Three studies of highly fluoride-exposed populations in China
and India have found that high fluoride exposure
is associated with reduced male fertility.



By Dr. Mercola original ↓*

Fluoride May Be Your Next Contraceptive?

29 October 2013

In the US, where about two-thirds of drinking water supplies are fluoridated,
men may be putting their fertility at risk with every sip of water from the tap.

Research has long linked fluoride in drinking water with sperm damage and other threats to reproductive health, although many Americans remain completely in the dark about the association.

Yet, most of the studies on fluoride use sodium fluoride, which while extremely toxic is pharmaceutical-grade fluoride — NOT the kind added to water supplies. So researchers set out to determine the toxicity of the fluoride compounds that are actually in most water supplies. Here’s what they found…

Fluoride in Drinking Water May Be Toxic to Sperm

First a bit of background… sodium fluoride was the first of the fluoride waste materials to be used for fluoridation, but now is rarely used. It’s the most well known, as this is ‘pharmaceutical grade’ compound used in toxicology studies and other research into the potential health dangers of fluoride.

The other two types of fluoride, sodium silicofluoride and hydrofluorosilicic acid, are the compounds actually used for water fluoridation. Sodium silicofluoride and hydrofluorosilicic acid are the waste products from the fertilizer industry and are actually classified as hazardous wastes.

The new study compared the toxicity of three fluoride compounds – sodium fluoride and two silicofluorides used for water fluoridation – to determine their effect on the growth, feeding and reproduction of roundworms. They found:1

“Exposure to these compounds produced classic concentration-response toxicity profiles… This suggests that silicofluorides have similar toxicity to NaF [sodium fluoride].”

Since sodium fluoride appears to be equally toxic as the fluoride chemicals added to drinking water, it raises serious concern about another recent study, which found sodium fluoride may damage sperm chemotaxis, the process by which sperm are attracted toward an egg, which plays a critical role in allowing fertilization to occur.2 Though this study involved mice, if the findings apply to humans it could put men at risk of becoming infertile…

Fluoride May Be Dangerous to Male Fertility

Past research, too, has shown that fluoride has potentially disastrous effects on the male reproductive system.

The Fluoride Action Network (FAN) reported:3

  • Exposing ram semen to 0.38 parts per million (20 umol/L) of fluoride for five hours caused a “statistically significant decrease in the motility of spermatozoa” along with other changes that would “undoubtedly affect the physiological function of the sperm.”

  • Infusing testes with modest amounts of fluoride (4.75 parts per million) inhibited the synthesis of testosterone

  • Sixty animal studies have found that fluoride adversely impacts the male reproductive system, including:


Decreases in testosterone levels

Reduced sperm motility

Altered sperm morphology

Reduced sperm quantity

Increased oxidative stress

Reduced capacity to breed


It’s not only animal studies that have shown an emerging problem,
however. As FAN explained:4

“Consistent with the in vitro and animal research, studies of human populations have reported associations between fluoride exposure and damage to the male reproductive system. Most notably, a scientist at the Food & Drug Administration [FDA] reported in 1994 that populations in the United States with more than 3 ppm fluoride in their water had lower  ‘total fertility rates’ than populations with lower fluoride levels … 

While 3 ppm is a higher concentration than used in water fluoridation programs (0.7 to 1.2 ppm), it is still considered a ‘safe’ level by the EPA. To date, no U.S. health agency has attempted to replicate… [these] findings.

However, three studies of highly fluoride-exposed populations in China and India have found that high fluoride exposure is associated with reduced male fertility. In addition, five studies from China, India, Mexico, and Russia have found that high-fluoride exposure is associated with reduced male testosterone levels.”

Fluoride Is an Undeniable Poison to Your Entire Body

Men of reproductive age may now be thinking twice about the fluoride in their drinking water, but this is not the only population that stands to be harmed. 

Study after study has confirmed that fluoride is a dangerous, toxic poison that bio-accumulates in your body while being ineffective at preventing dental decay. For instance, there are 25 studies showing that fluoride reduces IQ in children, and approximately 100 animal studies have also linked fluoride to brain damage.5

One particularly striking animal study6 showed that fluoride ingestion had a profound influence on the animals’ brains and altered behavior. Pregnant rats given fluoride produced hyperactive offspring. And animals given fluoride after birth became apathetic, lethargic “couch potatoes.” Other research has linked fluoride toxicity with the wide-ranging problems listed below.


Increases lead absorption

Disrupts collagen synthesis

Increases manganese absorption, which is also linked to lower IQ in children

Crippling skeletal fluorosis and bone fractures

Genetic damage and cell death

Increased tumor and cancer growth

Disrupts immune system

Inhibits antibody production

Brain damage, and lowered IQ



Severe eye problems, includingblindness

Impaired thyroidfunction

Bone cancer (osteosarcoma)

Inactivates 62 enzymes

Muscle disorders

What’s Better Than Fluoride for Preventing Cavities?

Well, practically anything, since ingesting fluoride in drinking water does NOT decrease rates of tooth decay. In all seriousness, anyone who has ever read Dr. Weston Price’s book Nutrition and Physical Degeneration will soon recognize that diet is the most important factor in dental health.

 Dr. Price wrote this book in 1920 and it is full of pictures of cultures with excellent dental health that rapidly deteriorated once processed Western food was introduced. When Dr. Price studied these native diets, he noticed some similarities in the foods that kept them so healthy. Among them:

  • The foods were natural, unprocessed, and organic (and contained no sugar except for the occasional bit of honey or maple syrup).

  • The people ate foods that grew in their native environment. In other words, they ate locally grown, seasonal foods.

  • Many of the cultures ate unpasteurized dairy products, and all of them ate fermented foods.

  • The people ate a significant portion of their food raw.

  • All of the cultures ate animal products, including animal fat and, often, full-fat butter and organ meats.

I believe that if you have an optimal diet, without processed foods and sugars from infancy, you can be virtually assured of cavity-free teeth. All of the brushing and flossing in the world will not give you the healthy teeth that following the eating patterns described above will, so if you value your pearly whites,
get started eating a healthier diet today.

Join the Fight to Get Fluoride Out of Drinking Water

There’s no doubt about it: Fluoride should not be ingested. Even scientists from the EPA’s National Health and Environmental Effects Research Laboratory have classified fluoride as a “chemical having substantial evidence of developmental neurotoxicity.” Furthermore, according to the Centers for Disease Control and Prevention (CDC), 41 percent of American adolescents now have dental fluorosis—unattractive discoloration and mottling of the teeth that indicate overexposure to fluoride.

Clearly, children and adults alike are being overexposed, and their health, reproduction and development put in jeopardy. Why? At least when it comes to topical application, you have a choice. You can easily buy fluoride-free toothpaste and mouthwash. But you’re stuck with whatever your community puts in the water, and it’s very difficult to filter fluoride out of your water once it’s added. Many do not have the resources or the knowledge to do so.

The only real solution is to stop the archaic practice of water fluoridation in the first place. Fortunately, the Fluoride Action Network has a game plan to END water fluoridation, both in the United States and Canada. Clean pure water is a prerequisite to optimal health. Industrial chemicals, drugs and other toxic additives really have no place in our water supplies. So, please, support the anti-fluoride movement by making a donation to the Fluoride Action Network today.


* We keep our own copies off line for safe keeping…







Kevin Mugur Galalae


Prenatal Fluoride Exposure and Cognitive Outcomes -Mexico




 Ovarian Cancer & Fluoride





Comments are closed.

You may be exposed by breathing, eating, or drinking
the substance, or by skin contact.

HRS logo f

 FLUORIDES – Toxic Profile – U.S. Dept. of Health Services

A Toxicological Profile for Hydrogen Fluoride, and Fluorine,
Draft for Public Comment was released in September 2001.
This edition supersedes any previously released draft or final profile.

“However, the fluoride accumulates primarily
in the bones or shell rather than in edible meat.” ↓


                                                                Extract from:

30 SEPTEMBER  2003 


This public health statement tells you about fluorides, hydrogen fluoride,

and fluorine and the effects of exposure presented in the toxicological

profile. These profiles were specifically prepared by ATSDR for

hazardous substances which are most commonly found at

facilities on the CERCLA National Priorities List

(Superfund sites) and are intended to

describe the effects of exposure

from chemicals at these sites.

The Environmental Protection Agency (EPA) identifies
the most serious hazardous waste sites in the nation.

These sites make up the National Priorities List (NPL) and are the sites targeted for long-term federal clean-up activities. Fluorides, hydrogen fluoride, and fluorine have been found in at least 188 of the 1,636 current or former NPL sites. However, the total number of NPL sites evaluated for these substances is not known. As more sites are evaluated, the sites at which fluorides, hydrogen fluoride, and fluorine is found may increase. This information is important because exposure to these substances may harm you and because these sites may be sources of exposure.

When a substance is released from a large area, such as an industrial plant, or from a container, such as a drum or bottle, it enters the environment. This release does not always lead to exposure. You are exposed to a substance only when you come in contact with it. You may be exposed by breathing, eating, or drinking the substance, or by skin contact.

If you are exposed to fluorides, hydrogen fluoride, and fluorine, many factors determine whether you’ll be harmed. These factors include the dose (how much), the duration (how long), and how you come in contact with it/them. [♦]

You must also consider the other chemicals you’re exposed to and your age, sex, diet, family traits,

lifestyle, and state of health.

♦ And nutritional status. 

Original document ↓ ↓ 

Full Original Document

double blue line

“The medical profession is being bought by the pharmaceutical industry,
not only in terms of practice of medicine, but also in terms of teaching
and research. The academic institutions of this country are allowing
themselves to be the paid agents of the pharmaceutical industry.
I think it’s disgraceful.” – 
Arnold Seymour Relman,

former editor-in-chief of the New England Medical Journal,
and professor of medicine at Harvard University.





Overall, Dr. John Polya made a major contribution to
chemical research and chemical education in
Australia. He published over 120 scholarly
works and supervised numerous
research students.

~ ARE WE SAFE? – John Polya ~

A Laymans Guide-Polya intro

Large file


  ‘Are We Safe’ the book   

↑   Full Text   ↑



Pólya, János Béla (1914–1992)

by Attila Urmenyhazi

Dr. János Béla Pólya (John Bela Polya), (1914-1992), FRACI, scientist, academic, and internationally recognised authority in organic chemistry, was born in 1914 in Budapest, Hungary (Austro-Hungarian Empire) into a secular Jewish family. He was the son of Dr. Jenõ (Eugene) Pólya, a university professor and famous surgeon (Polya Gastrectomy operation), and Livia Polya from a gifted, orthodox Jewish family. The Polya family was assimilatory and secular, changing their name to Pólya from Pollak in about 1880 and converting to Catholicism.


John Polya studied at a Catholic Piarist school in Budapest which also had Jewish, Presbyterian and Muslim students. He then studied chemistry at the renowned Eidgenössische Technischen Hochschule in Zurich, Switzerland, in the 1930s. In 1936 he completed his tertiary studies at the Swiss Federal Institute of Technology in Zurich. From 1937 to 1939 he was a Rockefeller Scholar in Organic Chemistry at the University of Manchester and the Imperial College, London; subsequently working as a Rockefeller Scholar on vitamin A at the two universities. While in England he became acutely aware of the Nazi threat to Jews. His sister, Susie, and brother, Michael, left Hungary before World War II for America and Australia, respectively. His father visited America and was urged by medical colleagues to remain in the USA. Confident in his eminent professional position, however, his father returned to Budapest and was killed by the Nazis in December 1944 just prior to liberation. John Polya’s mother and younger sister survived the Holocaust in Hungary and emigrated to Australia after the war. The 1930s to early ’40s saw the arrival, in Australia, of a highly educated Hungarian elite, mainly Jewry fleeing repression by a Hungarian government gradually compliant with Nazi Germany’s demands.


Polya arrived in Sydney from the UK in 1939. He recalled preferring Australia over Chile as a destination for a refugee because at that time he spoke English but no Spanish. He initially worked as a chemist for Sir Edward Hallstrom (Hallstrom Pty. Ltd, refrigerators) (1939-1940). He then moved to Melbourne and worked as an industrial chemist for the well-known chemist, entrepreneur, wine expert and humanist Oscar Mendelsohn (1940-1945). His attempt to enlist failed because his “reserve occupation” was deemed more necessary to the nation then his military service. He subsequently worked for the Munitions Supply Laboratories at Maribyrnong, Melbourne (1945-1946) (arising from his discovery of the chemical basis of premature corrosion in RAAF planes). His stellar academic career took off from 1946 when he was appointed Lecturer in Organic Chemistry at the University of Tasmania. At the time, he was staunchly opposed to the fluoridation of Australia’s drinking water and published Are We Safe. A Layman’s Guide to Public Health (Cheshire, Melbourne 1964). In 1952 he was awarded a Doctor of Science from the University of Tasmania, and in 1955 was appointed Associate Professor of Chemistry at the university.

Polya had married Robin Barker in 1940. Their daughter (Michal) was born in 1941, Gideon in 1944, Rosemary (1948), John (1955) and David (1960). All of his children obtained their first degrees from the University of Tasmania and all pursued studies with a scientific leaning. Michal (BSc, DipEd, mathematics teacher), Gideon (BSc, PhD, biochemistry academic), Rosemary (BA, BSc, DipEd, ALA, teacher, librarian), John (BMus, mathematics and computing teacher) and David (BSc, PhD, geology academic).

Polya believed in the responsibility of scientists to speak out in the public interest and in 1977-78 became the president of the Royal Australian Chemical Institute, Tasmania branch. In 1978-79, he was chairman of the Organic Chemistry Division of the Royal Australian Chemical Institute. Highly regarded, respected and fondly remembered by his former chemistry and pharmacy students, he retired from the University of Tasmania in 1979.

As an academic and scientist Polya was known to be a very passionate and opinionated person who spoke bluntly, freely, and without fear of consequences. He was an extraordinarily gifted, multilingual polemist with deep interests in philology, science, philosophy, theology, literature and music.  He was also a great cook and had a great love for Jewish culture as reflected in gastronomy, music, humour and literature.

Overall, Dr. John Polya made a major contribution to chemical research and chemical education in Australia. He published over 120 scholarly works and supervised numerous research students. With Dr Kitty Got (a Jewish Hungarian refugee who arrived in Australia in 1956) he introduced the teaching of biochemistry to the Chemistry Department and notably encouraged women scientists. He believed that a vital truth from the Jewish experience was the importance of “being different”, which he perceived as contributing to a better and culturally richer humanity. He died on 15 December 1992 in Melbourne

brown devider

Dr. János Béla Pólya ( John Bela Polya ), (1914-1992), FRACI,

A scientist, academic, and internationally recognised authority in organic chemistry.

Associate Professor John Polya, then in the Department of Chemistry

at the University of Tasmania, was very outspoken about fluoridation.

 → He claimed in 1973 that his staff and equipment had been taken

away from him because of this.  

Polya believed in the responsibility of scientists

to speak out in the public interest…

brown devider

Original →  HERE

Dr John (Janos Bela) Polya ( see photo above), then 54, was a conservative intellectual with a European background and education, starting in his native Hungary. He was an internationally recognised authority in organic chemistry. 

“As an academic and scientist Polya was known to be a very passionate and opinionated person who spoke bluntly, freely, and without fear of consequences. He was an extraordinarily gifted, multilingual polemist with deep interests in philology, science, philosophy, theology, literature and music,” the Australian Dictionary of Biography says of him.

Polya was a staunch advocate of academic freedom and a leading supporter of justice for Professor Sydney Sparkes Orr, dismissed by the University of Tasmania after his successful advocacy of a Royal Commission into that institution. The book “Dreyfus in Australia” by Polya and Robert Solomon provides an insider account of the long- running Orr case that had at its heart the key issue of academic freedom7.

As a refugee from fascist Hungary, Polya embraced and very actively exercised the freedom of speech he found in Australia (his eldest son, former Associate Professor of Biochemistry Gideon Polya, has vigorously followed the same ethical path).

Exercise of such freedom of speech by scientists and scholars is crucially important for informed public discussion in the public interest, as exampled by John Polya’s book “Are we safe? A Layman’s Guide to Controversy in Public Health”. Dr, later Prof, Polya was stridently opposed to fluoridating drinking water. He died in 1992.

5 hbp:// 6 hbp://

7 For other accounts see “Orr” by W.H.C. Eddy, “Gross moral turpitude: the Orr case reconsidered” by Cassandra Pybus, and “”Not merely malice”: the University of Tasmania versus Professor Orr” by Clyde Manwell and C.M. Ann Baker in “Intellectual Suppression” edited by Brian MarPn, C.M. Ann Baker, Clyde Manwell and Cedric Pugh – information and photo personally supplied by JB Polya’s son and daughter, Gideon and Rosemary Polya.

Civil Liberties in Australia: Ch 7 – Tasmania





“Because of the affinity of strontium and fluoride to form
highly insoluble Sr90F2, the consumption of water
that has been artificially fluoridated may
increase the danger of  Strontium 90
to man and animals.”




Iodine -131  ↔ →  Strontium 90.

In 1958 Barry Commoner [] and others showed that the teeth of every baby in the US had some level of strontium 90 accumulation as a direct result of fall-out from nuclear testing in the 1940s and 1950s.

An article published in a 1958 Dental Digest by Dr. James G Kerwin, Director of the Central Division of the Passaic Department of Health in New Jersey, sheds more light on the issue of strontium and fluoride interactions:

“Because of the affinity of strontium and fluoride to form highly insoluble Sr90F2, the consumption of water that has been artificially fluoridated may increase the danger of Strontium 90 to man and animals.”

“For obvious reasons it is necessary for the body to be able to get rid of any radioactive strontium as quickly as possible. Under normal circumstances the body does this, but only at a slow rate. If, however, fluoride has been consumed, for example, from artificial fluoridated drinking water, then Strontium-90 and the fluoride may precipitate as the highly insoluble Sr90F2 within the body. This means that the rate at which Strontium-90 is excreted or thrown off will be even slower than ordinarily occurs.” [♦♦]

“It is known that fluorides are powerful inhibitors of enzyme action, and that many types of enzymes are affected, including some that are essential for cellular oxidation…The wide variety of enzymes known to be poisoned by fluoride accounts for the different manifestations of fluorosis; that is, the different symptoms associated with fluoride poisoning. “

“Life processes are so delicately balanced that any change, any departure from the normally established order at the cellular level, is likely to be for the worse. Thus a combination of radioactive strontium and fluoride, forming Sr90F2, could do great harm. If the entire population of the United States received small doses of additional radiation from Strontium-90 which was combined with fluoride in the body, and thus was prevented from being excreted, it is likely that several thousands among the generation of ten million children may be definitely handicapped because of gene mutation due to internal radiation.”

“There is still another aspect to consider; that is, a population that is exposed generation after generation to an increasing amount of Strontium-90 and fluoride may quite likely exhibit a higher death rate coupled with a lower birth rate, both due to harmful radiations and the resulting mutations.”


  Barry Commoner was an American biologist, college professor,

and politician. He was a leading ecologist and among the

founders of the modern environmental movement.

One of Barry Commoner’s lasting legacies is his

four laws of ecology, as written in

‘The Closing Circle’ in 1971.***



 ❝ …Radioactive elements are structurally similar to their non-radioactive counterparts, differing in the number of neutrons the atoms contain. This is why nutrition is important in preventing or blocking damage from exposure to radioactive elements.

If you do not obtain sufficient amounts of calcium, potassium, and other minerals in your diet, your body may absorb radioactive elements that are similar in structure to these nutrients.

For example, if you do not obtain enough calcium, your body will absorb radioactive strontium 90 or other elements that are similar in structure to calcium, if they are available. Similarly, if you obtain sufficient potassium from your diet, your body will be less likely to retain any radioactive cesium 137 it encounters, as this element is similar to potassium.

If the cells are able to obtain all nutrients they need from your diet,

they will be less likely to absorb radioactive substitutes,

which are then more likely to be discarded from the body… 

This may be the best book on ecology ever written.
It is certainly the most sober, rigorous, well organized
statement of what our environmental problems are, how we
got them, and what we should do about them.


Brushing with Fluoride Can Give You a Radiant Smile:


  The Secret History of the Atomic Bomb  


Dangerous Synergism – Fluorides & SR-90


Professor Karl Grossman


The Hoax of Eco-Friendly Nuclear Energy

Nuclear advocates in government and the nuclear industry are engaged in a massive,
heavily financed drive to revive atomic power in the United States—
with most of the mainstream media either not questioning
or actually assisting in this promotion.


Cesium-137 – Eliminator – Free


Australian Atomic Confessions Documentary





Bill Gates and his “plans” for plutonium




See also →  Clear Intent – introduction ←more on nuclear weapons




Air pollution caused more than 1.2 million premature deaths
in China in just one year

Pollution - China

Fluoride Vol. 37 No. 2 125–132 2004 Research Report 125


Daishe Wu,a,b,c Baoshan Zheng,a Xiuyi Tang,d Shehong Li, a Binbin Wang,

a Mingshi Wang,a,b Guiyang, China  



31 JULY 2013

Written by Aviva Shen

     China’s air pollution levels have reached dire levels, even breaking the upper limits of the Air Quality Index earlier this year. In a sign the government is serious about tackling this crisis. The China Daily announced Thursday that China will spend $275 billion over the next five years to reduce emissions and launch anti-pollution programs.

The funds, which exceed the total economic output of Hong Kong last year, will target emissions in the densely populated area surrounding Beijing, where residents have suffered through off-the-charts pollution and all its accompanying illnesses. Air pollution caused more than 1.2 million premature deaths in China in just one year, while some Beijing schools are building air-purified domes over playgrounds so children can play “outside” safely. Anti-pollution protests have grown more and more prevalent all over the country.

These unsustainable conditions are directly linked to China’s rapid industrialization. Most of Beijing’s pollution stems from factories and power plants outside the city. China’s coal production has tripled in the past ten years as the nation’s energy consumption has exploded. The Chinese government, up til this year, has aggressively encouraged economic growth at the expense of the environment and public health.

But now that the environmental repercussions cannot be ignored, the government has done a hard about-face. New promised anti-pollution measures include speedy installation of pollution control equipment on coal-fuelled refineries, restrictions on high energy consumption industries like steel, cement and glass, and use legal action to force industries to upgrade their emissions standards.

Before Thursday’s announcement, China had already pledged $16 billion to specifically help Beijing build and update sewage and garbage treatment, plant new forests and curb illegal construction.

The nation also invested twice as much as the U.S. in clean energy projects last year.

This post was originally published at ClimateProgress.

Read more:

See also: 


 106 Fluoride Vol. 36 No. 2 106-112 2003 Research Report

For Correspondence: Prof Wuyi Wang, Institute of

Geographical Sciences and Natural Resources

Research, Chinese Academy of Sciences,

Beijing 100101, China. E-mail:




Yonghua Li, Wuyi Wang, a Linsheng Yang, Hairong Li

Beijing, China

SUMMARY:  Data on coal-burning endemic fluorosis throughout China and on the exposure-response relationship between concentrations of fluoride determined in coal samples and the prevalence of dental fluorosis reported from 17 representative surveillance stations in Southwest China were used to estimate the safety threshold for coal fluoride. Coal-burning endemic fluorosis occurs mainly in the mountainous areas of this part of China, where the prevalence of the disease is closely linked to geochemical parameters of the local environment. In these regions the incidence of dental fluorosis has a significant positive correlation with the concentration of fluoride in coal. The safety threshold of coal fluoride is estimated to be 190 mg/kg by the criterion of 0% incidence of dental fluorosis.

Keywords: China; Coal fluoride; Endemic fluorosis; Safety threshold.


     Fluorine (F), the most electronegative and reactive of the halogens, is a common chemical element in the earth’s crust in combined form. F concentrations in rocks and soils are well documented, but data on the F concentration in coal are relatively limited.

1-4 Swaine reported the total F concentration in coal ranges from 20 to 500 mg/kg.

5 Statistical data indicate that the mean concentration of F in coal worldwide is 80 mg/kg, but in China it is 200 mg/kg.

6 In the mountainous areas of Southwest China, it is even higher— up to 3106 mg/kg in local coal.

7 Fluoride in coal can be released into the ambient environment as atmospheric F, waterborne F, and residue F during mining, handling, and combustion.

6-8 In Southwest China, F

See also↓

End line

The Chinese have some coal for power stations. 

Queensland’s coking coal, also known as metallurgical coal
is needed by them to make steel.
Not very easy to do with steaming coal or solar power!

Coking Coal 
Coking coal, also known as metallurgical coal, has low sulphur and phosphorus content and can withstand high heat.
Coking coal is fed into ovens and subjected to oxygen-free pyrolysis, a process that heats the coal to approximately 1,100 degrees Celsius, melting it and driving off any volatile compounds and impurities to leave pure carbon. The hot, purified, liquefied carbon solidifies into lumps called “coke” that can be fed into a blast furnace along with iron ore and limestone to produce steel.

Steam Coal
Steam coal also known as thermal coal, is suitable for electric power production.
Steam coal is ground into a fine powder that burns quickly at high heats and is used in power plants to heat water in boilers that run steam turbines. These need to have Scrubbers on the smoke stacks.









In the township Xinyuan, we examined 250 schoolchildren, aged 7-14 years.
The goiter prevalence was 91% and dental fluorosis 20.80%.
The average level of iodine in drinking water was 5.21 mg/l,
and that of fluoride 0.88 mg/l.




Lin Fa-Fu, Aihaiti, Zhao Hong-Xin,

Lin Jin, Jiang Ji-Yong, Maimaiti, and Aiken.


… Xinjiang Institute for Endemic Disease Control and Research; Office of Leading Group for Endemic Disease Control of Hetian Prefectural Committee of the Communist Party of China; and County Health and Epidemic Prevention Station, Yutian, Xinjiang. Cretinism in iodine-deficiency areas is well known, yet the milder forms of somatic and psychomotor maldevelopment and thyroid dysfunction caused by iodine deficiency may be more difficult to detect. DeQuervain, in 1936, called this milder form “semi-cretinism,” while in 1980 Laggasse used the term “cretinoidism.” It was formally named “subclinical endemic cretinism” at a symposium on subclinical cretinism held in Xinzhou, Shanxi province in 1985.

Currently, attention is being focused on these disorders in China and abroad.

The Hetian prefecture in Xinjiang has reportedly been one of the Asian

areas most severely affected by iodine deficiency disorders (IDD).

During the period 1987-1989, we made a systematic survey of

subclinical endemic cretinism in this district under

a UNICEF aid Project.


General conditions and selection of affected areas – The entire region of Xinjiang in central Eurasia is affected by iodine deficiency. The study area, located between the southern border of Tarim basin and the northern slope of Kunlun Mountains, is arid with sandy soil and an annual precipitation less than 50 mm. The cultivated alluvial plain extends from south to north with a steepening gradient. The geographical distributions of endemic goiter and endemic fluorosis are characterized by marked vertical zones. The inhabitants are of lower socioeconomic status, with an annual mean income of about 200 yuan (RMB) per person.

Area with high fluoride and low iodine levels (Area A) – In the township Xinyuan in the lower reaches of Kliya river in the county of Yutian, north of the highway, we examined 250 schoolchildren, aged 7-14 years. The goiter prevalence was 91% and dental fluorosis 20.80%. The average level of iodine in drinking water was 5.21 mg/l, and that of fluoride 0.88 mg/l.

Area with low iodine level (area B) – In the townships of Langan and Jiayi in the alluvial plain before the mountains and to the south of the highway, we examined 256 schoolchildren, aged 7-14 years. The goiter prevalence was 82% and dental fluorosis of 16.00%. The average water iodine level was 0.96 mg/l and that of fluoride 0.34 mg/l. ...

Green strip



M&F Cretins f


Image of 3 Cretins copy

More info on iodine → HERE

Green strip

  Iodine and IQ f  


Green strip

Green strip

Newsweek 24 Feb.2015

  Water fluoridation in England linked to higher rates of under-active thyroid  

See also 

Iodine Awareness Week –
Antimicrobial Resistance Emergency






The radiative forcing of climate change associated with

emissions of SO2F2 depends critically on the
atmospheric lifetime of SO2F2.


The use of the fluoride-containing pesticide, sulfuryl fluoride as a food fumigant (trade name ProFume ®) as well as the fluoridation of drinking water, also contribute to the release of fluoride from anthropogenic sources. Based on available data, phosphate ore production and use, as well as aluminium manufacture are the major industrial sources of fluoride release into the environment.

Sulfuryl fluoride (SO2F2) is a radiatively active industrial chemical released into the atmosphere in significant (ktonne/year) quantities. The potential for SO2F2 to contribute to radiative forcing of climate change needs to be assessed.  Long path length FTIR/smog chamber techniques were used to investigate the kinetics of the gas-phase reactions of Cl atoms, OH radicals, and O3 with SO2F2, in 700 Torr total pressure of air or N2 at 296 ± 1 K. Upper limits of k(Cl + SO2F2) < 9 × 10−19, k(OH + SO2F2) < 1.7 × 10−14 and k(O3 + SO2F2) < 5.5 × 10−24 cm3 molecule−1 s−1 were determined. Reaction with Cl atoms, OH radicals, or O3 does not provide an efficient removal mechanism for SO2F2. The infrared spectrum of SO2F2 is reported and a radiative efficiency of 0.196 W m−2 ppbv−1 was calculated. Historic production data estimates are presented which provide an upper limit for expected atmospheric concentrations. The radiative forcing of climate change associated with emissions of SO2F2 depends critically on the atmospheric lifetime of SO2F2.

Further research is urgently3 + SO2F2) < 5.5 × 10−24 cm3 molecule−1 s−1 were determined. Reaction with Cl atoms, OH radicals, or O3 does not provide an efficient removal mechanism for SO2F2. The infrared spectrum of SO2F2 is reported and a radiative efficiency of 0.196 W m−2 ppbv−1 was calculated. Historic production data estimates are presented which provide an upper limit for expected atmospheric concentrations.

The radiative forcing of climate change associated with emissions of SO2F2 depends critically on the atmospheric lifetime of SO2F2.

Further research is urgently needed to define the magnitude of potential non-atmospheric sinks.

See also →
 Aust. Defence Bases Contaminated With PFOA (Fluoride)






Most animals manufacture Vitamin C, in their livers, 
which gives them some protection from fluoride damage,


On poor diets, low in Vit. C,  humans and guinea pigs
will suffer more damage than most other animals
even on low doses of fluoride. Guinea pigs
are therefor better experimental
animals than rats or mice.
[ If we must experiment on animals. ]

Lab Rat f

Full text   HERE ← 

Vitamin C Supplements for Guinea Pigs
Dr. Jeff Rhody, DVM

     For the biologic machinery of the body to work correctly, vitamin C is required.
Most animals can manufacture vitamin C in their bodies and do not require vitamin C in the diet.
Guinea pigs, humans, and other primates share a gene mutation that makes production of vitamin C impossible. For this reason, these animals require a dietary source of vitamin C.

Guinea pigs who do not receive enough vitamin C in their diet can suffer from vitamin C deficiency (commonly known as scurvy in humans). Affected guinea pigs may have a rough hair coat, lack of appetite, dental pain, delayed wound healing, lameness, and an inability to fend off infections. Guinea pigs with a slight vitamin C deficiency may show no visible signs of disease; however, their immune system may be compromised leading to decreased ability to fight off other illnesses….

Full text   HERE ← 

Vitamin C 

John A. Yiamouyiannis, Ph.D.

Vit.c strip



The Global Decline in Tooth Decay correlates
with reduced Airborne Lead (Pb) but water
Fluoridation prevents further progress.

Information Supplied by Dr. Geoffrey Norman Pain

Full text  → HERE


Fluoride increases the lead level in blood and in calcified tissues of rodents [Sawan 2010].

Australian median Lead levels in children have not declined since the final cessation of the use of Lead in motor fuel in 2002, indicating Lead from water, food and contaminated soil are still producing unacceptable poisoning of the general population [Taylor 2014].
[ See our paper on  → LEAD IN AVIATION FUEL ]

Most of the world recognizes that the only safe level of Lead is Zero, however Australian agencies including the NHMRC, Food Standards Australia and New Zealand (FSANZ), the Therapeutic Goods Administration (TGA) and the Australian Institute of Health and Welfare have been reluctant to coordinate systematic data collection including blood analysis or to lower allowable or action levels for Lead [Taylor 2014].
Their continued endorsement of deliberate Lead contamination through water Fluoridation prevents them from trying to achieve the Zero target.


Reduction of airborne Lead pollution following the phase-out of Tetraethyl Lead in motor fuel is the most plausible explanation for the very large observed global reduction in decay rates and the fact that there is no discernible difference in dental decay rates versus national Fluoridation status. 

The deliberate addition of Lead as a major contaminant of phosphate fertilizer industrial waste used in Fluoridation plus the exacerbation of Plumbosolvency caused by Fluoridation must cease if further reduction in blood Lead levels and tooth decay is to be accomplished.

It is time for the governments of the few remaining countries where Fluoridation persists to place an immediate ban on the practice and force industry to permanently immobilize their toxic waste.

The fact that WHO now rates Fluoride as more toxic than Lead should be a wake-up call to politicians everywhere.

↓  The two make a bad combination  ↓

Electrical Grounding – more lead in drinking water –

Vit.c strip

  Dr. Paul Connett talks about Impact of Fluoride on Brain  

Vit.c strip


Vit.c strip

Autism – Fluoride & Aluminium

Vit.c strip


Vit.c strip

 Old ‘adds’ from the past! 








The papers, books, and links below testify to the importance of zinc
for plants, animals and humans.  Illnesses, episodic violence,
hyperactivity, learning disabilities, depression, crime,
behavioural problems, reduced sexual reproduction
and alcohol intolerance, anorexia nervosa,
are associated with zinc deficiencies
in humans, and as it is a given that
fluorides are antagonists of zinc,
the hazards of fluorides in

food, air and water need
to be reduced urgently.

  Fluorides, Zinc and Anorexia Nevosa  


blue line

Physical chemical effects of zinc on in vitro enamel…


Item 6 is of interest to andrologists!


LEO SPIRA (#9) on the right, 3rd from the front. (See also the end of this post)

 26 January 1947 – The Belmont Hotel in Chicago.

PFPC NEWSLETTER #5 – “Spira, Fluoride and the Zinc Link”

Editors note:

We recommend this short read especially item 5 and 6.
Note Item on * Down Syndrome

2000 PFPC (Feel free to distribute)

20 May 2000








. . . -. . . .


Hi Everyone,

In the past we have written about fluoride/aluminum complexes and the ability of aluminum to greatly potentiate the toxicity of fluoride, especially as it relates to G protein activation and thyroid pathology. Fluorides directly imitate the biochemical activity of thyrotropin, the thyroid-stimulating-hormone (TSH).

In this newsletter we will turn our attention to zinc and its importance in thyroid function, and the role of fluoride in inducing zinc deficiency.

It is TSH that mediates iodine, zinc and selenium (Bellisola et al, 1998; Arreola et al, 1993).

Abnormal zinc metabolism occurs commonly in thyroid disease. Zinc levels in red blood cells have become an important marker of peripheral tissue response to thyroid hormones, indicating duration of pre-existing thyroid disease (Tiran 1993). In the clinical laboratory, the main interest of zinc determination in red blood cells now concerns thyroid pathology (Vitoux et al, 1999). A hyperthyroid person will reveal highly elevated serum zinc levels, but a reduced zinc content in whole blood, while the reverse changes are observed in the hypothyroid patient. There a reduced serum zinc content is seen, with highly increased zinc content in whole blood.

Zinc is thought to be of importance to thyroid hormone synthesis in the liver. Zinc may play a role in thyroid hormone metabolism in “low T3” patients and may in part contribute to conversion of T4  to T3 in humans. In some cases supplementation with zinc normalized the T3/rT3 ratio  (Licastro et al, 1992; 1993)

However, after treatment for thyroid dysfunction, normalization of zinc in red blood cells occurs, lagging about 2 months behind normalization of plasma T4 and T3 levels (Yoshida 1996; Varga, 1994). This is a clear sign that many cases of “zinc deficiency” are NOT caused by an actual nutritional lack of zinc, but by thyroid dysfunction. The symptoms of “zinc deficiency” are identical to the ones ascribed to hypothyroidism.

Many nutritional deficiencies are not due to an actual lack of said nutrients in the diet, but due to hypothyroidism. Thyroid hormones modulate nutrient and vitamin transport. When hypothyroid, the entire blood chemistry changes. Just as is the case with calcium (May 1950), magnesium (Shibutani et al, 1989), sex hormone-binding globulin (Foldes et al, 1990), somatomed in (Marek et al, 1981), etc., so zinc urine concentrations are elevated in hyperthyroidism (Nishi et al, 1980), but decreased in hypothyroid patients (Tsou et al, 1993; Buchinger et al, 1988).

We hope that this Newsletter is not too complicated. If you do have any questions, please don’t hesitate to contact us.

Best wishes to all,

Andreas Schuld
  Parents of Fluoride Poisoned Children (PFPC)  
Vancouver, BC, Canada
↑  Much info here on salt fluoridation ↑

blue line


In 1953 Leo Spira, MD, Ph.D. published his book “The Drama of Fluorine – Arch Enemy of Mankind.” Drawing upon his many articles which had been published in the Lancet, the British Dental Journal, and other recognized medical journals, the great British physician summarized his findings of over 30 years of pioneering research into various disease states he ultimately found to be related to fluoride intake.

One of the most frequent symptoms of early fluorosis observed by Spira were “mottled” and deformed nails which would often split or chip.

“The commonest feature, however, was the occurrence of raised longitudinal ridges on the surface of the finger and toe-nails…In addition, dull, opaque, chalky-white specks, patches or transverse bands, such as are familiar to everybody, appeared frequently on several nails…”

Further Spira wrote, “There was a frequent complaint that the hair was falling out prematurely.”

In addition Spira reported of several skin diseases and wrote, “Patients felt apprehensive and irritable and were subject to frequent attacks of depression and even melancholia with loss of energy and general lassitude”.

The symptoms Spira was describing were later referred to in other European literature as the “Spira-Syndrome” (Schoehl, 1985). It must be clear to any endocrinologist that the conditions Spira was describing in the early part of the past century are identical to those now commonly ascribed to zinc deficiency and/or hypothyroidism.

On a courtesy visit to the US, Spira had asked the PHS if they also had observed such symptoms, and was told that this was not quite the case. However, recent documents obtained show that the opposite was true. Even Dean himself had noticed such symptoms and wrote about them already in 1932:

“The hair of some of these mottled enamel cases is unusually coarse, almost like horse hair. Finger nails are apparently not normal. Two of the three local physicians state that there is apparently an unusually large account of skin disorders among those using the city water supply. Future surveys will attempt to obtain this additional dermatological data in order to determine whether it correlates with the mottled enamel.”

[from: T. Dean’s monthly report to Surgeon General Hugh S. Cumming, March 4, 1932, part of the H. Trendley Dean collection, History of Medicine Section, National Library of Medicine).]

Discussing a study at 8ppm, we find 3 to 5 blotches per nail:

“Another change noted was in the nails. From 10-20 percent of the younger individuals examined has a rather unusual type of nail structure, the most characteristic aspect being transverse white blotches often completely across the nail, usually symmetrical and on all the nails, there very frequently being from three to five of these per nail. [ See our image of nails below. ] The incidence of these finally decreased with age, the oldest patient being 57. In the control area with 139 high school students examined, none showed transverse striations.”

[Abstracts of the proceedings of the meetings of the Technical Advisory Committee on the fluoridation of water supplies with the Departmental Working Committee for the Newburgh-Kingston Demonstration; Harold C. Hodge, chairman; April 24, 1944; part of the H. Trendley Dean collection, History of Medicine Section, National Library of Medicine).]

blue line


Zinc is an essential trace mineral which was first recognized as dietary essential in 1934. Prior to 1963, zinc deficiency in human was unknown. The first deficiency was noted in Iran and Egypt (Prasad et al, 1963).  Today, it is recognized that a nutritional shortage of zinc is common throughout the world.  AUSTRALIA – A marginal deficiency of zinc appears to be prevalent in many segments of populations in developed countries, and more severe deficiencies are widespread in many parts of the world. Iron and zinc deficiencies, often occurring simultaneously for reasons which will be described elsewhere, are considered the leading world health dietary mineral problems.
[Zinc deficiencies are very common in Australian Soils]

Zinc plays a vital role in biochemical function as it is the essential component of the active site of a multitude of enzymes. Thirty years ago, only three enzymes that required zinc for their activities were identified; today more than 300 such enzymes have been classified (Prasad, 1996).

In 1974, the Food and Nutrition Board of the National Research Council of the National Academy of Sciences decided to establish a recommended dietary allowance for zinc as an essential element for humans.  It is only in the last 10 to 20 years that extensive research into zinc has taken place. Alone between 1990 and 1998 over 3600 studies on zinc were added by the National Library of Medicine.

The discovery of zinc “finger proteins” within the past two decades has further led to vastly improved understanding of how cells replicate and divide.  Zinc is required for each step of cell cycle in microorganisms and is essential for DNA synthesis (Prasad, 1989). Considering that nuclear receptors for the steroid/thyroid superfamily (which are all G-protein-coupled) are also all zinc finger proteins, it is not surprising that zinc deficiency impairs the metabolism of thyroid hormones, and rogens, and  growth hormones. Recent research also indicates that zinc finger proteins are required for binding of G proteins and GTP hydrolysis (Vitaleet al, 1998).

Zinc is probably involved in more body functions than any other trace mineral.

Zinc is also involved in blood pH regulation.

blue line


Zinc deficiency affects function of the testes, liver and muscles, and affects the structure of bones, teeth, hair and skin. Deficiency of zinc is associated with short stature, anaemia, increased pigmentation of skin (hyperpigmentation) -> Spira’s “mottled skin”, enlarged liver and spleen (hepatosplenomegaly), impaired gonadal function (hypogonadism), impaired wound healing, and immune deficiency, and cancer.

In animals, studies on reproduction have shown a significant reduction in the number of alive newborns in a group of rats submitted to severe zinc deficiency, confirming that zinc is a very important trace element overall during pregnancy (Sole et al, 1995). In rhesus monkeys, marginal zinc deficiency during gestation and during the 1st yr of life was found to be associated with significantly delayed skeletal maturation and defective mineralization (!). This abnormality of bone mineralization suggests that zinc plays an important role in endochondral bone formation (Leek et al, 1984).

In rats, growth rate, DNA level, RNA level, organ/serum zinc contents and alkaline phosphatase activities are markedly affected by zinc deficiency. Zinc deficiency significantly reduced growth rate by up to 60% in rats. (Okegbile et al, 1998; Ninh et al, 1998)

Of potentially extreme importance to humanity are the findings in test animals that zinc deficiency induced a chronic immunosuppression that persisted through the third generation of their offspring (Beach et al, 1982). (Of course similar effects lasting several generations have also been documented in fluoride-related literature.) Needless to say, if this also holds true for zinc-deprived humans, humanity certainly faces a grim future filled with disease.


Suboptimal zinc status can seriously impair human health, performance, reproductive functions, and mental and physical development (Roth and Kirchgessner, 1999). Zinc deficiency affects testicular functions adversely in man and animals. Zinc is essential for spermatogenesis. (Prasad, 1989)

Growth retardation, male hypogonadism, skin changes, poor appetite, mental lethargy and delayed wound healing are some of the manifestations of chronically zinc-deficient human subjects. In others, dermatological manifestations, diarrhea, hair loss, mental disturbances and intercurrent infections predominate. (Prasad, 1989). Several studies have shown that zinc deficiency is common in older people (Abbasi and Shetty, 1999)

Low levels of zinc are associated with advanced gastrointestinal cancer (Jia, 1991) – as is selenium, and lung cancer (Piccinini et al, 1996). A study testing zinc and copper levels in erythrocytes, plasma, and whole blood in 35 various cancer patients, comparing to 24 normal individuals, found a decrease in zinc in all three blood constituents of the cancer patients (Aldor et al, 1982).

Depending on the country, 5-30% of children suffer from moderate zinc deficiency, responsible for small-for-age height (Favier et al, 1992).

*Down Syndrome

As reported in PFPC NEWSLETTER #2, the number of excess Down Syndrome births due to water fluoridation is estimated to be several thousand cases annually throughout the world. All patients with with Down Syndrome have higher TSH levels (Licastro et al, 1992;1993), so it is obvious that the disease would occur more frequently in areas where a direct TSH mimick is in the public water supplies. In Down Syndrome, hypothyroidism is the most common endocrinological deficit (Bucci et al, 1999).  A close correlation was found between changes in plasma levels of zinc and changes in TSH in patients with chronic renal failure (Arreola et al, 1993).

In a study on children with Down Syndrome, ALL children had lower zinc levels than controls (Toledo et al, 1997).  Zinc supplementation was successful in DS and restored the T3/rT3 ratio to normal in several trials. (Licastro et al, 1992;1993), further showing evidence that zinc is essential for T4 to T3 synthesis in peripheral tissue.


Despite the widespread incidence of childhood zinc deficiency and strong evidence that zinc deprivation during periods of rapid growth affect brain development and behavior in animals, there is little research on the behavioral effects of zinc deficiency in children or adults. (Penland, 2000)

A new study on rats, published in the March 2000 issue of Brain Research, shows that a proper zinc supply to the brain is necessary for improvement and maintenance of learning ability. Synaptosomal zinc in the hippocampal formation and cerebral cortex was significantly decreased by 12 weeks of zinc deprivation. The results suggested that the decrease of vesicular zinc in the hippocampal formation and cerebral cortex is involved in the transient learning impairment of adults rats (Takeda et al, 2000).

Zinc levels in brain influence other brain metal content. One study investigated the effect of severe zinc deficiency on the distribution of nine elements in brain regions. Zinc deficiency caused an elevation in copper concentrations in most brain parts. Levels of calcium, manganese, sodium and potassium, in certain brain regions, also appeared to be altered by the zinc status of an animal. The levels of other metals were dependent on zinc nutriture. The data suggest the hypothesis that changes in brain metal content, associated with zinc deficiency, contribute to the behavioral abnormalities that occur. (Wallwork et al, 1983)

In rats fed green tea and black tea (extremely high in both F- and Al), zinc contents in the cerebrum were gradually decreased with the increase of tea leaves dose and tea concentration. While cerebrum calcium concentrations were also significantly reduced, aluminum levels increased (Zeyan et al, 1998).

In plasma, zinc and copper are in a direct oppositional relationship. Clinical observations and research have indicated that the copper/zinc ratio appears to be more decisively important than either of the individual metals alone. Zinc deficiency results in elevated blood levels of copper, due to the dynamic competition of these metals in the body. Elevated blood copper and decreased zinc levels have been associated with episodic violence, hyperactivity, learning disabilities, and depression in humans.

The high incidence of zinc deficiency in assaultive young males was illustrated in a recent study which found elevated serum copper and depressed plasma zinc concentration, compared to normal controls.
This study confirmed earlier clinical observations of zinc depletion in more than 4,000 behavior disordered patients (Walsh, 2000).

Dietary zinc deficiency also decreases plasma concentrations of vitamin E (Bunk et al, 1989).

Studies on depression showed significantly lower zinc levels in major depressed patients than in normal volunteers and it was suggested that lower serum zinc in depression may be due to interference in the liver (Maes et al, 1999).


Fluorides cause “zinc deficiency”. Both organic and inorganic fluoride compounds have shown to inhibit zinc containing enzymes, such as carbonic anhydrase (Dugad et al 1988,1989; Gelb et al, 1985) which is also now used as a marker for thyroid dysfunction (Hori et al, 1998). [Note: Carbonic anhydrase inhibitors reduce cerebrospinal fluid (CSF) production!]

Zinc depletion follows experimental fluorosis in mice (Kanwar and Singh,1981). In the liver a significant fall in the levels of zinc was registered. Effects were directly dose-dependent, just as is the case with fluoride-induced iodine deficiency. The liver is the major organ for thyroid hormone synthesis. Only 20% of T3, the biologically active thyroid hormone is manufactured in the thyroid itself.  The rest occurs in peripheral tissue, mainly the liver.

In rats fed pure spring water (natural FØ concentration = 0.2 ppm) or spring water enriched with NaF to result in 0.8, 1.1 or 2.2 ppm FØ during180 days, zinc ions were depleted in most tissues (Boeckhhaebisch & Oliveira, 1997).

→ → → Other work has also shown that a fluoride intake in rodents comparable to the total intake by man in fluoridated areas, leads to zinc deficiency in the testis, liver and kidneys (Krasowska, 1992, 1996).

Zinc protection from fluoride-induced testicular injury
Alicja Krasowska Institute of Biology,
University of Białystok, Poland


Li et al (1994) showed that excessive fluoride intake since early childhood reduced mental work capacity and that the effect on zinc metabolism was a mechanism of influence by excessive fluoride intake,  echoing earlier reports by Shen et al (1992).

Hlynczak et al (1980) also found significant alterations in workers exposed to fluorine compounds.

*Mottled teeth anyone?

Zinc deficiency causes mottled teeth in rats (Teraki  and Ishiyama, 1990).

Zinc and fluoride were water quality variables associated with mottling in a study investigating the prevalence of dental mottling in school-aged lifetime residents of 16 Texas communities (Butler et al, 1985). Platelets from zinc-deficient rats take up significantly less external calcium. Low zinc status in rats impairs calcium uptake and aggregation of platelets  stimulated by fluoride. (Emery & O’Dell, 1991; 1993)

blue line


During a conference held in 1996 on zinc and child health Dr. M.K. Bhan of the All India Institute for Medical Sciences in New Delhi found that low plasma zinc levels were associated with an increase in infectious disease morbidity in children. Robert Black of Johns Hopkins summarized studies suggesting that zinc can prevent diarrhoea, acute lower respiratory infections and pneumonia.  Keith West of Johns Hopkins School of Hygiene and Public Health showed that zinc deficiency may limit the impact of vitamin A intake on resistance to infection, child growth, and visual dark adaptation. The relationship between HIV progression and zinc deficiency was discussed by Andrea Ruff of Johns Hopkins, who found that progressors to AIDS had lower zinc levels than either non-progressors or HIV-negative individuals.

Fluoride kills.

Conference Report: Zinc for child health

3619 Citations from between 1990 and 1998 at

For complete list of references, please send message to and put REFS#5 in subject box.

blue line


The recommended daily requirement for zinc is
9-11mg for young people 14-18 years; and 8-11mg for adults 19 years and older.

Males require more zinc than females.
Pregnant or lactating females also show higher requirements (12-13mg/day).
Vitamin B6 improves zinc absorption.
Foods rich in zinc are seafood, meat and wheat germ. 

blue line

~ In Australia ~
More than one in three males (37%)
and one in ten females (9%) have inadequate zinc intakes.

blue line
The-Jornal-Of-NutritianZinc Status in Human Immunodeficiency Virus Infection1,3

… Adequate zinc status is essential for T-cell division,
maturation and differentiation; lymphocyte response
to mitogens; programmed cell death of lymphoid
and myeloid origins; gene transcription;
and biomembrane function…
blue line

blue line




Recommended by us


blue line


Full text here ↓ ↓ ↓


blue line

 See also: ‘The Drama Of Fluoride by Leo Spira’


More ↓

Fluorine-induced endocrine disturbances in mental illness.


Courier Mail (Brisbane) page 15 – 12 Nov 2013

Zinc stops the streptococcus pneumonia bacteria ( pneumonia )
from taking up manganese, an essential
metal it needs to cause disease … 

“Zinc is the second most abundant trace element in the human body.  
It is an essential mineral of exceptional biologic and public health importance.
Nearly 10 percent of the proteins encoded in the human genome require zinc for
their proper structure and function, including over 300 enzymes.


Signs of Zinc deficiency?
blue line

Ecology of Australia: the effects of nutrient-poor soils and intense fires.



We all need clean water!





Australian native animals are somewhat F. tolerant. 1080 (Sodium fluoroacetate) is used to kill dingos (dogs) (not strictly native to Australia). Mammalian animals including humans suffering with severe fluorosis, yield milk with a VERY low levels of fluoride. Nature protects her young!

See The Fifty + Links Below 

– Alligators – Koalas (a) Koalas (b) 

Ants,  |  Fish|  Australian Lung Fish,  |  Caddis Fly,

Caimans,  |  Camels,  |  Cattle,   |   Chinchillas,  |  Daphnia,

  Deer,   |  Dogs ,  |  Ducks|   Elephants (1), |  Elephants (2) ,

Fresh Water Fish«♦» |  Frogs (1),  |  Frogs (2),  |  Frogs (3),  |  Geese, |

Goats, | Guinea pigs ♦ see  below,  |   Horses,  | Race Horses 

Honey Bees,  |  KangaroosLizards,  |  Sheep,  |  Starling control, |

 Silver Foxes, |  Snails (1), Snails (2),  |  Rats (1),  |  Rats (2),  |

 Rats (3) Research-1934|Tadpoles (1), | Tadpoles Chinese research (2)

Tadpoles/frogs (3),  |  Zebrafish,  |  Pigs,  Dromedary camels

  RACEHORSES (DEATHS)  More on F. & Horses ←


PubMed logo

Fluoride associated with fetal malformation in the 1940s.



Zebrafish and Fluoride






End line

…Fluoride pollution in the aquatic ecosystems is mainly due to industrial activities (such as mining and processing of phosphorus rock and the manufacturing of aluminum) and agricultural activities (the applications of fluoride-containing fertilizers and pesticides) Fluoride levels in surface waters of rivers and lakes generally range from 0.01 mg/L to 0.3 mg/L, whereas fluoride-containing industrial wastewater was shown to contain fluoride at 96.8 mg/L [4], and in extreme cases, may have levels as high as 3000 mg/L to 5000 mg/L [5]. …




Cesium Contamination- Bluefin tuna-Fukushima  


Fluoride & 1080 – Sodium fluoroacetate ← Your pet dog


Industrial Fluoride Pollution YouTube    (Cattle)


«♦»  The toxicological experiments have shown that RAINBOW TROUT
Oncorhynchis mykiss is the most sensitive species to fluoride.

This is very comprehensive  –  ↓ ↓ ↓ –  but disturbing !

Insects, birds, animals, humans and insecticides




Extract: ‘The Complete Dinosaur’ + info from China

 Yes they suffered from fluorosis!




     ❝…Aquatic organisms living in soft waters may be more adversely affected by fluoride pollution than those living in hard or seawaters because the bioavailability of fluoride ions is reduced with increasing water hardness. Fluoride can either inhibit or enhance the population growth of algae, depending upon fluoride concentration, exposure time and algal species. Aquatic plants seem to be effective in removing fluoride from contaminated water under laboratory and field conditions. In aquatic animals, fluoride tends to be accumulated in the exoskeleton of invertebrates and in the bone tissue of fishes.

The toxic action of fluoride resides in the fact that fluoride ions act as enzymatic poisons, inhibiting enzyme activity and, ultimately, interrupting metabolic processes such as glycolysis and synthesis of proteins.

Fluoride toxicity to aquatic invertebrates and fishes increases with increasing fluoride concentration, exposure time and water temperature, and decreases with increasing intraspecific body size and water content of calcium and chloride. Freshwater invertebrates and fishes, especially net-spinning caddis fly larvae and upstream-migrating adult salmons, appear to be more sensitive to fluoride toxicity than estuarine and marine animals. Because, in soft waters with low ionic content, a fluoride concentration as low as 0.5 mg F-/l can adversely affect invertebrates and fishes, safe levels below this fluoride concentration are recommended in order to protect freshwater animals from fluoride pollution…❞

Lung fish f

Fluoridation MAY have implications for the Australian Lungfish Neoceratodus forsteri,
a protected species, which inhabit
the Wivenhoe Dam, if recycled water is returned to this dam.

We do NOT have Greywater in Queensland only Blackwater.

This is the reason why recycled water is a dangerous absurdity.

blue line blue line national-ocean-service-logo

Is any of this  toxic to coral ?

We think so !


Recycling sewage water with all of the above + mad cows disease must not to be considered – EVER.

blue line


Foods that contain Fluoride




2 salmon

Professor Paul Engelking says fish and fluoride don’t mix



…But technically speaking, the tadpoles didn’t die of “mercury poisoning” or “cadmium poisoning,” or “hydrofluoric acid” poisoning. They died because the chemicals ‘wrecked’ their immune systems leaving the tadpoles  vulnerable to all the germs and parasites in their environment…

4-frogs-on-a-stick-f fish-in-jepody


Fluoride Vol.27 No.4 220-226 1994 



  Effects of Sodium Fluoride on Carp and Rainbow Trout by K.S. Pillai and

Fluorides MAY explain some of the fingerling problems with Australian Bass (Macquaria novemaculeata) Fluoride can either inhibit or enhance the population growth of algae, depending upon the fluoride concentration…

British Patent GB 8236; filed 18th April 1896: ‘Fluorine  compounds for the purpose of destroying insects.’

Two blue butterflies


Honey bee

     Colony Collapse Disorder:

   ↓   The Bee Holocaust   ↓  (BBC 4)


Fipronil (Manufactured by Bayer) is highly toxic for bees *, crustaceans, insects and zooplankton, as well termites, rabbits, the fringe-toed lizard  and certain groups of gallinaceous birds. It appears to reduce the longevity and fecundity of female braconid parasitoids.

 Fipronil -Fluocyanobenpyrazole, C12H4Cl2F6N4OS,  is a slow-acting FLUORIDE BASED POISON. It is a broad spectrum insecticide that disrupts the insect central nervous system by blocking the passage of chloride ions through the GABA receptor and glutamate receptor I(GluCl), components of the central nervous system. This causes hyperexcitation of contaminated insects’ nerves and muscles.

 Seeds treated with Fipronil, produce flowers toxic to the bee’s brain — They get lost going home !

see also:


 Henry Miller has been exposed as a science fraud and professional liar who shills for Monsanto.

monsanto-nn 'MONSANTO' See Film Below 3

 Monsanto Caused 291,000 Suicides In India  

 ↑ ↑ ↑ ↑ ↑ ↑ ↑

Monsanto curse png copy

At-a-glance: from

  • Monsanto is boasting its partnership with 4-H Youth Development Organization, the country’s largest and most influential youth organization with more than 6.8 million members in 80 countries worldwide

  • According to the annual reports, 4-H is receiving funding from Monsanto, Bill & Melinda Gates Foundation, Cargill, DuPont, United Soybean Board, Coca-Cola, and Pfizer

  • Monsanto has been hijacking society at every turn, including government institutions, colleges and universities, media agencies, and so-called representatives of “science”

  • Monsanto recently attempted to brainwash young children with a 16-page book called ““Biotechnology Basics Activity Book” full of colorful pages and cartoon characters touting the “benefits” of GMO

Strong evidence also suggests ‘Neonicotinoid Pesticides

Manufactured by Bayer, is also a major bee killer.

France will outlaw this soon.

croplife-exposure-f fluoride-line

NON-TOXIC ALTERNATIVES for eradicating insect infestations in grains and museum objects can be oxygen depriving gases such as nitrogen or carbon dioxide, the later being used in sailing ships of old, – a lighted candle placed in the grain barrel prior to it being sealed up. No need for poison sprays, food irradiation, fluorides, or bromides etc.      – Simple safe and cheap, but not patentable!


Fluoride In New Zealand Birds: A Review


Pet Birds and Teflon Poisoning




Fipronil – (Contains Fluoride. ) It is used in Australia to spray locusts.



Animal Justice Party 

snail-we-all-need-clean-water BLUE-RED-BLUE strip


‘Termidor’ (Fipronil) is also used against termites.

Fipronil is broad spectrum insecticide that disrupts the insect central nervous system  by causing hyper excitation of nerves and muscles. Fipronil is a slow acting poison.  When mixed with a bait, it allows the poisoned insect time to return to the colony and kill other insects in the same nesting site.

Fipronil is highly toxic for crustaceansinsects (including bees and termites)
and zooplankton, as well as rabbits, the fringe-toed lizard,
and certain groups of gallinaceous birds.


David Suzuki f

FLUPYRADIFURONE-Yet Another Bee Killer


flupyyradifurone Image of two dogs

Frontline image

‘FRONTLINE’ for fleas, on pets contains Fipronil (Fluoride)  


Fluoride and 1080 – Sodium fluoroacetate

BLUE-RED-BLUE strip vet-poisions-info-service-logo


Fluoride Toothpaste – Pet Dogs – RSPCA – UK


Dogs and fluoride toothpaste-a no no!

dog-toxicBLUE-RED-BLUE strip

Australian native wild animals are somewhat F. tolerant. 1080 (Sodium fluoroacetate) is used to kill dingos (dogs) (not strictly native to Australia). Mammalian animals including humans suffering with severe fluorosis, yield milk with a VERY low levels of fluoride. — ‘Nature protects her young’ —

Fluoride Toxicity in Domestic and Wild Animals


Read More:




SULFOXAFLOR  a systemic insecticide which acts as an  insect neurotoxin and is a member a class of chemicals called sulfoximines which act on the central nervous system of insects. [Including BEES]





More info on  →  Daphnia and Fluoride
The water flea is the generally acknowledged standard aquatic, animal and the measure of its toxicity
test has been prescribed
as the obligatory item by many countries.

Sometimes Daphnia may be used in certain environments
to test the effects of toxins on an ecosystem, which makes them an indicator genus …

[Notice the young inside! ]


Credit photo to






Manataka American Indian Council™
Fluoride – dangerously high levels found in pet food
By Melissa Solis – Houston Pet Care Examiner

Pet food has a new level of danger hiding behind its labels. Environmental Working Group (EWG) just released a new study that showed high levels of fluoride in eight of ten pet foods tested. Consuming fluoride can lead to many serious health concerns.

Three studies show that boys who drink fluoridated tap water between the ages of 6 and 8 face a heightened risk of osteosarcoma, the rare but deadly form of bone cancer associated with fluoride (Bassin 2006; Cohn 1992; DHHS 1991). Scientists suspect that boys’ rapid growth may make them more susceptible to bone cancer (Bassin 2006)

OSTEOSARCOMA IS THE MOST COMMON PRIMARY BONE TUMOUR IN DOGS;  it is estimated to occur in over 8,000 dogs each year in the U.S., primarily in larger breeds (Chun 2003; Dernell 2001; Priester 1980; Withrow 1991). According to a recent expert review, the actual incidence is probably higher, since not all cases are confirmed and registered (Mueller 2007). Large dogs with fast growing bones are especially at risk. Only 5% of all osteosarcomas develop in dogs weighing less than 30 pounds and giant dogs generally develop osteosarcoma at a younger age compared to smaller-sized dogs and (Cooley 1997; Misdorp 1979).

A dog drinking adequate water would be exposed to 0.05-0.1 mg fluoride per kg of body weight daily, depending on the dog’s water consumption. A 10-pound puppy that eats about a cup of dog food a day would ingest approximately 0.25 mg fluoride/kg body weight/day based on average fluoride content in the 8 contaminated brands tested by EWG. At that rate, the puppy would consume 2.5 times more fluoride than EPA’s legal limit in drinking water.

When fluoride in drinking water is taken into consideration, a 10-pound puppy would be exposed to 3.5 times more fluoride than EPA allows in drinking water. Large breed puppies may be exposed to even more fluoride…


This research was carried out by the Queensland Government in 1952

Logo Qld Ag.Science ffFluorosis Sheep ff


↑  Full text – Three small books – Original Government research.  ↑



Melbourne. 1938

BULLETIN NO. 121 Observations on the Toxicity of Fluorine for Sheep

     CSIRO was original formed in 1926 as CSIR (Council for Scientific and Industrial Research)



Teflon sss copy

— P.T.F.E. —

( The F. is for fluoride )

Perfluoro-octanoic acid (PFOA), an ingredient of Teflon and also known as ‘C-8’, is a suspected carcinogen now found in humans, other animals and plants. PFOA is very persistent. Released into the environment it looks as if it will take literally millions of years to biodegrade.


Teflon – The Devil We Know 

Just get rid of the non-stick fry pan...



See also:

Merck Veterinary Manual

Dupont- Deadly Ch.

Teflon Cookware (contains Fluoride)

 polytetraflouethylene (PTFE).

“Like so many products developed for the sake of convenience  without concern for human health,  Teflon coated non-stick cookware when heated has proven to be a primary source of a dangerous toxic fluoride derivative, specifically perfluorinated chemicals.”

The chemical industry doesn’t want you to be afraid of Teflon pans, but you should be.


PTFE is odorless and invisible and bioaccumulates. When heated above 260ºC it is lethal to birds (pets) and will give humans flu like symptoms. A Teflon pan reached 721°F in just five minutes as measured by a commercially available infrared thermometer. However safe alternatives are now now available (from their web site):

 CeraMax Ceramic cookware is forged with a super-efficient thermal conductive base which instantly radiates heat throughout the entire pan in seconds.100% PTFE and PFOA free Ordinary non-stick pans often contain the chemical compounds PTFE and PFOA in their coatings. These chemicals can alter the original taste of your food and can even release toxins into your food..

See also   “Dump Dangerous Pots and Pans Laced With Cancer-Causing Chemicals”





Department of Biochemistry and Medical Chemistry,

Szczecin, Poland 

Department of Maxillofacial Surgery,

Hospital of the Ministry of Interior,  Kielce, Poland

 Published: 22 April 2020

Original →  HERE

This paper is mostly about humans – however:

   EXTRACT: In the pineal glands taken from the common merganser (Mergus merganser), Kalisinskaetal et al. recorded very high fluoride contents (mean > 1000 mg/kg dw), which even exceed the concentrations observed in the bones of these birds.

   Such a high concentration of fluoride in the gland is explained by the incompleteness of the blood–brain barrier in birds, which facilitates the penetration of various substances into the central nervous system.


. PFOA foam f

PFOA image f

Problems with fire fighting chemicals

ABCNews logo


It was revealed in September 2015 [This problem will be here for a long time.]

 the chemicals had leaked from Williamtown Air Force base into ground and surface water, prompting bans on bore water and closing fishing grounds. “Stop trying to downplay the seriousness of this issue. They are incredibly serious toxins, and it is a real worry for those people living around them.-” Mariann Lloyd-Smith, National Toxics Network senior advisor. – The New South Wales Environment Protection Authority (EPA) has started a statewide investigation into the historical use of the firefighting foam.

Editors comment: The RAAF once used harmless environmentally friendly fermented blood and bone, and liquorice to make fire fighting foam, – it made the plants grow fast…





Toxic Flame Retardants



Cesium Contamination- Bluefin tuna-Fukushima





   Guinea pigs have a requirement for vitamin C,

They  cannot manufacture or store vitamin C.
(Lack of the enzyme L-gulono-gamma-lactone oxidase)




new-devider-fq   gold-ribbon-1413076