewers, -
 Brick cleaners, – 
Brick makers, -
 Casting cleaners, – 
Cement workers, – 
Ceramic workers, -
 Chemists, – Copper cleaners, 
- Cryolite makers, – 
Crystal glass polishers, – 
Dye makers, – 
Electric arc welders, – 
Electroplaters, – 
Enamel etchers, – Fermentation workers, – 
Fertilizer makers, – Filter paper makers, 
- Fluoborate makers, – 
Fluoride compound makers, – 
Fluorine makers, –  
Fluorocarbon makers, – 
Fluor-chemical makers, 
- Fluosilicate makers, – Freon makers, – 
Genetron makers, 
- Glass etchers, 
- Graphite purifiers, – 
Hydrogen fluoride makers, – 
Isotron makers, 
- Laundry workers, – 
Metal cleaners, 
- Metal polishers, 
- Neon sign makers, 
- Oil well acidifiers, –  
Ore dissolvers, – 
Petrol refinery workers, 
- Phosphate rock workers, 
- Plastic makers, 
- Polish makers, 
- Pottery workers, 
- Power station workers, 
-  Quartz crystal makers, 
- Rocket fuel handlers, 
- Rocket fuel makers, 
- Silicon chip makers, – 
Stainless steel cleaners, – 
Stainless steel makers, 
- Steel casting pickles, 
- Steel millworkers, – Stone cleaners, – 
Tile makers, 
- Uranium refiners, 
- Yeast makers, and
 Zinc workers.

   Hydrogen Fluoride in the workplace air:

Fluoride air pollution can have a devastating effect on the total environment.

Angus Lazores is a Mohawk Indian.
For centuries before the white-man reached Canada and the United States, the Mohawks hunted, fished, trapped, and farmed the islands of the Gulf of  St. Lawrence, now known as the St. Regis Akwesasne Indian Reserve.

Angus Lazores, along with 1,500 other Mohawks, lives on Cornwall Island, a part of the reserve straddling the borders of  Quebec, Ontario, and Upper New York State. The St. Regis Mohawk Band settled Cornwall Island just over a hundred years ago; they soon became known as an efficient and self-sufficient agricultural community. In 1959, there were 45 farmers, forty cattle barns and 364 dairy cattle on the Island.

Twenty years later, only eight farmers and eight cattle barns were left.

During the twenty years the cattle population was decimated;

all the bees on the Island had disappeared; crop yields had fallen;

partridges, after which the Akwesasne Reserve is named, had declined drastically; and the white pine trees on the Island were dying.

In 1959, Reynolds Metals Company had built an aluminium smelter on the south bank of the St. Lawrence River near Massena, New York State. Cornwall Island is downwind of the smelter at least 60 per cent of the time.

Angus Lazores dates his problems on the Island to 1962, just three years after the smelter became operational.

In that year, cattle became lame and developed swellings on their legs, eventually the lameness became so severe that the animals could no longer graze normally. They laid down to eat on pasture and then crawled to the next place to eat. With increasing age the cows had difficulty drinking cold water, and chewing was obviously painful. The animals would grab hay but let it go after unsuccessful attempts at mastication.

The first pregnancy and calving were usually uneventful, but the cows had small udders and too little milk for the calf. At the third pregnancy and delivery, the native cows had usually deteriorated, being unable to drink or chew properly. Cows died during delivery and neonatal calf mortality was high. If cows survived the third pregnancy they were sold for slaughter.

By 1971, the majority of farmers had switched from dairy to beef cattle and by November 1977, there were only 177 cattle on the Island compared with 364 in 1959.

The cause of the cattle disease was admitted only after many years. In 1969, officials of the Canadian Ministry of the Environment had expressed concern to Reynolds Metals about fluoride emissions impacting on the Island. Four years later, the St. Regis Local Council authorised an investigation into pollutants emitted by the smelter. In July 1973, the Council were advised that damage to the pine trees on the Island was due to fluoride gases.

Two years later, urine samples from Cornwall Island cattle showed abnormal levels of fluoride.

In November 1975, Angus Lazore’s cattle were examined by a veterinarian called Abbey, sent by Reynolds Metals. He claimed that internal and external parasites were responsible for the condition of the cattle – fluoride wasn’t even mentioned.

The Mohawk elders were disturbed by Abbey’s diagnosis and approached Professor Lennart Krook, an eminent veterinary scientist at Cornell University.

Krook ran extensive diagnostic and pathological tests on the St. Regis cattle, then announced his findings:

“Owing to extensive and serious chronic fluoride poisoning no cattle born on Cornwall Island were going to live for more than five years.”

During 1977 and 1978, the situation which had developed on the Island was investigated by a team of scientists from the New York State College of Veterinary Medicine, Cornell University. Leaders of the team were Professor Krook and Dr George Maylin. In the introduction to their published report, they point out:

“Of all pollutants that affect farm animals, fluorine has caused the most severe and widespread damage. The object of the present study is to record yet another man-made fluorine pollution disaster and to interpret the pathogenesis of the osseous changes in view of recent advances in the understanding of bone metabolism.”

While Krook and Maylin focused on the cattle, Dr Clancy Gordon of the University of Montana, examined 2,600 plant samples from Cornwall Island and found very high levels of fluoride in all the vegetation tested.

University of Illinois scientists were then recruited to see if the Islanders themselves were suffering health problems resulting from excessive exposure to fluoride. Doctors Bertram Carnow and Shirley Conibear reported:

“Significant numbers of people with abnormalities of the muscular, skeletal, nervous and blood systems.”

In addition, Cornwall Island physicians had noted high rates of anaemia, rashes, irritability, diabetes, high blood pressure and thyroid disease.

Carnow and Conibear concluded that there had been;

“Unquestionably heavy exposure to fluorine compounds that has affected all the life studied.”

They recommended an immediate reduction in smelter fluoride emissions. Chief Francis of the Mohawk Indian Band put it more dramatically, he advised anyone living in areas where smelters might be built, to:

“Block the project. Block them with everything you have. If you fail then move. Move as quickly as you can because there’s no money that can buy your health back.”

Reynolds Metals spent its first ten years of operation spewing over 130 kilos of fluoride emissions an hour, directly downstream to Cornwall Island. Even after New York State regulations forced the company to reduce its emissions to 30 kilos an hour by 1975, Reynolds’ “gift” to the Mohawks had been an appalling –



The Mohawk way of life became a victim of a preventable man-made plague. And you don’t have to go to Canada to find fluoride pollution problems. For more than a century, the Hunter Valley Region of New South Wales has produced some of Australia’s finest wines.

On Tuesday July 8 1980, the Tyrrell’s and the Tulloch’s, Reg Drayton and Dr Max Lake together with Chris Barnes, who, as President of the Hunter Valley Vineyard Association represented virtually all the other wine-makers, held a press conference at the Hilton Hotel, Sydney.

Their message was simple – they could foresee the day when the Hunter Valley was finished as a wine-growing area. And the reason? For the past ten years the ALCAN aluminium smelter at Kurri-Kurri had rained 600 to 700 tonnes of fluoride pollutants onto the surrounding landscape annually. The wine-makers said they had known nothing about these fluoride emissions until 10 months previously, yet fluoride pollutants have, in the past, reduced grape yield and decimated vineyards in Spain, Greece, Bulgaria and the Rhone Valley.

Ever since the beginning of the industrial revolution but particularly in the second half of this century, wholesale pollution of air and of the countryside with fluoride fumes and fall-out has taken place; and the most common and most dangerous fluoride air pollutant is HYDROGEN FLUORIDE.

As mentioned previously, Dr Jag Cook, from Britain’s National Chemical Emergency Group – which is responsible for mopping up any major toxic spills in the UK – has said: “Hydrogen fluoride is about the only chemical that really scares me.”

Hardly surprising since amongst other things, hydrogen fluoride (HF) eats up glass and dissolves most metals.

Alright, you say, its dangerous, but I don’t live near a factory that releases HF into the atmosphere, nor do I work in an environment where HF is present. But consider this. Demand for lead-free petrol is growing quickly and the processes for making it involve the use of HF to achieve high octane ratings without using lead. In fact, between 1.26 and 3.14 kilos of HF are used in the production of every six barrels of alkylate.

As a result HF is present in the exhaust gases from vehicles using lead-free petrol. The levels of HF, three inches from the exhaust outlet measure 30 parts per billion, and remember at that concentration, HF can impair reflex activity in rats by acting as a CNS depressant – in other words, a mind-dulling drug.

HYDROGEN FLUORIDE, aka:, is used by an increasing number of industries, and it is also produced as a pollutant by an increasing number of industries.

A series of accidents in the United States have recently demonstrated that industrial HF sites are a major threat to public safety.

For instance, an HF leak on 30 October 1987 at the Marathon refinery in Texas City left 700 people in need of urgent medical treatment. Dr Fred Millar, of the Environmental Policy Institute, said that only luck had prevented the accident from becoming the major industrial catastrophe of the year. He pointed out:

“The release was from the vapour space of a storage tank. If the same release had been of HF liquid thousands would likely have died in the ensuing gas cloud. It would have been our Bhopal.”

A few months later, another HF leak occurred at Mobil’s refinery in Torrance, California. This caused a raging 41-hour fire and millions of dollars worth of damage. An official report of the accident suggested:

“The consequences may have been so great as to warrant regulations to direct industry to phase out its use or substitute processes with less environmental hazards.”

In March 1988, there was another HF leak, this time in Tulsa, Oklahoma. There, an accident at the Sun refinery produced a three-mile-long cloud which engulfed the town. Only a prompt evacuation limited the casualties to 36 persons (none fatal).

A recent test by the US Government showed that relatively small amounts of HF liquid will release a dense, ground-hugging gas cloud which remains lethal for five kilometres.

In Britain, the location of HF manufacturing plants are, according to the Health and Safety Executive, officially secret – to prevent them becoming targets for terrorists.

Many people, particularly those working in the pot-rooms of aluminium smelters, are exposed to relatively high concentrations of hydrogen fluoride. What can it do to them? Well, lets see.

In the spring of 1986, one of the most modern aluminium smelters in the world went into production in Portland, Victoria. The smelter had been built by the Aluminum Company of America (ALCOA), who also owned a much older smelter at Point Henry, Geelong.

Two years later, on the 2nd December 1988, the Melbourne Age reported:

 “Twelve workers from the 35 per cent State-owned Portland aluminium smelter have issued common-law claims against the joint-venture seeking damages for occupational asthma. 

The chairman of the Aluminium Development Council, Mr. Bruce Heister, said the incidence of occupational asthma varied from smelter to smelter but the reasons for this were not clear. 

Damages for a case of occupational asthma were claimed against another big aluminium producer, Comalco, at its Queensland smelter a few months ago. 

The cause of pot room asthma is suspected to be an agent, or agents, in emissions from smelter pot lines. 

Since production started in Portland in October 1986, 65 workers have been diagnosed as having occupational asthma.”

In other words, after just 25 months in operation, 65 workers at one of the most modern aluminium smelters in the world had been affected by mysterious agents in the pot room.

Worse was to follow. On 27 April 1989, the Melbourne Herald reported:


“A senior Victorian union official claims workers at Geelong’s ALCOA smelter are suffering respiratory ailments potentially as deadly as those found in the asbestos industry. 

Mr. Royre Bird, slate secretary of the Federated Iron-workers Association, has called for a national inquiry into respiratory disease in aluminium smelter workers after a report by New South Wales researchers found evidence of long-term irreversible lung damage.
The report, by a team from Newcastle University medical school, found workers at Alcan Aluminium’s Kurri-Kurri smelter suffered reduced lung function equivalent to smoking a packet of cigarettes a day. 

Mr. Bird, who has worked in the industry for 18 years, claimed the findings had serious implications for the aluminium industry world-wide and for workers at Geelong. He said he believed that apart from respiratory diseases, aluminium workers were at risk of contracting cancer. 

He claimed to have observed a “slow but gradually developing history of cancers” at the Point Henry Plant in Geelong.

He also claimed workers at the Portland smelter, partly owned by the State Government, were suffering higher rates of pot room asthma than at Point Henry. 

Union solicitors had confirmed 176 cases of pot room asthma at Point Henry since 1964, compared with 76 at Portland. At least 20 more cases were being processed by other solicitors, he said.”

A few days later, a cancer specialist supported Mr Bird’s claim when the Melbourne Sun published the following article on May 1 1989:


”Workers at aluminium smelters are at risk of developing cancer as well as chronic asthma, according to a leading cancer specialist. At least 39 smelter workers across Australia are believed to have already died from work-related cancer.
Dr Cyril Minty, a specialist at the Peter McCallum cancer hospital, said fumes emitted from the smelters’ pot rooms could contain cancer-causing chemicals as well as irritants that produced the respiratory condition known as ‘pot room asthma’
Dr Minty said more than six sufferers of industrial asthma from Portland and ALCOA’s Geelong smelter had been referred to him during the past year.”

Now, there is no mystery at all. The major pollutants in the pot room are gaseous and particulate fluorides; and HYDROGEN FLUORIDE is the most common fluoride gas.





But, industrialists live in fear of conclusive evidence linking a pollutant to ‘new’ occupational or Neighbourhood diseases. The reason is obvious. Employers and their insurers will face claims for compensation.

A “Neighbourhood disease” is one affecting people living in the vicinity of a pollutant producing factory.)

Industries with major fluoride pollution problems are amongst the most powerful interest groups in society. Fluoride emissions are amongst the most difficult of all pollutants to control effectively, and in a highly competitive economic system, many companies will fight for their very lives to avoid spending large amounts of money to control pollution since this will, almost inevitably, increase the price of the end-product.

Certain sections of industry will go to great lengths to suppress stories about fluoride pollution. Such reports might encourage people to sue for damages or, result in pressures for tougher anti-pollution laws.

The first symptoms of exposure to trace amounts of hydrogen fluoride are NOT physiological but psychological, and include such symptoms as confusion, fatigue, partial loss of memory and mental dullness. To put it another way, behaviour is exquisitely sensitive to minute traces of hydrogen fluoride (and other pollutants) in the environment.

Unfortunately, the tests to which chemical substances are usually subjected in efforts to determine their so-called “maximum permissible doses or concentration” do not take into account possible changes in mental function, and also would often fail to pick up long-term or chronic effects on the organism.

Minute concentrations of hydrogen fluoride inhaled over lengthy periods of time CAN DAMAGE VITAL COMPONENTS OF THE IMMUNE SYSTEM – this leaves the individual vulnerable to opportunistic diseases.

Last century, canaries were taken down coal mines because of the presence of trace amounts of deadly gases in the mines. The gases were undetectable by smell but if the canary died, the miners got out – quickly!

Some scientists suspect that FROGS have become analogous to the coal-mine canaries. All over the world frogs are disappearing and no-one knows why. The best guess so far is that pollution of the environment is responsible. I’d like to tell you about an experiment I recently completed.

In the adult human the immune system weighs about two pounds and consists of around a trillion lymphocytes and about 100 million trillion molecules called antibodies that are produced and secreted by the lymphocytes.

In a mouse, the immune system consists of about 300 million lymphocytes and around a trillion antibodies.

The smallest known immune system, that of a tadpole, is estimated to have a million lymphocytes and an antibody repertoire of about 10 million. Smaller immune systems do not exist presumably because such systems would recognize antigen so infrequently that they would provide little, if any, protective advantage.

I exposed tadpoles  [ see below] to a number of increasingly common environmental pollutants, including mercury, cadmium and hydrofluoric acid – which is hydrogen fluoride in water, and both gas and acid have the same formula, HF.

Incredibly low concentrations of these chemicals proved lethal to the tadpoles.

But technically speaking, the tadpoles didn’t die of “mercury poisoning” or “cadmium poisoning,” or “hydrofluoric acid” poisoning. They died because the chemicals ‘wrecked’ their immune systems leaving the tadpoles vulnerable to all the germs and parasites in their environment.

The significance of this is that scientists still evaluate the toxicity of a chemical by determining what amount of the chemical causes obvious damage or death.

For instance, lets look at a common chemical – sodium fluoride.

It would take at least 3 grams of sodium fluoride to kill a healthy adult. That’s the amount in 3,000 litres of fluoridated water.

If you ingested about 8 milligrams of sodium fluoride daily for ten years or more, you would develop a well-defined disease called skeletal fluorosis, which affects bones, tendons and secondarily, the nervous system. If an infant ingested 2 milligrams of fluoride daily, they would develop dental fluorosis or ‘mottled’ teeth.

Apparently therefore, the only problems that low doses of sodium fluoride can cause are either dental fluorosis or skeletal fluorosis. The CLINICAL symptoms of these conditions are easily detected – ‘mottled’ teeth and ‘bony outgrowths’ and the calcification of tendons in skeletal fluorosis.



Experiments have shown water containing 1 to 4 parts per million can have an effect on the Central Nervous System – a mind-dulling effect! Experiments have also demonstrated that fluoride at a concentration of just 0.6 parts per million can disturb antibody production, and thus interfere with the functioning of the immune system.

And many experiments have shown that concentrations of fluoride of about 4 parts per million can damage DNA – the vital core of every living cell.

In other words, at very low concentrations, fluoride can cause subtle changes in enzyme activities, nerve action potentials, altered behavioural reaction, and the immune system…

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About Dr. Smith

Dr Geoffrey Ernest Smith, L.D.S., R.C.S. (Eng.)
Dental Surgeon, (retired)

Curriculum Vitae

  • Born: 1 November 1932, Married, 5 children, 4 grandchildren.

  • Educated: Lawrence House School, St. Annes on Sea, Lancashire. Rossall School, Fleetwood, Lancashire. University of Manchester, Turner Dental School.

  • 1956: Qualified L.D.S., R.C.S. Royal College of Surgeons. (England).

  • 1957-59 Post-Graduate Studies. Queens University, Belfast.

  • 1959-60 Travelling Fellowship, UK Medical Research Council; WHO Regional Office, Brazzaville. 

    Based University of Ibaden W. Nigeria. Field work: Nigeria, Ghana, Sierra Leone, Gambia and Liberia.

  • 1961-68 General Dental Practice, London and Dublin.

  • 1965-66 Consultant, Aspro-Nicholas, Ireland, Ltd., Dublin.

  • 1969-71 Consultant, Glaxo Group Ltd., London.

  • 1972-74 Consultant, PIA Ltd., Lopex Group, London, New York.

  • 1974-76 Consultant, Nicholas International, Slough & Melbourne.

  • 1976-79 General Dental Practice and School Dentistry, Melbourne.

  • 1979-80 Hospital Dentist, Proserpine, North Queensland.

  • 1987-88 Anthropology and Archaeology, University of Melbourne.

  • 1980 – Consultant, Environmental and Public Health.

Some relevant papers in the scientific literature:

  • NZ Med.J. 1983;96, 1067-1068.

  • Persp. Biol.Med. 1986 29, 560-

  • New Scientist 1983;5 May 286-287

  • Trends.Pharm.Sci.1986, 7, 10

  • Fluoride, 1983, Editorial, Autumn.

  • Sci.Tot.Environ. 1987, 63, 1-11.

  • Aust.Dent.J. 1984, 29, 199-200.

  • Endeavour, 1987, 11, 16-

  • J.R.Coll Gen.Pract.1984, 34, 350-351.

  • Sci.Prog.(Oxf.), 1987, 71, 23

  • Xenobiotica, 1985, 15, 177-186.

  • The Scientist, 1987, 1, 24.

  • Sci.Prog.(Oxf.), 1985, 69, 429-442.

  • Sci.Tot.Env., 1988, 68, 79-86.

  • Sci.Tot.Env. 1985, 43, 41-61.

  • NZ Med.J. 1985, 90, 556-557.

  • NZ Med.J. 1985, 98, 454-455.

  • Pers.Biol.Med. 1988, 31, 440-45

  • NZ Med.J. 1988, 100, 669-670.

  • Sci.Tot.Env., 1988, 76, 167-

  • New Scientist, 1985, 1467-, 50-51.

  • NZ Med.J. 1988, 101, 802.

  • NZ Med.l. 1985, 30, 232-233.

  • Med.Hypoth., 1986, 19, 139-

  • Med.J.Aust., 1985, 143, 283-286.

  • Mutat.Res., 1990, 241, 339-

  • Med.J.Aust., 1986, 144, 152.

  • Fusso Kenkyo, 1990, 11, 38-48

  • Lawyer, 1986, 4(3), 6. (Japanese)

  • Fluoride, 1986, 19, 105-107.

  • Probe, 1989, 31, 1-2.

  • Nature 1986; 323, 198.

  • Aust. Dent.J., 1985, 30, 232-233


The Secret War was originally prepared as a personal submission to the

Brisbane City Council’s Task Force on Fluoridation, in March 1997.

Paper : Original Paper 
Citation : Caries Res 2001;35:125-128
 Title : Fluoride Deposition in the Aged Human Pineal Gland Author(s): J. Luke
Info : Figures: 2; Tables: 0; References: 32

 Keywords : Calcium; Distribution; Fluoride; Human pineal gland; Hydroxyapatite; Pineal concretions
Abstract : The purpose was to discover whether fluoride (F) accumulates in the aged human pineal gland. The aims were to determine (a) F-concentrations of the pineal gland (wet), corresponding muscle (wet) and bone (ash); (b) calcium-concentration of the pineal.

Pineal, muscle and bone were dissected from 11 aged cadavers and assayed for F using the HMDS-facilitated diffusion, F-ion-specific electrode method. Pineal calcium was determined using atomic absorption spectroscopy.

Pineal and muscle contained 297+/-257 and 0.5+/-0.4 mg F/kg wet weight, respectively; bone contained 2,037+/-1,095 mg F/kg ash weight. The pineal contained 16,000+/-11,070 mg Ca/kg wet weight.

There was a positive correlation between pineal F and pineal Ca (r = 0.73, p<0.02)

but no correlation between pineal F and bone F.

By old age (50-70), the [ pineal gland ] has readily accumulated F

and its F/Ca ratio is higher than bone.

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 ” … Having said all of this, the absence of evidence of measurable impact is no excuse for not promulgating the best and most appropriate environmental standards.

For far too long we have provided industry with de facto subsidies by not enforcing the best and most appropriate environmental regulations. In not doing so we provide industries with special arrangements to pollute an economic advantage.

Aside of the human health or environmental responsibilities, this is simply not equitable or reasonable…”
Professor Mark P. Taylor, Environmental Science, Department of  Environment and Geography, Faculty of Science,
Macquarie University, Sydney, Australia

  ♦ Editors note: 


 are sensitive to fluorides.

Water Samples – Probably Carcinogenic

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Tooth Decay in the Developing World:

A Vaccine To Prevent Cavities


Geoffrey E. Smith

His Tooth Vaccine 

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See also:

Fluoride and the Phosphate Connection


 [This was complicated by fluoride tablets being used out side fluoridated areas.]


Fluoridation Queensland Logo




A List Of Essential Nutrients For Protection
Against Coronavirus:

Vitamin C.
Vitamin D3.
Ivermectin on hand and
Hydroxychloroquine on hand.

“Covid-19 Consumer Protection Act” ! ! !
Here is a great quick read from a medical doctor who 
is being censored for using some of the above mentions:


“… John Yiamouyiannis, whose unyielding opposition to fluoridation
was based on his painstaking independent study and research,
was a constant thorn in the side of those who promote
fluoridation but were unwilling to face him in
open debate, except in court, where
they had no other choice … “

~ Fluoride Vol. 33 No. 4 151-153 2000 In Memoriam ~




John A. Yiamouyiannis, Ph.D.


The indiscriminate fluoridation of water systems is not
the solution to the problems of tooth decay.
In the absence of sufficient Vitamin C, fluoridation
will lead to Vitamin C depletion, dental fluorosis,
  and to abnormal levels of metabolites in blood tissues.

Yiamouyiannis ff

The National Health Federation
P.O. Box 688, Monrovia, California 91017
Washington,  D.C.,  20002


Vitamin C plays an important role in the orderly deposition of fluoride into various tissues. In higher fluoride areas, Vitamin C increases fluoride excretion and normalizes soft and hard tissue fluoride levels and thus prevents the development of fluorosis.  At lower fluoride levels, Vitamin C increases the incorporation of fluoride into teeth.  Fluoridation of water systems in not the solution to optimal incorporation of fluoride into teeth; in cases of Vitamin C deficiency, fluoridation may lead to fluorosis.

– 7 August 1974 –

While fluoridation of public water systems has been advocated and encouraged by the national and state public health services, a number of questions concerning the need to add fluoride to public waters have gone unanswered.

Mother’s milk, containing as little as 0.01 to 0.05 ppm fluoride confers as much caries resistance on the child as other infants consuming 1 to 2 ppm fluoride present in commercially prepared formulas (1, 2).

In unfluoridated areas, containing natural fluoride levels of 0.1 to 0.5 ppm and even less, there exists a certain part of the population that are free of caries.  In fact, in Nigeria, a population has been found where over 98% of the population is caries free and the fluoride level in their water is within the above range (3).

In a study at Great Lakes Naval Base, the previous life-long residence of caries-free recruits, were examined to determine if any trace elements could be correlated with the low incidence of caries.

The level of fluoride in the drinking water was not implicated.

It has also been noticed that primitive areas in which the people of the area eat unrefined food have a relatively low caries rate as compared to later when these areas became “civilized” and their diets begin to consist of more refined foods.

In these cases, caries rates often soar and addition of fluoride to the water supply is unable to restore the previous caries rate (5, 6, 7).

In areas and among people where nutrition is poor, mottling is observed at levels below the 1 ppm level used to fluoridate public water systems (at levels as low as 0.4 ppm fluoride).

This has been noticed in India (8) as well as among American Negroes whose mottling rate, in the 1-ppm range is higher than that of whites in the same area.

In a comprehensive study in Japan, the fluoride levels associated with the lowest incidence of caries ranged from 0.2 to 0.4 ppm  (9).

In the 1930’s it was found that the ingestion of fluoride causes scurvy-like symptoms and that this was associated with a decrease in the Vitamin C levels of various tissues. Similarities in the symptoms of scurvy and mild fluorosis were also observed  (10, 11).

In 1954, in an area containing 0.34 to 0.8 ppm fluoride in the water, 23% of the children 4-7 years old exhibited mottling (dental fluorosis). The Vitamin C contents in blood for normal children (without mottling) averaged 0.78 mg %. In the mottled enamel group, the blood Vitamin C levels of most children were extremely low (0.15 to 0.3 mg % in 29%, and 0.0 to 0.15 mg % in 31%. Treatment of these subjects with Vitamin C brought substantial improvement  (12).

In 1964-65, the death rate of guinea pig population in Australia had reached epidemic proportion. (The Guinea pig is the only non-primate known that cannot synthesize its own Vitamin C). This death rate was eventually attributed to slightly higher levels of fluoride in feed pellets. Symptoms of sub-acute Vitamin C deficiency were observed. Fluorosis was diagnosed as the cause of death (13). In rats and mice (both of which synthesize their own Vitamin C, no such death rate was reported. U.S.P.H.S. [United States Public Health Service] experiments are performed with rats – they do not use guinea pigs (14). Both in the U.S. (15) and Russia (16) Vitamin C is recognized as being capable of retarding the development of fluorosis.

In guinea pigs exposed to fluoride, Vitamin C was found to normalize altered blood Ca, P, and sugar levels, as well as fluoride levels and ash contents in teeth and bone, and fat glycogen, and fluoride levels in the liver. Fed to men exposed to elevated fluoride uptakes, 100mg of Vitamin C increased the excretion of fluoride from 3-5.5 mg/day to 6-8.5 mg/day (17).

Most important, however, are the following findings:

1.)  in guinea pig, fluoride added to the diet cannot make teeth more insoluble (caries-resistant) than the addition of Vitamin C to the diet and-

2.)  in low fluoride areas, dietary supplementation with Vitamin C leads to fluoride deposition in teeth equal to the of higher fluoride areas (18, 19).

In conclusion, it appears that Vitamin C is and essential factor in the deposition of fluoride in, as well as the exclusion of fluoride from, various tissues in the body.

While increased fluoride in teeth had been correlated to caries-resistant of teeth, adequate Vitamin C levels in the diet in areas of 0.1 to 0.5 ppm fluoride (and even 0.01 to 0.05 ppm in the nursing infant) leads to adequate uptake by the teeth. Indeed in animals that manufacture there own Vitamin C (e.g. rats), Fluoride is found not to have a caries protective effect until it reaches levels of 10-20 ppm (14); at these levels it acts as a strong antibacterial in the mouth.

The indiscriminate fluoridation of water systems is not the solution to the problems of tooth decay. In the absence of sufficient Vitamin C, fluoridation will lead to Vitamin C depletion, dental fluorosis, and to abnormal levels of metabolites in blood tissues.

Adequate intake of Vitamin C may explain why people or populations in low fluoride areas can be caries-free.


(1)           Y. Ericsson, U. Ribelius, Caries Research 5, 78 (1971);

(2)           F.J. McClure, Personal communication.

(3)           A. Sheiham, British Dental Journal 123, 144 (1967;

(4)           J.P. Quinn, NDRI-PR-68-03, (June 1968) 11pp. US Nat.Tech.

Inf. Serv. Reo. No. AD0839 129;

(5)           S.J. Barnaud Journal 2, Med. Trop. 29, 593 (1969);

(6)           J.A. Cran, Australian Dental Journal 2, 277 (1957);

(7)           F. Prader, Schweiz. Mschr. Zahnhk. 71 885 (1961);

(8)           R.S. Nanda, Indian Journal of Dental Research 60, 1470 (1972);

(9)           Y. Imai, Koku Eisei Gakkai Zasshi 22, 144 (1972);

(10)          P.H. Phillips, J. Biol. Chem. 100,

(Proc. Am. Soc. Biol. Chem. 8) Lxxix (1933);

(11)          P.H. Phillips, F.J. Stare, C.A. Elvenhem, J. Biol. Chem. 106, 41 (1934);

(12)          N.A. Ivanova, Voprosy Okhrany Materinstva I Detstva 4, 29 (1959);

(13)          F.F.V. Atkinson, G.C. Hard, Nature 211, 429 (1966);

(14)          N.M. Stiles, National Institute Of Dental Research,

Personal Communication;

(15)        J.W. Suttie, P.H. Phillips, The Pharmacology and Toxicology of Fluorine,

                J.C. Muhler, M.K. Hine, Ed.

(Bloomington, Indiana University Press, 1959) pp 70-7;

(16)          V.S. Andreeva Voprosy Okhrany Materinstva I Detstva 4, 25 (1959);

(17)          R.D. Gabovich, P.N. Maistruck, Voprosy Pitaniya 22, 32 (1963);

(18)          D. J. Thompson, P. H. Phillips, J. Dent. Res. 45, 845 (1966);

(19)          D. Triers, C.G. Elliott, M.D. Smith, J. K. Dent. Res. 47, 1171 (1968);

(20)       W. Buttner, Advances in fluorine Research and Dental Caries Prevention,  J. L. Hardwick, H.R.Held, K.G. Konig, Ed. (New York Pergamon Press, 1965) pp. 19-30;

(21)        Hardwick & Bunting, J. Dent. Res. 50 (Supplement, Pt. 1), 1212 (1971)



John Y


It is also worth noting the link between fluoride and other micronutrients;
in particular Vitamin C. – Vitamin C is an essential nutrient for humans
and is required by the body to form collagen in bones,
cartilage, muscle and blood vessels. It also aids in
the absorption of iron.[449]

     It has been documented that in the absence of sufficient Vitamin C, water fluoridation will lead to Vitamin C depletion, dental fluorosis and to abnormal levels of metabolites in blood tissues.[450] Animal studies have also demonstrated the link between Vitamin C and fluoride intake.[451] The findings of this latter study support the previous observations noted by Yiamouyiannis. This fact should be of particular interest to the Irish health authority as a national health survey has documented the inadequacy of  Vitamin C intake in boys and girls aged from 5 to 12 years in Ireland.[452] A more recent study among adults aged 65 years and over, similarly showed a low intake of  Vitamin C in addition to inadequate intakes of calcium, Vitamin A and Vitamin D.[453]

Alarmingly, it is possible that water fluoridation may itself  be contributing to Vitamin C depletion as well as dental fluorosis in Ireland. Interestingly, it has also been observed that in low fluoride areas, dietary supplementation with Vitamin C leads to fluoride deposition in teeth equal to that of higher fluoride areas with water fluoridation.[454],[455] It is plausible therefore, to assume, that exposure to fluoride compounds in drinking water may increase the associated risk of  Vitamin C depletion, a contributory factor associated in the development of both dental fluorosis and periodontal disease.

Indeed the coexistence of fluorosis and signs of  Vitamin C deficiency have been reported[456],[457] and it has been suggested that low concentrations of  Vitamins A, C and D tend to worsen the symptoms of fluorosis and accelerate the development of fluoride toxicosis.[458]

Footnotes    (↵ returns to text)

1.) 449 Mayo Foundation for Medical Education and Research↵

2.) 450 Vitamin C and Fluoridation- John A. Yiamouyiannis Ph.D.↵

3.) 451 Verma RJ, Sherlin DM , Fluoride and Vitamin C, Human & Experimental Toxicology, 2001 Dec;20(12):619-23.↵

4.) 452 Irish Universities Nutrition Alliance (IUNA), 2001, Adequacy of micronutrient Intakes in Ireland, Results from the National Food Consumption Surveys,↵

5.) 453 Irish Universities Nutrition alliance (2008), National Teens Food Survey↵

6.) 454 D. J. Thompson, P. H. Phillips, Journal of Dental Research. 45, 845 (1966).↵

7.) 455 D. Triers, C.G. Elliott, M.D. Smith, J. K. Dent. Res. 47, 1171 (1968).↵

8.) 456 Krishnamachari K. A. V. R, and Laxmaiah N., Lack of effect of massive dose of vitamin C on fluoride excretion in fluorosis during a short clinical trial,
The American Journal of Clinical Nutrition 28: November 1975, pp. 1234-1236.↵

9.) 457 Pandit, C. G., I. N. S. Raghavachari, D. S. Rao And V. Krishnamurthy. Endemic fluorosis in South India. A study of the factors involved in the production of mottled anamel in children and severe bone manifestations in adults. Indian J. Med. Res. 28: 559, 1940.↵

458 Suttie JW, Phillips PH: Fluoride ingestion and vitamin metabolism. In Fluorine and Dental Health: the Pharmacology and Toxicology of Fluorine. JC Muhler and MK Hine eds.

BloomingtonI, N: Indiana University Press, 1959, pp 70-77.↵


Vitamin C – Stone-Pauling-Kalokerinous & Ebola


Dr. Archie Kalokerinos, MBBS, FACNEM-


 Vitamin C Foundation


5 Guinea pigs

Guinea pigs have a requirement for vitamin C,
because they can not cannot manufacture or store vitamin C
(lack of the enzyme L-gulono-gamma-lactone oxidase).

…Then I happened across a report on fluoride and it’s negative effect
on collagen synthesis. I immediately switched to water without fluoride
and the results were magical. This may be unique to my chemistry.
But given the volume of research, it might be worth recommending…

Vitamin C Reduction of F. Induced Embro-Toxicity In Rats 

Dr. Archie Kalokerinos – Interview

 See also  →  “Healing is Voltage” 

See also  Book #54 on our

Fluoridation Book List




For 50-69 years old people from Basle the cancer death rate
was declining before water fluoridation and has changed
to a raising trend of cancer deaths after the
introduction of water fluoridation.

elevated C. Basel f

Basel Stops F. f

Switzerland Ends Fluoridation After 41 Years…

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Relations Between Water Fluoridation and Cancer in man and
animals and Effectiveness in USA and Europe.
© by Rudolf Ziegelbecker, Graz

Extract from the above web site


Elevated Cancer Increments In Basle

After The Introduction Of Water Fluoridation

… Also water fluoridation in Basle is useless, as shown by KREUZER and myself (ZIEGELBECKER) at the SGSG-conference in Basle on 2 December 1989. The lessening of dental caries in Basle results from other reasons than fluoridation. The analyses of cancer data, however, which were made available to me by the sanitary department, yielded (in agreement with the results from far more comprehensive data from the USA) an alarming connection between fluoridation and cancer deaths in Basle. The diagram shows the change of the percentage of female cancer deaths among the female citizens over time, before and after the introduction of water fluoridation in Basle. During the 13 years before water fluoridation (1950-1962) the increase factor of the percentage of female cancer deaths was about 2.38. After introduction of the drinking water fluoridation (in May 1962) the increase factor raised in the following 21 years almost suddenly to 39.74 which is the 16-times larger value! With the males the situation is similar, the increase factor raised during the same time from 23.58 to 49.7.

3. Erhöhte Krebszuwächse in Basel nachEinführung der Fluoridierung:Auch

die Basler Wasserfluoridierung istnutzlos, wie von KREUZER und mir

auf der Basler SGSG-Tagung am 2. Dez. 1989gezeigt wurde. Der

Kariesrückgang in Basel hat andere Gründe als dieFluoridierung. Wohl

aber ergab die Analyse der mir vom Sanitätsdepartement zurVerfügung

gestellten Krebsdaten (in Übereinstimmung mit den Ergebnissen aus

weitumfangreicheren Daten aus denUSA) einen besorgniserregenden

Zusammenhangzwischen der Fluoridierung und den Krebstodesfällen in

Basel. Das nachfolgendeDiagramm zeigt die zeitliche Änderung des

Anteils der weiblichen Krebstoten ander weiblichen Bevölkerung vor

und nach Einführung der Wasserfluoridierung inBasel. In den 13

Jahren vor der TWF (1950-1962) lag der Zuwachsfaktor desAnteils

weiblicher Krebstoter bei 2,38. Nach Einführung der TWF (im Mai

1962)erhöhte sich der Zuwachsfaktor in den folgenden 21 Jahren

(1963-1983) nahezuschlagartig auf 39,74; also um das 16-fache. Bei

den Männern ist es ähnlich: DerZuwachsfaktor stieg in der gleichen

Zeit von 23,58 auf 49,7 an.

Fazit:Die falschen Lehrmeinungen über die Fluoridierung gehören

dringendkorrigiert, die Zensur der Kritik und der Boykott durch

dieZahnärzteorganisationen und “Fluorkommissionen” abgeschafft. Die

Fortsetzung dernutzlosen und Fluorose- sowie Krebs fördernden,

gesundheitsbelastendenTrinkwasserfluoridierung in Basel erscheint


Résumé: The wrong doctrines about fluoridation should be urgently corrected, the censorship of critique and the boycott by dentists’ organizations and “fluoridation commissions” must be abolished. The continuation (remark: water fluoridation was stopped in Basle in 2003) of the [ineffectiveness of] effectless fluoridaton in Basle which promotes fluorosis and cancer and strains health appears to be  [irresponsible?] unresponsible.

More details from the attached Compendium of Summaries number 57, p.45:  Bei den 50 -69-jährigen Baslern war die Krebstodesrate vor Beginn der TWF überhaupt fallendund ist dieser Trend nach Einführung der TWF in einen steigenden Trend an Krebstotenumgeschlagen.


For 50-69 years old people from Basle the cancer death rate was declining before water fluoridation and has changed to a raising trend of cancer deaths after introduction of the drinking water fluoridation.

4. Material sent to the EFSA by my father (who died in January 2009)  already in 2006 which contains a heavy critique of the EU’s Scientific Committees’ “Opinions” on the tolerable upper intake level of fluorides,

and in details even more competent information needed for an objective
risk assessment of chemicals used for drinking water fluoridation –
if desired I could send this material also to you.


       Rudolf Ziegelbecker Jr.

German version: FluoridBSTW

Fluorid im Basler Trinkwasser
Gemeinsame Untersuchung des Kantonalen Labors
Basel-Stadt und des Wasserlabors IWB

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R. Ziegelbecker Grazf

Obituary →  Fluoridation – Rudolf Ziegelbecker

Large file 


Large file ↓  Rudolf Ziegelbecker Jr.  ↓



Ovarian Cancer & Fluoride

And more on cancer and fluoride ↓ 

hear tape below






We are told by the Queensland Government, that no adverse effects
have been seen in Townsville from fluoridation, but Queensland
Health has never provided any health studies to justify this claim!

‘The Townsville Report’ was used as part of ‘The Evidence’ to
 justify the fluoridation of Queensland’s public water supply.

Factors Often Overlooked In Fluoridation Research

Map of queensland

The Original


~ Fluoridated on 2nd August 1965 ~


A Survey by W. Vidoroni, G.S. Sternberg and G.N. Davies 
Division of Dental Services, Department of Health,
Brisbane, Queensland, Australia, and Dental School,
University of Queensland, Turbot St, Brisbane, Australia.

            Our criticisms:


❝ …Another feature of Townsville is the mobility of the population.
This is reflected by the fact that although 900 children were examined
only 317 or 35 per cent could be classified as lifetime residents, due to large RAAF Base.
Data on the remaining non-lifetime residents will be reported in a separate paper. ❞ 

 So Where Is It ?

This additional data was promised in the report,
but despite numerous telephone requests to one
of  the authors of this report at the time –
W. Videroni, none was ever provided.

 More criticisms:


 …In addition to assessing the dental caries experience
of the children, data was collected on the following:

fluorosis, idiopathic enamel opacities, enamel hypoplasia, tetracycline staining, traumatic injuries to teeth, oral mucosal disease, pathology of bone, dentofacial anomalies, periodontal status (soft deposits, calculus, intense gingivitis), need for periodontal treatment, prosthetic status, denture requirements, conditions needing immediate attention and fillings and extractions required for individual teeth. These data will be reported in a subsequent publication. ❞.

So Where Is It ?

This additional data was promised in the report, but despite
numerous telephone requests to one of the authors of this report
at the time – W. Videroni – none was ever provided.

Were the RAAF & Lavarack Barracks Base transient residents included?

More Comments:

•  Lost in this long list fluoridation negative outcomes,
 is one of major concern, Dental Fluorosis:

•  Dental fluorosis is not reversible and in more severe cases,
causes disfigurement, pitting, staining and positional defects.

  Dental fluorosis is also associated with skeletal fluorosis.

 dental problem in Australia.

Qld. Health – Demographics 

As can been seen from the newspaper cutting below,
Fluoridation of Townsville’s water supply
has not had the expected results:

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Townsville Kicked in the teeth

CHILDREN will no longer receive free dental care at school.

    The Queensland Government has stopped offering treatment in its school dental vans because there is not enough staff to man them. A letter sent out through schools this week says the vans would only examine students and provide a report to parents on the state of their teeth. They will then have to seek treatment at private dental practices or take their chances with long public dental waiting lists. “Due to expected demand for treatment you may have to wait for a while for an appointment for non-urgent treatment,” the letter said.Queensland Parents and Citizens Association Northern Region president Mick Cutler said parents wanted to see the dental van visits saved. “We would be disappointed but we also understand the reality of change,” Mr Cutler said. “We’d hope they have the capacity to recruit more staff. “When Queensland children have the highest rate of tooth decay in Australia of course we would like to see the dental vans continue. “We also need to continue the education of our children on better eating habits that don’t contribute to (poor dental health).” In the past all Queensland children from four years old to year 10 at school have been eligible for public sector oral health services. The non-emergency dental treatment is provided at schools through dental clinics and dental vans on a rotational basis. Parents sign a consent form allowing the dentists to check for cavities and put in fillings, if necessary. In February 2006 the Townsville Bulletin reported annual free dental check-ups for students across the twin cities would be stretched out to every two years. At the time a health spokeswoman blamed the wait on the twin cities’ population surge. Leading dental researcher Dr Kerrod Hallett said he had been shocked by the sorry state of  Townsville tots’ teeth on his last visit to the city. Dr Hallett is trialling a new mouth guard, designed to stop tooth decay, at Vincent State School. The paediatric dentist hopes the trial will stop young children from suffering the pain of tooth aches and rotting teeth. “I examined 16 children on the first day,” he said. “Out of those one had a healthy mouth. “I was quite shocked myself.” Caption: BRUSHED . . . children such as Annabella Schmid, 4, will no longer receive treatment through school dental vans.

Illus: Photo Library Heading: Health General State Government Health Local Section:
… © Copyright …

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F. the Townsville Report heading


~  UPDATE  NOVEMBER  2010  ~
Townsville has been fluoridated for 40 + years
by the Townsville Council.

… Earlier this year [2012] we asked Townsville Council if they knew of any general health studies that had been done in Townsville for water fluoridation the then Mayor and Director’s representative replied:

  Townsville Council …. not aware of any specific studies (fluoridation health studies) not commissioned any – not aware of any …. contact Qld. Health to see if they have done any …

We are told by the Queensland Government that no adverse effects have been seen in Townsville from fluoridation, but Queensland Health has never provided any health studies to justify this claim – the reason why – they have never looked at anything but teeth. Fluoride, when ingested travels throughout the body in the blood plasma, but supposedly it only  beneficially affects teeth? If you don’t look – you don’t find — Just keep on parroting fluoridation is “safe and effective”

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Full 91 page original text→ HERE ←

Extract Davies page 16

Full 91 page original text→ HERE 

Links To Fluoridation Webs Sites Around The World

Dental Statistics -OECD Non-F. Countries



 Fluoridation  Black Americans, Indigenous Australians & Shri Lankans




















…What needs to be done? – An ideal way would be to supplement
the diet of 
children in the endemic areas with calcium,
magnesium and vitamin C. 
This was also the basis
of Chinese research work in early 1990’s …

Green strip


Reverse Osmosis fluoride free
water in South East Queensland:




The upper limit of fluoride level in drinking water for a  tropical
country like India is 0.5 ppm – (Less is better).

The upper limit of safe total intake of fluoride from food and water per day
for an adult is 5 milligrams (WHO-2002).

The TOTAL daily intake through water and food determines the development of fluorosis.

First ever cases of endemic skeletal fluorosis and its neurological manifestations in the  world were recorded from Podili, Darsi and Kanigiri areas of Andhra Pradesh in 1937. Subsequently cases of fluorosis were recorded from Nalgonda and other areas of the Andhra Pradesh state and other parts of India. It is now estimated that 60 million people are living in these endemic areas and are at risk of contacting the disease and 2 million people are crippled because of it.

The incidence of fluorosis affected districts in India are listed alphabetically: Assam2; Andhra Pradesh= 17; lBihart=8; Delhi4; Gujarat= All except Dang; Haiyana 12; Jammu & Kashmir=1; Kamataka 14; KeraI=3; Maharashtra= 10; Madhya Pradesh= 10; Orissa= 3; Punjab=13; Rajasthan= All 32 districts; Tamil 4adu 8; Uttar Pradesh= 7 and West Benga=4. Hence, skeletal fluorosis continues to be a major public health problem in India.

The factors, which govern the development of fluorosis, are the following:

1. )    High levels of fluoride in drinking water supplies and in the foodstuffs grown in these endemic areas.

2.  )   Tropical weather and hard manual labor by affecting the intake of water.

3.  )   Poor nutrition and diets deficient in their content of calcium, magnesium and vitamin C aggravate fluoride toxicity. High intake of calcium reduces the amount of absorbed into the bones. Magnesium has peculiar relationship with fluoride and its optimum intake helps in elimination of fluoride from the body. Vitamin C is beneficial in some way in reducing fluoride toxicity. Diets deficient in calories and calcium intake increase the incidence of fluorosis (WHO-2002).

4.  )  Renal disease aggravates fluorosis by increased deposition of fluoride in the bones.
A diseased kidney cannot handle fluoride excretion from the body and hence its increased deposition in the bones.
See also → HERE

See also ↓

Ameliorative Effects of Quercetin on Sodium Fluoride-Induced Oxidative Stress in Rat’s Kidney

5.  )  Presence of abnormal amounts of certain trace elements in the drinking water supplies such as strontium, uranium etc. Strontium levels in drinking water supplies in some endemic areas are high and strontium is a bone-seeking element like fluoride and both these aggravate the bony changes.

In a study of 94 drinking water samples from the endemic fluorotic villages of Prakasam and Nalgonda districts the fluoride and strontium levels are as follows: Fluoride levels ranged between 0.1 to 9.5 ppm for a mean of 2.009 ppm and strontium levels ranged between 4.91 to 9931.74 ppb for a mean of 1670.02 parts per billion. Any water strontium levels of over 1000 ppb are suspect. Increased levels of fluoride and strontium appear to playa role in the severe forms of fluorosis that is witnessed in some endemic areas of fluorosis in Andhra Pradesh especially in Prakasam where fluoride levels are not very high. Some elements like uranium are nephrotoxic and may aggravate fluorosis problem. Levels of trace elements such uranium, selenium, zinc, iron, lithium, lead, barium, aluminum etc were in abnormal concentrations in some of the drinking water supplies of villages in Prakasam and Nalgonda districts of Andhra Pradesh. Aluminum is known to increase fluoride absorption from the gut and hence its abnormal concentration in water supplies is harmful. The role of these other elements except those of strontium, aluminum and uranium in fluoride metabolism are not known at present time.

De fluoridation plants are based on adding aluminum compounds to the water containing high levels of fluoride. It may be noted hundreds of crores of rupees were spent on erecting these plants in 1980’s 1990’s and not a single one of them are working now. So is the fate of household de fluoridation units. Besides there is the risk of increased intake of aluminum which is being incriminated for the causation of Alzheimer’s disease as well as motor neuron disease for which there is no therapy like AIDS disease. Tea contains exceptionally high content of fluoride and each cup may add up to 2 milligrams of fluoride. Ideal solution for skeletal fluorosis is its prevention by providing safe drinking water and providing such water For cultivation of crops in these endemic areas, This appears to be only a very distant dream. Government of India envisaged health for all by 2000 in 1987 AD and planned provision of safe drinking water to all villages by that date. The plans failed miserably and now the authorities have become more pragmatic. One’ of the ‘millennium development goals’ of 2002 is to provide safe drinking water to half the rural population by 2015. Hence prevention of fluorosis is not possible in the near future and at least efforts must be made to lessen the suffering of the people by improving their nutrition.

The situation in certain parts of Andhra such as Nalgonda is very grim. When Daver first recorded cases of skeletal fluorosis from this region in 1945 AD and by Siddique in 1955 AD, there were no children with deformed limbs. These cases with deformed limbs were recorded in 1970’s. Rural nutrition in the past few decades has gotten worse in many parts of state and in other parts of the country. National institute of nutrition conducted surveys of status of rural nutrition in 715 villages of seven states in 2002 and 2003. Eighty villages in AP were studied and AP has the distinction of having highest incidence of dental fluorosis among seven states. The results of nutrition study are contained in National nutrition monitoring bureau reports 21 and 22, which reveal the appalling state of nutrition of rural population in the country. Sixty percent of the children below 6 years of age are malnourished and a third of them suffer from severe under weight problem. Only a third of the children studied were getting an optimum diet. Calcium intake is exceptionally low in all states and calcium does playa big role in Nalgonda having very high incidence of skeletal fluorosis in our country. Drinking water supplies of Nalgonda have a high content of fluoride because the calcium content of Nalgonda soils and rocks is low. This allows more fluoride to seep in to the water supplies (Netherland study- 1986). Siddiqui study revealed that avenge daily intake of calcium in Nalgonda individuals was only 300mg whereas it was 900 mg in Punjab. NNMB report of 2002 reveals that calcium intake of rural population is appallingly low in Andhra and hardly averages around 300 milligrams whereas it should be around 800-1000 milligrams in growing children. Hence, there are no children with deformed limbs in Punjab villages with simdar levels of fluoride in drinking water supplies. Nutrition status study of 50 children in Sharbanapuram village of Aler Mandal of Naigonda revealed that only one boy was normal as per BMI and percentile studies. Midday meal scheme has not helped these children in Nalgonda in this regard. Children hardly get 300 calories of diet and most of the times the food supplied is not even hygienically good. National family health survey-Ill by the government of India in 2006 in 29 states revealed that 45 % of the children below are undernourished and 57 % of women are anemic. All government policies to improve nutrition of rural folk have been a dismal failure so far.

What needs to be done? One has to be pragmatic. Ideal way would be to supplement the diet of children in the endemic areas with calcium, magnesium and vitamin C. This was also the basis of Chinese research work in early 1990’s. The best way would be to give a glass of milk and a banana to every school- going child in these endemic areas. Milk is perishable and can be adulterated. A pouch containing 300 ml of milk becomes very expensive. A tablet containing 5,0 milligrams of calcium, 300 milligrams of magnesium and 40 milligrams of vitamin C with vitamin D to improve absorption of calcium would be cheap and practical in addition to midday meal scheme, which needs to be improved. Since anemia is widely prevalent in rural population it may be advisable to add iron also to this tablet. The skeletal fluorosis was discovered in our country in Prakasam district seventy ears ago and this continues to be major health problem even today. All efforts to contain this disease have been a dismal failure so far. Unless pragmatic steps are taken this problem going to affect the lives of millions of people in our country for a long time to come.

There are three factors for the causation of skeletal fluorosis, Water, food and nutrition. Ask for Krishna water for drinking and cooking for every fluorotic village. Try for Krishna water for cultivation in future. Improve the nutrition of the population especially those of growing children by whatever means practicable. Avoid drinks such as tea, which is very rich in fluoride and also seafoods. Avoid analgesics, which could damage the kidneys and aggravate the problem in these areas where studies reveal a higher incidence of kidney disease.


A diet rich in calcium, magnesium and vitamin C would greatly benefit the population living in areas where soil and water fluoride levels are high while measures to provide safe drinking water could benefit by reducing fluoride intake, a diet enhancing calcium, magnesium and vitamin C intakes would considerably reduce the basic pathology associated with fluorosis. However these food sources should be grown in no fluoride areas and provided for consumption in region with fluorosis.

Though there are several food sources only those which are commonly consumed in region of AP and which are affordable to the lower socio-economic groups are suggested. The resource for this compilation is the:-


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Volcanoes and fluorosis 

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Black Tea Source, Production, & Consumption:
Assessment of Health Risks of Fluoride Intake In New Zealand +

Very Comprehensive ↑

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Nutrient Data Laboratory Beltsville 
Human Nutrition Research Center Agricultural Research Service
U.S. Department of Agriculture

Contains fluoride values for 427 foods across 23 food groups.

Fluoride Content Of Major Foods

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“Nutritive Value of Indian Foods” 

See also →
 Selenium Deficiencies And Mass Extinctions



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Fluorides are filtered from the blood by the kidneys and excreted in the
Renal function is quite important and may be closely related to the
differences of susceptibilities to F toxicity in the general population.
F may accumulate more readily in individuals who have impaired
renal function and, therefore, affects them more seriously. 

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Dental F. 3 Photos DENTAL FLUOROSIS is the visible mottling of teeth.

(see photos above)



   Dental fluorosis also indicates the deposition of fluoride in the bones – skeletal fluorosis, causing brittleness in later life, – especially hip fractures in humans. It can occur after teeth formation but will NOT be recorded on the teeth after they are formed. Dental fluorosis is not reversible and in more severe cases, causes disfigurement, pitting, staining, positional defects, (malocclusion) brittleness, and facilitates decay.

In these cases fluoride causes the exact problem it is supposed to prevent.

The eruption of teeth, animal and human, is delayed in the presents of fluorides.

This delay accounts for the original statistical error – inferring that fluoride reduces decay in children’s teeth [it just] skews the data…



The fallacy on which the claim that fluoridation reduces dental decay relies almost exclusively on the direct comparison of the raw data on the prevalence of dental caries in 5 year-old children in fluoridated and un-fluoridated areas.

Indeed, in some locations – but by no means all – there may appear to be a slight difference, but this vanishes entirely in older children who have their permanent teeth.

Predictably, pro-fluoridation lobbyists invariably avoid referring to this far more relevant age group in their attempts to convince listeners of the supposed beneficial effects of fluoride.

 Making a direct comparison of children’s teeth is difficult because there are many things that can affect a child’s dental health. But the greatest problem with this comparison is that fluoridation inhibits the rate at which the teeth of young children emerge from their gums.

 The delay may be from a few months to well over a year, so in fluoridated areas, the teeth of young children are fewer in number, and younger, than those of children in unfluoridated areas. This renders any comparison of crude data on dental decay in children of the same age completely unscientific and misleading – in scientific terms, it is a comparison between unmatched samples, and entirely meaningless.

 It is therefore improper to compare the dental health of same-age young children, because the effects of fluoridation must be compared on teeth of the same dental age, and not simply according to the chronological age of the children themselves...

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The eruption of teeth, animal and human, is delayed in the presents of fluorides.
This delay accounts for the original statistical error – inferring 
that fluoride reduces decay in children’s teeth
– it just skews the data…

Thais has black teeth



1.  Drink Rain Water (from tanks).
This is difficult if you live in a town house, a home unit or a rented property.

2.  Use Reverse Osmosis Filtration.*  (RO) This three-stage system it will need regular servicing and wastes 2-4 times the saved water to continually wash the filtering membranes. See image below *

3.  Use Steam Distillation. These systems produce clean water using electricity.

4.  Buy Bottled Water.

Bottled water is responsible for over 60,000 tones of greenhouse gas emissions every year. Only 35% of plastic PET gets recycled in Australia the rest becomes landfill. Many brands of bottled water are not fluoride free even if they are labelled “Spring Water.” There is a push to fluoridate bottled water. We can presume this will solve the problem for the bottlers who would otherwise need to install and maintain R.O. filters in their bottling factories, and make some dentists happy…

6.  Move To A Town With No Fluoride In The Water.
Many Australians allergic to fluoride have moved to Queensland only to find that Queensland is now being fluoridated… (thank you Anna)


1.)  Absorption Though Skin:- Cleaning, swimming, showering/bathing and clothes washing will result in some assimilation through the skin.

2.)  Boiling water will NOT remove fluoride; indeed it will increase the concentration. This will occur due to evaporation e.g. electric jugs, kettles, aquariums and (wet, atmospheric) cooling towers.

3.)  Boiling water will remove chlorine, but NOT fluorides.

4.)  Carbon filters will only remove fluoride for the first 30-50 liters.
There is some fluoride reduction (30-40%) progressively failing over time.
Carbon filters are therefore NOT an option for fluoride removal.

Reverse osmosis relies on pressure and a semi-permeable membrane to
remove contaminants from water. It can remove around 90% fluoride
in your water. Contaminants are trapped by the membrane and
flushed away as wastewater. The process requires between
2 and 4 litres of water to produce 1 litre of clean water.

                                          See also↓

Clean Drinking Water Out Of Thin Air


Toilet To Tap – Explanation

The best way to remove fluoride from water,
is to remove those from office who put it
there, and those who refuse to remove it –
Always vote them out of office:
If they won’t give you clean water,
do not expect clean politics !




Doug N Everingham MB, BS (Univ Syd 1946)

Member, Australian House of Representatives 1967-75 and 1977-84

Australian Minister for Health 1972-75

Vice-President [West Pacific Region’s nominee] at
World Health Assembly, Geneva 1975.

Doug. Everingham f

 Doug Everingham died on 24th August 2017- aged 94 


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With the kind permission of Dr. Everingham this recent letter is
[has] being forwarded to all Queensland MPs and others by:

Queenslanders Against Water Fluoridation Inc.  

Douglas Nixon ‘Doug’ Everingham [Born: 25 June 1923, Australia – Died: 24 August 2017, Brisbane]
a former Australian politician and minister. Everingham graduated with a Bachelor of Medicine and Surgery from the University of Sydney in 1946 and worked in public and private hospitals and as a family doctor.[1] Everingham was elected the Australian Labor Party member for the House of Representatives seat of Capricornia at a by-election in 1967.

In the Gough Whitlam government of 1972–75 he served as  Minister for Health.

From Dr. Doug Everingham Federal Health Minister ~

❝ As a family doctor and often Acting Government Medical Officer in Rockhampton  over in the 1950s I wrote a letter praising fluoridation published in the local The Morning Bulletin. This prompted the late Jack Harding and others to show me books that warned of fluoridation risks. I read those books and have continued to look at reports for and against fluoridation in medical journals and elsewhere.

Reports still not disproved suggest involvement of fluoride medication in contributing after some years of consumption to reduced immunities or increased intensity or numbers of cases of disorders in human and animal development or function of bones, joints, brain, thyroid and other tissues. Many of the water supplies now fluoridated were earlier showing dental signs of fluoride overdose in a significant proportion of their consumers, and fluoridation increases the prevalence and intensity of such dental fluorosis.

Reports criticize poor experimental and statistical methods in most surveys that favor water fluoridation. Official policy promoters overwhelmingly concede that public water supplies with 4 ppm (parts per million) fluoride are a hazard. Lower limits have been suggested for infants, sufferers from certain metabolic problems and persons exposed to hot conditions. No minimum concentration of fluoride in communal water supplies or in individual fluoride intake has been shown as necessary to produce fluorosis-free, cares-free teeth. despite repeated calls for such assessment by official reports.

Many studies suggest that the only proven caries-preventing function of fluoride lies in carefully applying toothpaste and dental therapy to tooth enamel, not in swallowing fluoride. Many authors want all artificial fluoridation stopped and natural water supplies limited to some unspecified fluoride level below 1 ppm.

Such authors include professionals formerly in charge of government promotion or direction of fluoridation. Among these are the former Dean of Melbourne’s dental school, Sir Arthur Amies, Dr John Colquhoun of New Zealand; and others in the few countries that still promote fluoridation, as well as many that have abandoned or avoided it.

Associate Professor Hardy Limeback, PhD, BSc, Doctor of Dental Surgery, Head of Preventive Dentistry, University of Toronto and Past President, Canadian Association for Dental Research, is typical of ever more researchers who have turned against fluoridation. In 1999 he apologized for inadvertently misleading colleagues and students, saying “For the past 15 years I had refused to study the toxicology information that is readily available to anyone. Poisoning our children was the furthest thing from my mind.”

It thus took that eminent expert 15 years to face plain and crucial scientific facts that were avoided and disparaged by traditional authorities and some industrial sponsors. I took me 10 years after graduating. It is time for remaining fluoridation promoters to get past collecting titles of supporting political authorities and stop ridiculing opponents as if they were all dupes, cranks, quacks or dimwits. They may then make a similar apology. ❞

Doug N Everingham MB, BS (Univ Syd 1946)

Member, Australian House of Representatives 1967-75 and 1977-84

Australian Minister for Health 1972-75

Vice-President [West Pacific Region’s nominee] at

World Health Assembly, Geneva 1975.

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Post script from Queenslanders Against Water Fluoridation: ❝  The Federal Labor Party has initiatives in Oral Health that they can be very proud of (starting the School Dental Service – Gough Whitlam sent 100 dental therapists to New Zealand for training to get it started quickly) and Labor introduced the Commonwealth Oral Dental Health Scheme. ❞

green line  Fluoride [ & Autism Spectrum Disorders ]

❝ I have been researching fluoride in water for the “neuro-typical” population and have found many risks for this population let alone the ASD population.  It would make sense that those with ASD would be just as susceptible if not more as they tend to have physiological systems that are more sensitive.  Here is a letter written to the Queensland Government regarding this issue.  There are many more doctors who have signed the letter but I have only included the main author.  As well here is a website that may be beneficial in your quest for more information.             

The Premier of Queensland, 
Queensland Minister for Health,
 Lord Mayor of Brisbane, 
Deputy Lord Mayor of Brisbane 
Councillors, all Cities and Shires of Queensland.

We wish to express our grave concerns regarding renewed proposals to fluoridate Queensland’s water supplies. Fluoridation began at a time when asbestos lined our pipes, when lead was added to petrol, and DDT was regarded as safe and effective. These chemicals have now been banned, but fluoridation remains untouched 
( Research is now raising doubts about fluoridation’s effectiveness. Data compiled by the World Health Organization shows tooth decay has declined at a similar rate in all Western Countries, irrespective of each country’s water or salt fluoridation status
 ( New evidence for potential serious harm from long-term fluoride ingestion is also emerging. This evidence is summarised on the following web sites: and .It is extraordinary that the same people who reject abundant global evidence about the association between fluoride ingestion and organ damage (e.g. to bones, teeth, glands, or immune system), embrace the questionable science used to support its benefits. They ignore the many confounding possible influences in this research. This includes sugar consumption (averaging 1 kg per week per person in Australia) much of which is hidden, other nutritional influences, general dental hygiene, use of floss and movements in population.Recent studies by several eminent researchers strongly suggest that fluoride works primarily by topical means through direct action on the surface of the teeth via toothpaste, or gels used in dental treatments ( see: XIII, ‘topical versus systemic effects’), (Centres for Disease Control {CDC}1999 and 2001). Ingestion of fluoride through drinking fluoridated water is not essential nor effective for caries reduction.Hardy Limeback, PhD, DDS, BSc, (Associate Professor and Head, Preventive Dentistry, University of Toronto and Past President of the Canadian Association for Dental Research) apologised in 1999 for inadvertently misleading both his colleagues and dental students in his support for fluoridation. Addressing them he concluded: “For the past 15 years, I had refused to study the toxicology information that is readily available to anyone. Poisoning our children was the furthest thing from my mind”( fluoridating agent most commonly used is a hazardous contaminated industrial – grade product from the phosphate fertilizer industry  ( overview .htm). We strongly reject the notion that placing a chronic poison in our water supplies is clever preventive medicine.We represent 1500 doctors, dentists, scientists and others working in the interests of public health who are primarily interested in prevention and early intervention in disease processes.The question of fluoridation was considered with care, openness and good representation by the Lord Mayor’s Task Force in 1997 which came down clearly against fluoridating Brisbane’s water supply ( ). It is astounding that this issue could again be raised in Queensland in an apparently coordinated national campaign. No new evidence has been put forward to our knowledge that would warrant a revision of the 1997 decision.We will urge Queenslanders to study the evidence showing potential serious harm from long – term fluoride ingestion and to inform their councilors and state members how they feel about this matter. ❞

Yours sincerely,

Dr Doug Everingham MBBS

1972-75 Australian Minister for Health

World Health Assembly 1975

National Delegation Leader
 and Vice-President for

Western Pacific WHO Region

More ↓

More  → HERE

WHO and ‘Dodgy’ Fluoridation Endorsement


Fluoridation Canberra 1963 [Killen not support]