Fluoride destroys 66 of the 83 known enzymes
and is thus a blocker to re-enamelization.
- The Obsolete Practice of Water Fluoridation -
Last update: 14 Sept. 2019
This section of our site will provide information on how to grow good teeth and how to care for them. Please revisit this post as we shall be adding more information in the near future.
Ramiel Nagel author of – ‘CURE TOOTH DECAY’
Including information on: Streptococcus mitis, Streptococcus mutans, Streptococcus oralis, and Streptococcus salivarius ↓
LARGE FILE → HERE ←
FLUORIDE DESTROYS ENZYMES:
The re-enamelization (remineralization) of tooth enamel requires the enzyme adenosine diphosphatase, as well as supplemental calcium and phosphate in the diet. Furthermore, remineralization of the dentine underneath the enamel requires calcium, phosphate and adenosine diphosphatase. Dentine has been shown to repair also, even after the onset of bacterial attack. Be aware, though, that re-enamelization (remineralization) cannot occur without adenosine diphsphatase and perhaps other enzymes. Fluoride destroys 66 of the 83 known enzymes and is thus a blocker to re-enamelization.
(Cholinesterase is compromised at .0095 ppm and Calcium adenosine diphosphatase at .00037 ppm of fluoride.)
→ → → CLICK HERE ← ← ←
See extracts below ↓ ↓ ↓
→ BIOFILMS ←
“…Biofilms can attach to a surface such as a tooth, or rock, and may include a single species or a diverse group of microorganisms. The biofilm bacteria can share nutrients and are sheltered from harmful factors in the environment, such as desiccation, antibiotics, and a host body’s immune system…”Biofilms can form on the teeth of most animals as dental plaque, where they may cause tooth decay and gum disease.
[ MMS (a) - MMS (b) – MMS (c) will control biofilms]
There is a need for Better Evaluation of fluoridation.
The “tooth patch” is a hard-wearing and ultra-flexible material made from hydroxyapatite, the main mineral in tooth enamel, that could also mean an end to sensitive teeth.
“This is the world’s first flexible apatite sheet, which we hope to use to protect teeth or repair damaged enamel,” said Shigeki Hontsu, professor at Kinki University’s Faculty of Biology-Oriented Science and Technology in western Japan.
“Dentists used to think an all-apatite sheet was just a dream, but we are aiming to create artificial enamel,” the outermost layer of a tooth, he said earlier this month.
Researchers can create film just 0.004 millimetres (0.00016 inches) thick by firing lasers at compressed blocks of hydroxyapatite in a vacuum to make individual particles pop out.
These particles fall onto a block of salt which is heated to crystallise them, before the salt stand is dissolved in water.
The film is scooped up onto filter paper and dried, after which it is robust enough to be picked up by a pair of tweezers.
“The moment you put it on a tooth surface, it becomes invisible. You can barely see it if you examine it under a light,” Hontsu told AFP by telephone.
The sheet has a number of minute holes that allow liquid and air to escape from underneath to prevent their forming bubbles when it is applied onto a tooth.
One problem is that it takes almost one day for the film to adhere firmly to the tooth’s surface, said Hontsu.
The film is currently transparent but it is possible to make it white for use in cosmetic dentistry.
Researchers are experimenting on disused human teeth at the moment but the team will soon move to tests with animals, Hontsu said, adding he was also trying it on his own teeth.
Five years or more would be needed before the film could be used in practical dental treatment such as covering exposed dentin — the sensitive layer underneath enamel — but it could be used cosmetically within three years, Hontsu said.
The technology, which has been jointly developed with Kazushi Yoshikawa, associate professor at Osaka Dental University, is patented in Japan and South Korea and applications are under way in the United States, Europe and China.
From ‘Keep 32′ [teeth]
No more fillings as dentists reveal new tooth decay treatment ↓
New info → HERE ← Jan. 2015
↓ SEE NEW INFO BELOW ↓
❝ The sign of fluoride over-dose [dental fluorosis] is now pandemic in fluoridated areas. The teeth are a mirror of what also occurs in our bones. ❞
In contrast to skeletal bone and dentine, which accumulate fluoride throughout life and in proportion to the absorbed dose of fluoride, enamel of teeth reflects the biologically available fluoride at the time of tooth formation. Enamel maturation of deciduous teeth is completed between the age of 2 to 12 months. In permanent teeth enamel maturation is completed at the age of 7-8 years, except in the third molars, in which it continues until the age of 12-16 years. Post-eruptive fluoride uptake of enamel is expressed only in the outer layer and depends on fluoride in saliva, food, dental plaque and dental products (WHO, 1994). In areas with low fluoride concentrations in drinking water (≤0.1 mg/L) the fluoride concentration at 2 micrometer depth of enamel averages 1700 mg/kg, with fluoride concentrations in water of 1 mg/L it is 2200-3200 mg/kg. When water contains 5-7 mg/L of fluoride the concentration in enamel has been 4800 mg/kg. Such concentrations usually are accompanied by dental fluorosis (NRC, 1993).
About.com Guide Updated 15 December 2003
About.com Health’s Disease and Condition content is reviewed by our Medical Review Board
Wednesday January 24, 2001 ––Cavity rates declined in several cities that stopped water fluoridation, new studies report, contradicting American Dental Association (ADA) predictions, according to Fluoride Action Network.
Fluoride added to over 62% of US water supplies is supposed to reduce tooth decay, but these six studies from dental journals show it hasn’t and, in fact, may have increased the likelihood of rotten choppers. “No increase in caries (cavities) was found in Kuopio (Finland) 3 years after the discontinuation of water fluoridation,” according to Caries Research
In fact, when Kuopio was compared to a similar never fluoridated Finnish town, cavity rates in both towns either remained the same or decreased six years after fluoridation was stopped in Kuopio.
Seven years after fluoridation ended in La Salud, Cuba, cavities remained low in 6 to 9 year olds, decreased in 10 to 11 year-olds, significantly decreased in 12 to 13 year olds, while caries-free children increased dramatically, reports Caries Research
East German scientists report, “following the cessation of water fluoridation in the cities Chemnitz (formerly Karl-Marx-Stadt) and Plauen, a significant fall in caries prevalence was observed,” according to Community Dentistry and Oral Epidemiology
Additional surveys in the formerly-fluoridated towns of Spremberg and Zittau found. “Caries levels for the 12 -year-olds of both towns significantly decreased… following the cessation of water fluoridation.”
Not only did decay rates remain stable during an 11-month fluoridation break in Durham, NC, between September, 1990, and August, 1991 but dental fluorosis declined in children born during that period, according to the Journal of Dental Research
In British Columbia, Canada, “the prevalence of caries decreased over time in the fluoridation-ended community while remaining unchanged in the fluoridated community,” reported in Community Dentistry and Oral Epidemiology.
In 1973, the Dutch town of Tiel stopped fluoridation. Researchers counted drilled, missing, and filled tooth surfaces (DMFS) of Tiel’s 15-year olds, then collected identical data from never-fluoridated Culemborg. DMFS initially increased in Tiel then dipped to 11% of baseline from 1968/69 to 1987/8 8 while never-fluoridated Culemborg’s 15-year-olds had 72% less cavities over the same period, reports Caries Research.
A recently released government report out of Canada (7) shows similar negative results and offers a reason.
Fluoridation was launched in the 1940′s when dentists believed fluoride’s beneficial effects were achieved internally, through the bloodstream then absorbed inside the teeth. The Canadians report that “this effect is likely to be minor…The evidence for a post-eruptive (topical) effect… is much stronger.”
Therefore, swallowing fluoride doesn’t reduce tooth decay but does cause dental fluorosis — white spotted, yellow or brown stained and sometimes pitted teeth.
According to the ADA’s website “Dental decay can be expected to increase if water fluoridation in a community is discontinued for one year or more, even if topical products such as fluoride toothpaste and fluoride rinses are widely used.”
“Well, science proves the ADA is wrong about the claimed benefits of water fluoridation and they are wrong about the safety of water fluoridation, too” says lawyer Paul Beeber, President, New York State Coalition Opposed to Fluoridation. “Studies have linked fluoridation chemicals to increased blood lead levels, neurological defects, brittle bones, thyroid cancer and more,” says Beeber. “It’s time for the media to challenge the $cience that supports water fluoridation and present the truth to the public. We’ve made it easy for the media to access the ‘Medline’ [PubMed] abstracts via the links below. No more excuses.”
Other US government studies and statistics support the findings of these six studies and the Canadian government report. For example, children in fluoridated-since-1945 Newburgh, New York, have no less tooth decay but significantly more dental fluorosis than children from never-fluoridated Kingston, New York, according to Community Dentistry and Oral Epidemiology June 1999:
(1) “Caries trends 1992-1998 <tel:1992-1998> in two low-fluoride Finnish towns formerly with and without fluoridation,” Caries Research, Nov-Dec 2000 – Reference <http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=110930%2019&dopt=Abstract>
(2) “Caries prevalence after cessation of water fluoridation in La Salud, Cuba,” Caries Research Jan-Feb. 2000 — Reference <http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=10601780&dopt=Abstract>
(3) “Decline of caries prevalence after the cessation of water fluoridation in the former East Germany, & quote; Community Dentistry and Oral Epidemiology, October 2000 — Reference <http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11014515&dopt=Abstract>
(4) “The effects of a break in water fluoridation on the development of dental caries and fluorosis,” Journal of Dental Research, Feb. 2000 — Reference <http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=10728978&dopt=Abstract>
(5) “Patterns of dental caries following the cessation of water fluoridation,” Community Dentistry and Oral Epidemiology, February 2001 — Reference <http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11153562&dopt=Abstract>
(6) “Caries experience of 15-year-old children in The Netherlands after discontinuation of water fluoridation,” Caries Research, 1993 — Reference <http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=8519058&dopt=Abstract>
DENTAL FLUOROSIS – THE PSYCHOSOCIAL ISSUE ↓
Julia Roberts – No toothpaste Smile
HAPPY ‘PRIMITIVE’ SMILES
DENTAL DISEASES – EXTRACTS FROM VARIOUS RESEARCH PAPERS:
Stunted children at 6 months of age were more likely to have non-emerged permanent molars at 6 years of age; Children from work social class families, those with height deficit for age at 12 months, child who did not attend day care center, brushing teeth less than once a day, and children with sweet consumption of at least once a day were risk factors for high level of primary dental caries at age six.
Non-nutritive sucking habits and digital sucking were the main risk factors for anterior open bite at age six.
Short breastfeeding and regular use of a pacifier were risk factors for posterior cross bite. Interaction between short duration of breastfeeding and the use of pacifier was also identified;
Dental caries at aged twelve was strongly and positively associated with dental caries at aged six.
The proportion of individuals with severe malocclusions at twelve years-old increased according to the number of malocclusions presented at six years-old;
Children who did not live with their biological father at birth, and children with higher dmf-t, reported a higher lifetime prevalence of toothache at age six years.
Population attributable risk for dental caries at age 12 were 3.1 % for deficit in height for age at twelve months and 64.9% for primary dental caries at age six.
The level of accuracy in predicting dental caries at age twelve by using life course socioeconomic, behavioral and clinical data was modest.
Dental caries are one of the most common chronic diseases world- wide. 90% of people have had dental problems or toothache caused by caries, and in low-to-middle income countries most caries remains untreated. Severe periodontitis affects 5–15% of most populations. Oral cancer is the eighth most common cancer worldwide and the most common in men in South East Asia. And 40–50% of people who are HIV positive have oral fungal, bacterial, or viral infections” (Lancet January 3, 2009)